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8688篇 您的检索式:作者名="OLSON"
    题名 作者 年代 出处 被引量
1The role of whole brain radiation therapy in the management of newly diagnosed brain metastases: a systematic review and evidence-based clinical practice guideline显示文摘Gaspar LE Mehta MP Patchell RA Burri SH Robinson PD Morris RE Ammirati M Andrews DW Asher AL Cobbs CS Kondziolka D Linskey ME Loeffler JS McDermott M Mikkelsen T Olson JJ Paleologos NA Ryken TC Kalkanis SN. 2010中国神经肿瘤杂志2010,8,1:98
2自组织团队的建设显示文摘团队是企业适应复杂动态变化环境的重要组织形式 ,高绩效的团队需要自组织工作。团队自组织工作的基本要素是团队成员之间存在有意义差异、互动沟通和运作氛围。通过案例介绍自组织团队建设的过程以及基本原理。姚立 刘洪 Olson Ed 2003系统辩证学学报2003,11,4:25
3大熊猫电刺激采精及精液冷冻保存研究显示文摘对卧龙自然保护区大熊猫研究中心的 9只 5 5~ 16 5岁的雄性大熊猫进行了 17次电刺激采精 .其中 3只是能进行自然交配并繁殖了后代的种公兽 .比较研究了精液离心、不同稀释液和冷冻方法对大熊猫精液超低温冷冻保存后的活力、运动状态、精子和顶体形态的影响 .细管冷冻是 1种较好的超低温冷冻精液的方法 .黄炎 李德生 张和民 杜军 张贵权 王鹏彦 Jogayle Howard Rrbacca Spindler Barbara Durrant Mary Ann Olson 2000四川师范学院学报(自然科学版)2000,21,3:16
4纸浆纤维悬浮液屈服应力及其在造纸工业中的应用研究进展显示文摘纸浆纤维悬浮液是以纤维为固相的非均相混合物,属多相流体。屈服应力作为纸浆纤维悬浮液最重要的流变参数之一,在制浆造纸高效、节能、低耗的生产操作技术及装备的优化设计方面扮演着重要的角色。本文就纸浆纤维悬浮液屈服应力的产生及其影响因素、测量方法及装置进行归纳和总结;在此基础上,阐述了纸浆纤维悬浮液的屈服应力在制浆造纸生产中的应用,并对纸浆纤维悬浮液屈服应力的进一步研究及其在造纸工业相关领域的发展应用提出了思路。沙九龙 王晨 James Olson 张辉 2016中国造纸学报2016,31,3:12
5射血分数保留的心力衰竭患者动脉硬化与运动的相关性显示文摘人类衰老以主动脉僵硬度增加为特征,通过降低动脉顺应性和增强收缩期波反射造成收缩期高血压。老年、高血压和肥胖是射血分数保留的心力衰竭(heart failure with preserved ejection fraction, HFpEF)发生发展最大的影响因素.Reddy YNV Andersen MJ Obokata M Koepp KE Kane GC Melenovsky V Olson TP Borlaug BA 练桂丽 叶鹏 2017中华高血压杂志2017,25,10:10
6Dual destructive and protective roles of adaptive immunity in neurodegenerative disorders显示文摘Inappropriate T cell responses in the central nervous system(CNS)affect the pathogenesis of a broad range of neuroinflammatory and neurodegenerative disorders that include,but are not limited to,multiple sclerosis,amyotrophic lateral sclerosis,Alzheimer’s disease and Parkinson’s disease.On the one hand immune responses can exacerbate neurotoxic responses;while on the other hand,they can lead to neuroprotective outcomes.The temporal and spatial mechanisms by which these immune responses occur and are regulated in the setting of active disease have gained significant recent attention.Spatially,immune responses that affect neurodegeneration may occur within or outside the CNS.Migration of antigen-specific CD4+T cells from the periphery to the CNS and consequent immune cell interactions with resident glial cells affect neuroinflammation and neuronal survival.The destructive or protective mechanisms of these interactions are linked to the relative numerical and functional dominance of effector or regulatory T cells.Temporally,immune responses at disease onset or during progression may exhibit a differential balance of immune responses in the periphery and within the CNS.Immune responses with predominate T cell subtypes may differentially manifest migratory,regulatory and