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| 1 | Development of processing maps for as-cast ZE41A magnesium alloy显示文摘The hot deformation behavior of as-cast ZE41A magnesium alloy was studied by employing both processing maps and microstructural observations. To obtain processing map, hot compression tests were performed over a range of strain rates (0.001-1.0 s-1) and temperatures (250-450 ℃). Power dissipation efficiency (η) and instability parameter [ξ (ε)] were evaluated and processing maps were developed for the strain of 0.5. The dynamic recrystallization (DRX) and instable zones were identified and validated through micrographs. The processing maps can be used to select optimum strain rates and temperatures for effective hot working of the material. | S. ANBU SELVAN S. RAMANATHAN R . KARTHIKEYAN B. K. RAGHUNATH | 2010 | 中国有色金属学会会刊:英文版2010，20，1： | 4 |
| 2 | A phase II study of lapatinib in patients with advanced biliary tree and hepatocellular cancer显示文摘 | Ramesh K. Ramanathan Chandra P. Belani Deepti A. Singh Michael Tanaka Heinz-Josef Lenz Yun Yen Hedy L. Kindler Syma Iqbal Jeff Longmate Philip C. Mack Georg Lurje Regina Gandour-Edwards Janet Dancey David R. Gandara | 2009 | Cancer Chemotherapy and Pharmacology2009，，4： | 2 |
| 3 | Phase II trial of dacarbazine (DTIC) in advanced pancreatic islet cell carcinoma. Study of the Eastern Cooperative Oncology Group-E6282显示文摘 | R. K. Ramanathan A. Cnaan R. G. Hahn P. P. Carbone D. G. Haller | 2001 | Annals of Oncology2001，，8： | 1 |
| 4 | Transparent YAG from powder prepared by homogeneous precipitation reaction—Al(NO3)3+Y(NO3)3+(NH4)2SO4+CO(NH2)2显示文摘 | S. Ramanathan S. K. Roy Y. J. Bhat | 2001 | Journal of Materials Science Letters2001，，23： | 1 |
| 5 | Pediatric systemic lupus erythematosus presenting with coronary arteritis: A case series and review of the literature显示文摘 | Arunima Agarwal Stephanie Biglarian Medical student Sophia Lim-Stavros Jodie K. Votava-Smith Anusha Ramanathan | 2015 | Seminars in Arthritis and Rheumatism2015，，1： | 1 |
| 6 | Phase I study of a MUC1 vaccine composed of different doses of MUC1 peptide with SB-AS2 adjuvant in resected and locally advanced pancreatic cancer显示文摘 | Ramesh K. Ramanathan Kenneth M. Lee John McKolanis Elizabeth Hitbold Wolfgang Schraut Arthur J. Moser Elizabeth Warnick Theresa Whiteside Jennifer Osborne Hyoung Kim Roger Day Monica Troetschel Olivera J. Finn | 2005 | Cancer Immunology, Immunotherapy2005，，3： | 1 |
| 7 | Sediment and heavy metal accumulation in the Cauvery basin显示文摘 | A. L. Ramanathan V. Subramanian B. K. Das | 1996 | Environmental Geology1996，，3： | 1 |
| 8 | A phase II study of lapatinib in patients with advanced biliary tree and hepatocellular cancer显示文摘 | Ramesh K. Ramanathan Chandra P. Belani Deepti A. Singh Michael Tanaka Heinz-Josef Lenz Yun Yen Hedy L. Kindler Syma Iqbal Jeff Longmate Philip C. Mack Georg Lurje Regina Gandour-Edwards Janet Dancey David R. Gandara | 2009 | Cancer Chemotherapy and Pharmacology2009，，4： | 1 |
| 9 | A phase II study of lapatinib in patients with advanced biliary tree and hepatocellular cancer显示文摘 | Ramesh K. Ramanathan Chandra P. Belani Deepti A. Singh Michael Tanaka Heinz-Josef Lenz Yun Yen Hedy L. Kindler Syma Iqbal Jeff Longmate Philip C. Mack Georg Lurje Regina Gandour-Edwards Janet Dancey David R. Gandara | 2009 | Cancer Chemotherapy and Pharmacology2009，，4： | 1 |
| 10 | A phase II study of lapatinib in patients with advanced biliary tree and hepatocellular cancer显示文摘 | Ramesh K. Ramanathan Chandra P. Belani Deepti A. Singh Michael Tanaka Heinz-Josef Lenz Yun Yen Hedy L. Kindler Syma Iqbal Jeff Longmate Philip C. Mack Georg Lurje Regina Gandour-Edwards Janet Dancey David R. Gandara | 2009 | Cancer Chemotherapy and Pharmacology2009，，4： | 1 |
| 11 | Differential Regulation of IL-8 by IL-1β and TNFα in Hyaline Membrane Disease显示文摘 | K. Y. Kwong C. A. Jones R. Cayabyab C. Lecart C. L. Stotts I. Randhawa R. Ramanathan N. Khuu P. Minoo R. A. deLemos | 1998 | Journal of Clinical Immunology1998，，1： | 1 |
