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19篇 您的检索式:作者名="Irvin Modlin"
    题名 作者 年代 出处 被引量
1Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:8
2The Epidemiology of Gastroenteropancreatic Neuroendocrine Tumors显示文摘Ben Lawrence Bjorn I. Gustafsson Anthony Chan Bernhard Svejda Mark Kidd Irvin M. Modlin 2011Endocrinology and Metabolism Clinics of North America2011,,1:6
3The diagnosis and management of adult intussusception显示文摘Dennis G. Begos Andras Sandor Irvin M. Modlin 1997The American Journal of Surgery1997,,2:4
4Current Status of Gastrointestinal Carcinoids显示文摘Irvin M. Modlin Mark Kidd Igor Latich Michelle N. Zikusoka Michael D. Shapiro 2005Gastroenterology2005,,6:2
5Chromogranin A—Biological Function and Clinical Utility in Neuro Endocrine Tumor Disease显示文摘Irvin M. Modlin Bjorn I. Gustafsson Steven F. Moss Marianne Pavel Apostolos V. Tsolakis Mark Kidd 2010Annals of Surgical Oncology2010,,9:1
6Neuroendocrine Tumors of the Gallbladder: An Evaluation and Reassessment of Management Strategy显示文摘Karim M. Eltawil Bjorn I. Gustafsson Mark Kidd Irvin M. Modlin 2010Journal of Clinical Gastroenterology2010,,10:1
7Neuroendocrine tumors of the diffuse neuroendocrine system显示文摘Bj?rn I Gustafsson Mark Kidd Irvin M Modlin 2008Current Opinion in Oncology2008,,1:1
8Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:1
9The Increasing Prevalence of Acalculous Cholecystitis in Outpatients: Results of a 7-Year Study显示文摘PAUL E. SAVOCA WALTER E. LONGO KARL A. ZUCKER MARVIN M. MCMILLEN IRVIN M. MODLIN 1990Annals of Surgery1990,,4:1
10Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:1
11Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:1
12神经内分泌肿瘤68Ga-DOTATATE显像新观点:图像解读、体内分布、辐射内照射剂量及分子机制显示文摘利用68Ga-1,4,7,10-四氮杂环十二烷-1,4,7,10-四乙酸-D-苯丙氨酸1-酪氨酸3-苏氨酸8-奥曲肽(DOTATATE)PET/CT显像对表达生长抑素受体(SSTR)的肿瘤进行定位,就必须将图像信息有效地与患者诊疗过程相结合,包括临床病史、组织病理学、生物标志物、肿瘤分期和分级以及已知的影像学检查结果,另外还要考虑到患者已完成的治疗及检查对68Ga-DOTATATE图像的影响(如影响正常生理性分布并导致假阳性结果)。该文针对68Ga-DOTATATE PET/CT显像在图像采集、分析、神经内分泌肿瘤诊断方案及治疗中的价值提供指导意见。胡桂兰 庞芮(译) 霍力(审校) Lisa Bodei Valentina Ambrosini Ken Herrmann Irvin Modlin 2019中华核医学与分子影像杂志2019,39,8:1
13The diagnosis and management of adult intussusception显示文摘Dennis G. Begos Andras Sandor Irvin M. Modlin 1997The American Journal of Surgery1997,,2:1
14Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:1
15Colorectal disease in spinal cord patients显示文摘Walter E. Longo Garth H. Ballantyne Irvin M. Modlin 1990Diseases of the Colon & Rectum1990,,2:1
16Gastric stem cell:an update显示文摘Irvin M Modlin Mark Kidd Kevin D Lye 2003Keio Med2003,52,2:1
17An Analysis of Rare Carcinoid Tumors: Clarifying These Clinical Conundrums显示文摘Irvin M. Modlin M.D. Ph.D. Michael D. Shapiro B.S. Mark Kidd Ph.D 2005World Journal of Surgery2005,,1:1
18Gastroenteropancreatic neuroendocrine tumours显示文摘Irvin M Modlin Kjell Oberg Daniel C Chung Robert T Jensen Wouter W de Herder Rajesh V Thakker Martyn Caplin Gianfranco Delle Fave Greg A Kaltsas Eric P Krenning Steven F Moss Ola Nilsson Guido Rindi Ramon Salazar Philippe Ruszniewski Anders Sundin 2008Lancet Oncology2008,,1:1
19Glucagon receptor gene mutations with hyperglucagonemia but without the glucagonoma syndrome显示文摘Pancreatic neoplasms producing exclusively glucagon associated with glucagon cell hyperplasia of the islets and not related to hereditary endocrine syndromes have been recently described. They represent a novel entity within the panel of non-syndromic disorders associated with hyperglucagonemia. This case report describes a 36-year-old female with a 10 years history of nonspecific abdominal pain. No underlying cause was evident despite extensive diagnostic work-up. More recently she was diagnosed with gall bladder stones. Abdominal ultrasound, computerised tomography and magnetic resonance imaging revealed no pathologic findings apart from cholelithiasis. Endoscopic ultrasound revealed a 5.5 mm pancreatic lesion. Fine needle aspiration showed cells focally expressing chromogranin, suggestive but not diagnostic of a low grade neuroendocrine tumor. Octreo Scan was negative. Serum glucagon was elevated to 66 pmol/L(normal: 0-50 pmol/L). Other gut hormones, chromogranin A and chromogranin B were normal. Cholecystectomy and enucleation of the pancreatic lesion were undertaken. Postoperatively, abdominal symptoms resolved and serum glucagon dropped to 7 pmol/L. Although H and E staining confirmed normal pancreatic tissue, immunohistochemistry was initially thought to be suggestive of alpha cell hyperplasia. A count of glucagon positive cells from 5 islets, compared to 5 islets from 5 normal pancreata indicated that islet size and glucagon cell ratios were increased, however still within the wide range of normal physiological findings. Glucagon receptor gene(GCGR) sequencing revealed a heterozygous deletion,K349_G359del and 4 missense mutations. This case may potentially represent a progenitor stage of glucagon cell adenomatosis with hyperglucagonemia in the absence of glucagonoma syndrome. The identification of novel GCGR mutations suggests that these may represent the underlying cause of this condition.Helen C Miller Mark Kidd Irvin M Modlin Patrizia Cohen Roberto Dina Panagiotis Drymousis Panagiotis Vlavianos Günter Klöppel Andrea Frilling 2015World Journal of Gastrointestinal Surgery2015,7,4:0
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