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| 1 | The Epidemiology of Gastroenteropancreatic Neuroendocrine Tumors显示文摘 | Ben Lawrence Bjorn I. Gustafsson Anthony Chan Bernhard Svejda Mark Kidd Irvin M. Modlin | 2011 | Endocrinology and Metabolism Clinics of North America2011，，1： | 6 |
| 2 | Current Status of Gastrointestinal Carcinoids显示文摘 | Irvin M. Modlin Mark Kidd Igor Latich Michelle N. Zikusoka Michael D. Shapiro | 2005 | Gastroenterology2005，，6： | 2 |
| 3 | Chromogranin A—Biological Function and Clinical Utility in Neuro Endocrine Tumor Disease显示文摘 | Irvin M. Modlin Bjorn I. Gustafsson Steven F. Moss Marianne Pavel Apostolos V. Tsolakis Mark Kidd | 2010 | Annals of Surgical Oncology2010，，9： | 1 |
| 4 | Neuroendocrine Tumors of the Gallbladder: An Evaluation and Reassessment of Management Strategy显示文摘 | Karim M. Eltawil Bjorn I. Gustafsson Mark Kidd Irvin M. Modlin | 2010 | Journal of Clinical Gastroenterology2010，，10： | 1 |
| 5 | The guanylate cyclase-C signaling pathway is down-regulated in inflammatory bowel disease显示文摘 | ? ystein Brenna Torunn Bruland Marianne W. Furnes Atle van Beelen Granlund Ignat Drozdov Johanna Emg? rd Gunnar Br? nstad Mark Kidd Arne K. Sandvik Bj? rn I. Gustafsson | 2015 | Scandinavian Journal of Gastroenterology2015，，10： | 1 |
| 6 | Rarity of colon cancer in Africans is associated with low animal product consumption, not fiber显示文摘 | Stephen J.D O’Keefe Mark Kidd Greg Espitalier-Noel Peter Owira | 1999 | The American Journal of Gastroenterology1999，，5： | 1 |
| 7 | Neuroendocrine tumors of the diffuse neuroendocrine system显示文摘 | Bj?rn I Gustafsson Mark Kidd Irvin M Modlin | 2008 | Current Opinion in Oncology2008，，1： | 1 |
| 8 | Management of Gastric Carcinoids (Neuroendocrine Neoplasms)显示文摘 | Mark Kidd Bjorn I. Gustafsson | 2012 | Current Gastroenterology Reports2012，，6： | 1 |
| 9 | Differentiating the impact of anatomie and non-anatomic liver resection on early recurrence in patients with Hepatocellular Carcinoma显示文摘 | Karim M Eltawil Mark Kidd Francesco Giovinazzo | 2010 | World Journal of Surgieal O''neology2010，843，： | 1 |
| 10 | Gastric stem cell:an update显示文摘 | Irvin M Modlin Mark Kidd Kevin D Lye | 2003 | Keio Med2003，52，2： | 1 |
| 11 | An Analysis of Rare Carcinoid Tumors: Clarifying These Clinical Conundrums显示文摘 | Irvin M. Modlin M.D. Ph.D. Michael D. Shapiro B.S. Mark Kidd Ph.D | 2005 | World Journal of Surgery2005，，1： | 1 |
| 12 | Rapid early Miocene acceleration of uplift in the Gangdese Belt,Xizang (southern Tibet),and its bearing on accommodation mechanisms of the India-Asia collision显示文摘 | Copeland P Mark H T Kidd W S F Xu R G Zhang Y Q | 1987 | Earth and Planetary Science Letters1987，86，： | 1 |
| 13 | Glucagon receptor gene mutations with hyperglucagonemia but without the glucagonoma syndrome显示文摘Pancreatic neoplasms producing exclusively glucagon associated with glucagon cell hyperplasia of the islets and not related to hereditary endocrine syndromes have been recently described. They represent a novel entity within the panel of non-syndromic disorders associated with hyperglucagonemia. This case report describes a 36-year-old female with a 10 years history of nonspecific abdominal pain. No underlying cause was evident despite extensive diagnostic work-up. More recently she was diagnosed with gall bladder stones. Abdominal ultrasound, computerised tomography and magnetic resonance imaging revealed no pathologic findings apart from cholelithiasis. Endoscopic ultrasound revealed a 5.5 mm pancreatic lesion. Fine needle aspiration showed cells focally expressing chromogranin, suggestive but not diagnostic of a low grade neuroendocrine tumor. Octreo Scan was negative. Serum glucagon was elevated to 66 pmol/L(normal: 0-50 pmol/L). Other gut hormones, chromogranin A and chromogranin B were normal. Cholecystectomy and enucleation of the pancreatic lesion were undertaken. Postoperatively, abdominal symptoms resolved and serum glucagon dropped to 7 pmol/L. Although H and E staining confirmed normal pancreatic tissue, immunohistochemistry was initially thought to be suggestive of alpha cell hyperplasia. A count of glucagon positive cells from 5 islets, compared to 5 islets from 5 normal pancreata indicated that islet size and glucagon cell ratios were increased, however still within the wide range of normal physiological findings. Glucagon receptor gene(GCGR) sequencing revealed a heterozygous deletion,K349_G359del and 4 missense mutations. This case may potentially represent a progenitor stage of glucagon cell adenomatosis with hyperglucagonemia in the absence of glucagonoma syndrome. The identification of novel GCGR mutations suggests that these may represent the underlying cause of this condition. | Helen C Miller Mark Kidd Irvin M Modlin Patrizia Cohen Roberto Dina Panagiotis Drymousis Panagiotis Vlavianos Günter Klöppel Andrea Frilling | 2015 | World Journal of Gastrointestinal Surgery2015，7，4： | 0 |
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