| 12 | Computational analysis of deleterious missense mutations in aspartoacylase that cause Canavan's disease显示文摘In this work, the most detrimental missense mutations of aspartoacylase that cause Canavan's disease were identified computationally and the substrate binding efficiencies of those missense mutations were analyzed. Out of 30 missense mutations, I-Mutant 2.0, SIFT and PolyPhen programs identified 22 variants that were less stable, deleterious and damaging respectively. Subsequently, modeling of these 22 variants was performed to understand the change in their conformations with respect to the native aspartoacylase by computing their root mean squared deviation (RMSD). Furthermore, the native protein and the 22 mutants were docked with the substrate NAA (N-Acetyl-Aspartic acid) to explain the substrate binding efficiencies of those detrimental missense mutations. Among the 22 mutants, the docking studies identified that 15 mutants caused lower binding affinity for NAA than the native protein. Finally, normal mode analysis determined that the loss of binding affinity of these 15 mutants was caused by altered flexibility in the amino acids that bind to NAA compared with the native protein. Thus, the pre- sent study showed that the majority of the substrate-binding amino acids in those 15 mutants displayed loss of flexibility, which could be the theoretical explanation of decreased binding affinity between the mutant aspartoacylases and NAA. | SREEVISHNUPRIYA K. CHANDRASEKARAN P. SENTHILKUMAR A. SETHUMADHAVAN R. SHANTHI V. DAISY P. NISHA J. RAMANATHAN K. RAJASEKARAN R. | 2012 | Science China(Life Sciences)2012，55，12： | 0 |
| 13 | Reckoning the SIX1 mutation's effects in branchio-oto-renal syndrome - A bioinformatics approach显示文摘 | B. Preethi V. Shanthi K. Ramanathan | 2015 | Frontiers in Biology2015，10，5： | 0 |
| 14 | 入院时即发生心源性休克的患者中的急诊血运重建:来自SHOCK试验和登记的报道显示文摘Aims: To determine clinical correlates and optimal treatment strategy in patients with cardiogenic shock(CS) on admission. Methods and results: In SHould we emergently revascularize Occluded Coronaries in cardiogenic shocK?(SHOCK) trial and registry patients with left ventricular(LV) dysfunction(n=1053), CS on admission occurred in 26% of directly admitted patients(n=166/627). Time from myocardial infarction to CS was shorter, initial haemodynamic profile poorer, and aggressive treatment less frequent in CS on admission than in delayed CS patients. CS on admission patients constituted a smaller relative proportion(11% ) of the transferred(n=48/426) when compared with the directly admitted cohort(P< 0.001). Inhospital mortality was higher(75 vs. 56% ; P< 0.001) with more rapid death(24-h mortality 40 vs. 17% ; P< 0.001) in CS on admission than in delayed CS patients. Emergency revascularization reduced in-hospital mortality in CS on admission(60 vs. 82% ; P=0.001) and in delayed CS patients similarly(46 vs.62% ; P< 0.001; interaction P=0.25). After adjustment for clinical differences, CS on admission was an independent predictor of in-hospital mortality(P=0.008). Conclusion: CS on admission patients have a worse outcome but benefit equally from emergency revascularization as delayed CS patients, emphasizing the need for rapid and direct access of CS on admission patients to facilities providing this care. | Jeger R.V. Harkness S.M. Ramanathan K. J.S. Hochman 杨海涛 | 2006 | 世界核心医学期刊文摘(心脏病学分册)2006，2，8： | 0 |
| 15 | A computational approach to explore the functional missense mutations in the spindle check point protein Mad1显示文摘在这个工作, Mad1 蛋白质的最有害的错误感觉变化那个原因癌症的各种各样的类型计算地被识别,那些错误感觉变化的底层绑定效率被分析。从 13 个错误感觉变化, IMutant 2.0, SIFTand PolyPhen 程序识别了分别地是不太稳定的、有害、损坏的 3 变体。随后,这 3 变体当模特儿被执行由计算他们的根平均数关于本国的 Mad1 在他们的符合构造理解变化摆平的偏差(RMSD ) 。而且,本国的蛋白质和 3 异种与有约束力的搭挡 Mad2 被停靠向底层解释那些有害错误感觉变化的有约束力的效率。停靠研究鉴别所有 3 异种比本国的蛋白质为 Mad2 引起了更低的有约束力的亲密关系。最后,正常模式分析决定这 3 异种的有约束力的亲密关系的损失被改变的灵活性在与本国的蛋白质相比绑在 Mad2 的氨基酸引起。因此,现在的学习证明在那些 3 异种的底层绑定氨基酸的那个多数显示了灵活性的损失,它能是与变异的 Mad1 和 Mad2 的减少的有约束力的亲密关系的理论解释。 | Merlin LOPUS Rao SETHUMADHAVAN P. CHANDRASEKARAN K. SREEVISHNUPRIYA A.W. VARSHA V. SHANTHI K. RAMANATHAN R. RAJASEKARAN | 2013 | Frontiers in Biology2013，8，6： | 0 |
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