effector functions when triggered by endogenous misfolded and aggregated proteins and cell-specific stimuli.The final result is altered glial and neuronal behaviors that influence the disease course.Thus,discovery of neurodestructive and neuroprotective immune mechanisms will permit potential new therapeutic pathways that affect neuronal survival and slow disease progression.Kristi M Anderson Katherine E Olson Katherine A Estes Ken Flanagan Howard E Gendelman R Lee Mosley 2014Translational Neurodegeneration2014,3,1:8
7Maintenance infliximab for Crohn’s disease: the ACCENT I randomised trial显示文摘Stephen B Hanauer Brian G Feagan Gary R Lichtenstein Lloyd F Mayer S Schreiber Jean Frederic Colombel Daniel Rachmilewitz Douglas C Wolf Allan Olson Weihang Bao Paul Rutgeerts 2002The Lancet2002,,9317:6
8Arabidopsis FtsZ2-1 and FtsZ2-2 Are Functionally Redundant, But FtsZ-Based Plastid Division Is Not Essential for Chloroplast Partitioning or Plant Growth and Development显示文摘FtsZ1 和 FtsZ2 是在共同本地化到中间质体的戒指的植物的 FtsZ 的种系发生地不同的家庭并且便于叶绿体的分割。在植物,在叶绿体分割改变了 FtsZ1 或 FtsZ2 原因剂量依赖者缺点的层次;因此,在 FtsZ 基因之间的功能的关系上的研究在 vivo 要求 FtsZ 层次的小心的操作。在 Arabidopsis thaliana, FtsZ2-1 和 FtsZ2-2 定义在二 FtsZ2 基因之间的功能的关系,我们在 ftsZ2-2 0 异种表示了 FtsZ2-1,并且反过来也如此,并且决定了叶绿体部门缺点是否在表示 FtsZ2 的不同全部的层次的植物被救。当 FtsZ2-1 或 FtsZ2-2 水平接近了时,完整的营救被观察全部的 FtsZ2 层次在野类型(WT ) 。另外, FtsZ2-2 与 ARC6 交往,为 FtsZ2-1 以前出现。这些数据显示 FtsZ2-1 和 FtsZ2-2 为在 Arabidopsis 的叶绿体分割是机能上地冗余的。严厉地为叶绿体分割验证每个 FtsZ 家庭的要求,我们在 vivo 用 FtsZ2 代替了 FtsZ1,并且反过来也如此,当在转基因的植物维持 FtsZ 水平时,在 WT 等于到全部的水平的。叶绿体部门缺点没被救,决定性地证明 FtsZ1 和 FtsZ2 为 WT 叶绿体数字的维护是非冗余的。最后,我们产生了完全缺乏 FtsZ 蛋白质的植物可行、肥沃的 ftsZ 三元组 0 异种和表演。因为质体是大概必要的细胞器,这些调查结果建议质体划分的一个 FtsZ 独立的模式可以发生在更高的植物。Aaron J. Schmitz Jonathan M. Glynn Bradley J.S.C. Olson Kevin D. Stokes Katherine W. Osteryoung 2009Molecular Plant2009,2,6:4
9Cybermaterials: materials by design and accelerated insertion of materials显示文摘Cybermaterials innovation entails an integration of Materials by Design and accelerated insertion of materials(AIM),which transfers studio ideation into industrial manufacturing.By assembling a hierarchical architecture of integrated computational materials design(ICMD)based on materials genomic fundamental databases,the ICMD mechanistic design models accelerate innovation.We here review progress in the development of linkage models of the process-structure–property-performance paradigm,as well as related design accelerating tools.Extending the materials development capability based on phase-level structural control requires more fundamental investment at the level of the Materials Genome,with focus on improving applicable parametric design models and constructing high-quality databases.Future opportunities in materials genomic research serving both Materials by Design and AIM are addressed.Wei Xiong Gregory B Olson 2016npj Computational Materials2016,,1:4
