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    题名 作者 年代 出处 被引量
1Crohn’s disease显示文摘Daniel C Baumgart William J Sandborn 2012The Lancet2012,,9853:8
2Ulcerative colitis显示文摘Ingrid Ordás Lars Eckmann Mark Talamini Daniel C Baumgart William J Sandborn 2012The Lancet2012,,9853:5
3Identification of syndrome X using intravascular ultrasound imaging and Doppler flow mapping显示文摘Background The purpose of this study was to assess the morphological changes and physiological function of coronary arteries in patients presenting with chest pain but having normal coronary angiograms, using intravascular ultrasound imaging (IVUS) and intracoronary Doppler (ICD) flow measurements, in order to elucidate the mechanism of syndrome X. Methods A total of 126 patients [67 males, 59 females, mean age (53.1±13.0) years] who experienced chest pain but had normal coronary angiograms were included in this study. ICD flow measurements of the left anterior descending coronary artery (LAD) were performed using a Cardiometrics FloMap Ⅱ system. Coronary flow velocity reserve (CFVR) was defined as the ratio of the average peak velocity during hyperemia to that at baseline, induced by an intracoronary bolus injection of 18 μg adenosine. A 3.2F or 2.9F 30 MHz mechanical rotating ultrasound catheter (CVIS, Boston Scientific) or a 3.0F 20MHz electronic ultrasound catheter (Endosonics) was used for IVUS. Results The mean CFVR value of the LAD was 2.71±0.74. Reduction of CFVR (<3.0) was found in 82 of 126 (65.1%) patients. IVUS images of the LAD were available for 109 patients. Plaque formation was detected in 76/109 (69.7%) patients. Based on the presence or absence of plaque formation as well as the reduction or non-reduction of CFVR, patients were divided into four groups: Group Ⅰ (n=10), normal IVUS findings and normal CFVR; Group Ⅱ (n=23), normal IVUS findings with reduction in CFVR; Group Ⅲ (n=29), IVUS evidence of plaque formation but normal CFVR; and Group Ⅳ (n=47), IVUS evidence of plaque formation with reduction in CFVR. Conclusion This study shows the important clinical value of a combination of IVUS and ICD in diagnosing patients with angiographically normal coronary arteries. Only 10% of patients studied (Group Ⅰ) were found to be truly free of coronary disease, while 20% of patients (Group Ⅱ) would be diagnosed as suffering from syndrome X.钱菊英 葛均波 樊冰 王齐兵 陈灏珠 Dietrich Baumgart Michael Haude Raimund Erbel 2004Chinese Medical Journal2004,,4:4
4Ulcerative colitis显示文摘Ingrid Ordás Lars Eckmann Mark Talamini Daniel C Baumgart William J Sandborn 20122012 (9853)2012,,9853:3
5Crohn’s disease显示文摘Daniel C Baumgart William J Sandborn 2012The Lancet2012,,9853:3
6The second European evidence-based Consensus on the diagnosis and management of Crohn’s disease: Special situations显示文摘Gert Van Assche Axel Dignass Walter Reinisch C. Janneke van der Woude Andreas Sturm Martine De Vos Mario Guslandi Bas Oldenburg Iris Dotan Philippe Marteau Alessandro Ardizzone Daniel C. Baumgart Geert D’Haens Paolo Gionchetti Francisco Portela Boris Vuce 2009Journal of Crohn’s and Colitis2009,,1:3
7Docosahexaenoic acid suppresses arachidonic acid-induced proliferation of LS-174T human colon carcinoma cells显示文摘AIM:To investigate the impact of arachidonic acid (AA) and docosahexaenoic acid (DHA) and their combination on colon cancer cell growth.METHODS:The LS-174T colon cancer cell line was used to study the role of the prostaglandin precursor AA and the omega-3 polyunsaturated fatty acid DHA on cell growth. Cell viability was assessed in XTT assays. For analysis of cell cycle and cell death,flow cytometry and DAPI staining were applied. Expression of cyclooxygenase-2 (COX-2),p21 and bcl-2 in cells incubated with AA or DHA was examined by real-time RT-PCR. Prostaglandin E2 (PGE2) generation in the presence of AA and DHA was measured using a PGE2-ELISA.RESULTS:AA increased cell growth,whereas DHAreduced viability of LS 174T cells in a time-and dose-dependent manner. Furthermore,DHA down-regulated mRNA of bcl-2 and up-regulated p21. Interestingly,DHA was able to suppress AA-induced cell proliferation and significantly lowered AA-derived PGE2 formation. DHA also down-regulated COX-2 expression. In addition to the effect on PGE2 formation,DHA directly reduced PGE2-induced cell proliferation in a dose-dependent manner. CONCLUSION:These results suggest that DHA can inhibit the pro-proliferative effect of abundant AA or PGE2.Piet Habbel Karsten H Weylandt Katja Lichopoj Johannes Nowak Martin Purschke Jing-Dong Wang Cheng-Wei He Daniel C Baumgart Jing X Kang 2009World Journal of Gastroenterology2009,15,9:2
8Clinical relevance of caspase-1 activated cytokines in acute pancreatitis: High correlation of serum interleukin-18 with pancreatic necrosis and systemic complications显示文摘Bettina Rau Katja Baumgart Adam S. Paszkowski Jens M. Mayer Hans G. Beger 2001Critical Care Medicine2001,,8:2
9Ulcerative colitis显示文摘Ingrid Ordás Lars Eckmann Mark Talamini Daniel C Baumgart William J Sandborn 2012The Lancet . 2012 (9853)2012,,:2
10Crohn’s disease显示文摘Daniel C Baumgart William J Sandborn 2012The Lancet . 2012 (9853)2012,,:2
11Corrigendum to “Insights into the epigenetic mechanisms controlling pancreatic carcinogenesis” [Cancer Lett. 328 (2) (2012) 212–221]显示文摘Angela L. McCleary-Wheeler Gwen A. Lomberk Frank U. Weiss Günter Schneider Muller Fabbri Tara L. Poshusta Nelson J. Dusetti Sandra Baumgart Juan L. Iovanna Volker Ellenrieder Raul Urrutia Martin E. Fernandez-Zapico 2013Cancer Letters2013,,1:2
12Right Portal Vein Ligation Combined With In Situ Splitting Induces Rapid Left Lateral Liver Lobe Hypertrophy Enabling 2-Staged Extended Right Hepatic Resection in Small-for-Size Settings显示文摘Andreas A. Schnitzbauer Sven A. Lang Holger Goessmann Silvio Nadalin Janine Baumgart Stefan A. Farkas Stefan Fichtner-Feigl Thomas Lorf Armin Goralcyk Rüdiger H?rbelt Alexander Kroemer Martin Loss Petra Rümmele Marcus N. Scherer Winfried Padberg Alfred K? 2012Annals of Surgery2012,,3:2
13What’s new in inflammatory bowel disease in 2008?显示文摘Ulcerative colitis and Crohn’s disease represent the two major forms of inflammatory bowel disease. In this highlight topic series of articles we cover the latest developments in genetics and epidemiology, intestinal physiology, mucosal immunology, mechanisms of epithelial cell injury and restitution, current medical therapy, modern surgical management, important extra- intestinal complications such as primary sclerosing cholangitis, cholangiocellular carcinoma and autoimmune hepatitis as well as endoscopic and molecular screening, detection and prevention of small bowel and colorectal cancer.Daniel C Baumgart 2008World Journal of Gastroenterology2008,14,3:2
14Imaging techniques for assessment of inflammatory bowel disease: Joint ECCO and ESGAR evidence-based consensus guidelines显示文摘J. Panes Y. Bouhnik W. Reinisch J. Stoker S.A. Taylor D.C. Baumgart S. Danese S. Halligan B. Marincek C. Matos L. Peyrin-Biroulet J. Rimola G. Rogler G. van Assche S. Ardizzone A. Ba-Ssalamah M.A. Bali D. Bellini L. Biancone F. Castiglione R. Ehehalt R. G 2013Journal of Crohn’s and Colitis2013,,7:2
15Ulcerative colitis显示文摘Ingrid Ordás Lars Eckmann Mark Talamini Daniel C Baumgart William J Sandborn 2012The Lancet2012,,9853:2
16Attenuation of myocardial stunning by the ACE inhibitor ramiprilat through a signal cascade of bradykinin and prostglandins but not nitric oxide显示文摘Ehring T Baumgart D Kragcar M 1994Circulation1994,90,3:2
17Contribution of Fluid Dynamics to Woodwind Instruments Investigations of Timbre and Pitch of Bassoon Bocals显示文摘The discussed topic of isolating certain parameters which describe the timbre in connection with bocals of bassoons is of major interest for manufacturers and artist likewise since a reduction in the overall shear stress of these kinds of technical diffusors is very helpful but not sufficient. The timbre seen from the side of the musician has to be the same. Up to now two criteria have been isolated which have a deep influence on the easier blowing of the bassoon and on the comparable timbre due to geometrical reasons. If the geometrical outer contour is designed carefully, it is possible to modify the shape and preserve the timbre.Roger GRUNDMANN Johannes BAUMGART Andreas RICHTER Hans KRüGER 2005Journal of Thermal Science2005,14,3:2
18Biomarkers of Brain Injury in Neonatal Encephalopathy Treated with Hypothermia显示文摘An N. Massaro Taeun Chang Nadja Kadom Tammy Tsuchida Joseph Scafidi Penny Glass Robert McCarter Stephen Baumgart Gilbert Vezina Karin B. Nelson 2012The Journal of Pediatrics2012,,3:2
19Estrogen replacement therapy, serum lipids and polymorphism of the apolipoprotein E gene显示文摘Garry PJ Baumgarter RN Brodie SG 1999Clin Chem1999,45,:1
20Identifying medication error chains from critical incident reports: A new analytic approach 显示文摘Huckels Baumgart S Manser T 2014The Journal of Clinical Pharmacol ogy2014,37,4:1
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