10Therapeutic Inhibition of miR-208a Improves Cardiac Function and Survival During Heart Failure显示文摘Rusty L. Montgomery Thomas G. Hullinger Hillary M. Semus Brent A. Dickinson Anita G. Seto Joshua M. Lynch Christianna Stack Paul A. Latimer Eric N. Olson Eva van Rooij 2011Circulation2011,,14:4
11Acute-on chronic liver failure显示文摘Rajiv Jalan Pere Gines Jody C Olson Rajeshwar P Mookerjee Richard Moreau Guadalupe Garcia-Tsao Vicente Arroyo Patrick S Kamath 2012Journal of Hepatology2012,,6:4
12MicroRNAs in Stress Signaling and Human Disease显示文摘Joshua T. Mendell Eric N. Olson 2012Cell2012,,6:4
13Status review of mercury control options for coal-fired power plants显示文摘John H Pavlish Everett A Sondreal Michael D Mann Edwin S Olson Kevin C Galbreath Dennis L Laudal Steven A Benson 2003Fuel Processing Technology2003,,2:4
14Cancer-Associated Fibroblasts Are Activated in Incipient Neoplasia to Orchestrate Tumor-Promoting Inflammation in an NF-κB-Dependent Manner显示文摘Neta Erez Morgan Truitt Peter Olson Douglas Hanahan 2010Cancer Cell2010,,2:4
15Soil and Crop Damages as a Result of Levee Breaches on Ohio and Mississippi Rivers显示文摘Kenneth R. Olson Lois Wright Morton 2013Journal of Earth Science and Engineering2013,3,3:4
16The Endothelial-Specific MicroRNA miR-126 Governs Vascular Integrity and Angiogenesis显示文摘Shusheng Wang Arin B. Aurora Brett A. Johnson Xiaoxia Qi John McAnally Joseph A. Hill James A. Richardson Rhonda Bassel-Duby Eric N. Olson 2008Developmental Cell2008,,2:4
17p53-mediated transcriptional regulation and activation of the actin cytoskeleton regulatory RhoC to LIMK2 signaling pathway promotes cell survival显示文摘响应 DNA 损坏的房间命运的中央仲裁人是 p53,它调整涉及房间周期拘捕,幸存和 apoptosis 的基因的表示。尽管 DNA 损坏开始的许多回答被描绘了,肌动朊细胞骨架管理者的角色大部分是未知的。我们现在显示出那 RhoC 和秘鲁利玛机场之代号 kinase (LIMK2 ) 2 是 genotoxic 代理人导致的直接 p53 目标基因。尽管 RhoC 和 LIMK2 在肌动朊细胞骨架规定有生长得很好的角色,我们的结果显示 LIMK2 的激活也有支持幸存的功能追随者 DNA 损坏。由调停 siRNA 的击倒或选择的药理学封锁的 LIMK 抑制敏化房间到无线电 -- 或化疗,以便当与 LIMK 抑制结合了时,当单身地管理了时,是尚不致命的治疗导致了房间死亡。我们的调查结果建议把 LIMK 禁止者与 genotoxic 治疗相结合能比单个代理人的管理更有效,并且加亮在肌动朊细胞骨架管理者和 DNA 之间的一个新奇连接导致损坏的房间幸存机制。Daniel R Croft Diane Crighton Michael S Samuel Filipe C Lourenco June Munro Jenifer Wood Karim Bensaad Karen H Vousden Owen J Sansom Kevin M Ryan Michael F Olson 2011Cell Research2011,21,4:3
18Automatic reconstruction of tree skeletal structures from point clouds显示文摘Yotam Livny Feilong Yan Matt Olson Baoquan Chen Hao Zhang Jihad El-Sana 2010ACM Transactions on Graphics (TOG)2010,,6:3
19Characteristics and origins of coal cleat: A review显示文摘S.E Laubach R.A Marrett J.E Olson A.R Scott 1998International Journal of Coal Geology1998,,1:3
20A Family of microRNAs Encoded by Myosin Genes Governs Myosin Expression and Muscle Performance显示文摘Eva van Rooij Daniel Quiat Brett A. Johnson Lillian B. Sutherland Xiaoxia Qi James A. Richardson Robert J. Kelm Eric N. Olson 2009Developmental Cell2009,,5:2
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