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14804篇 您的检索式:作者名="DAVIS J"
    题名 作者 年代 出处 被引量
1血管内超声去肾交感神经术治疗高血压:一项多中心,国际,单盲,随机,假手术对照试验显示文摘早期研究表明,基于射频的去肾交感神经术可降低中度高血压患者的血压。研究者研究了在没有使用抗高血压药物的情况下,使用血管内超声去。肾交感神经术是否会降低高血压患者的动态血压。刘青 叶鹏 Azizi M Schmieder RE Mahfoud F Weber MA Daemen J Davies J Basile J Kirtane AJ Wang Y Lobo MD Saxena M Feyz L Rader F Lurz P Sayer J Sapoval M Levy T Sanghvi K Abraham J Sharp ASP Fisher NDL Bloch MJ Reeve-Stoffer H Coleman L Mullin C Mauri L 2018中华高血压杂志2018,26,10:27
22018年美国糖尿病学会和欧洲糖尿病研究协会关于2型糖尿病高血糖管理的共识显示文摘美国糖尿病学会(ADA)和欧洲糖尿病研究协会(EASD)近期组织专家对2012—2015年间发表的关于成年2型糖尿病的管理共识进行了更新。专家组系统评价了2014年至今发表的文献,对高血糖的管理提出了新的共识。新共识包括更加强调2型糖尿病患者的生活方式管理、自我管理教育和支持。共识指出对于2型糖尿病伴肥胖者建议通过生活方式干预、药物和手术进行减重。Davies MJ D'Alessio DA Fradkin J 姜莹莹(编译) 2019中华预防医学杂志2019,53,2:19
3下肢浅静脉治疗术后的加压疗法:美国静脉论坛血管、外科学会、美国静脉学会、血管医学学会和国际静脉联盟的临床实践指南显示文摘一、指南的由来下肢浅静脉治疗术后应用压力治疗的方式是几代血管外科医师多年临床经验积累所得,现主要应用于大隐静脉剥脱术、下肢静脉点状剥脱术及静脉硬化治疗等。目前单就静脉硬化治疗而言,术后压力治疗的推荐也是基于相关文献及实验验证[1],但对于其他术式,尤其是对于大隐静脉的热消融术,目前还没有形成规范统一的建议,这也使得当前的压力治疗具有很大的临床差异性。Lurie F Lal BK Antignani PL Blebea J Bush R Caprini J Davies A Forrestal M Jacobowitz G Kalodiki E Killewich L Lohr J Ma H Mosti G Partsch H Rooke T Wakefield T 李丹(译) 陈程浩(译) 牛传强(译) 张靖(译) 黄建忠(译) 2020中华介入放射学电子杂志2020,8,4:16
4A novel chemopreventive strategy based on therapeutic microRNAs produced in plants显示文摘Sizolwenkosi Mlotshwa Gail J Pruss John L MacArthur Matthew W Endres Celestia Davis Lome J Hofseth Maria Marjorette Pena Vicki Vance 2015Cell Research2015,25,4:12
5Plasmonics for solid-state lighting: enhanced excitation and directional emission of highly efficient light sources显示文摘Light sources based on reliable and energy-efficient light-emitting diodes (LEDs) are instrumental in the development of solid-statelighting (SSL). Most research efforts in SSL have focused on improving both the intrinsic quantum efficiency (QE) and the stability oflight emitters. For this reason, it is broadly accepted that with the advent of highly efficient (QE close to 1) and stable emitters, thefundamental research phase of SSL is coming to an end. In this study, we demonstrate a very large improvement in SSL emission (above70-fold directional enhancement for p-polarized emission and 60-fold enhancement for unpolarized emission) using nanophotonicstructures. This is attained by coupling emitters with very high QE to collective plasmonic resonances in periodic arrays of aluminumnanoantennas. Our results open a new path for fundamental and applied research in SSL in which plasmonic nanostructures are able tomold the spectral and angular distribution of the emission with unprecedented precision.Gabriel Lozano Davy J Louwers Said RK Rodrı´guez Shunsuke Murai Olaf TA Jansen Marc A Verschuuren Jaime Gomez Rivas 2013Light(Science & Applications)2013,2,1:8
6Dietary advanced glycation end-products aggravate non-alcoholic fatty liver disease显示文摘AIM To determine if manipulation of dietary advanced glycation end product(AGE), intake affects nonalcoholic fatty liver disease(NAFLD) progression and whether these effects are mediated via RAGE. METHODS Male C57Bl6 mice were fed a high fat, high fructose, high cholesterol(HFHC) diet for 33 wk and compared with animals on normal chow. A third group were given a HFHC diet that was high in AGEs. Another group was given a HFHC diet that was marinated in vinegar to prevent the formation of AGEs. In a second experiment, RAGE KO animals were fed a HFHC diet or a high AGE HFHC diet and compared with wildtype controls. Hepatic biochemistry, histology, picrosirius red morphometry and hepatic mR NA were determined. RESULTS Long-term consumption of the HFHC diet generated significant steatohepatitis and fibrosis after 33 wk. In this model, hepatic 4-hydroxynonenal content(a marker of chronic oxidative stress), hepatocyte ballooning, picrosirius red staining, α-smooth muscle actin and collagen type 1A gene expression were all significantly increased. Increasing the AGE content of the HFHC diet by baking further increased these markers of liver damage, but this was abrogated by pre-marination in acetic acid. In response to the HFHC diet, RAGE-/-animals developed NASH of similar severity to RAGE+/+ animals but were protected from the additional harmful effects of the high AGE containing diet. Studies in isolated Kupffer cells showed that AGEs increase cell proliferation and oxidative stress, providing a likely mechanism through which these compounds contribute to liver injury. CONCLUSION In the HFHC model of NAFLD, manipulation of dietary AGEs modulates liver injury, inflammation, and liver fibrosis via a RAGE dependent pathway. This suggests that pharmacological and dietary strategies targeting the AGE/RAGE pathway could slow the progression of NAFLD.Christopher Leung Chandana B Herath Zhiyuan Jia Sof Andrikopoulos Bronwyn E Brown Michael J Davies Leni R Rivera John B Furness Josephine M Forbes Peter W Angus 2016World Journal of Gastroenterology2016,22,35:7
7Osteoprotegerin: A Novel Secreted Protein Involved in the Regulation of Bone Density显示文摘W.S Simonet D.L Lacey C.R Dunstan M Kelley M.-S Chang R Lüthy H.Q Nguyen S Wooden L Bennett T Boone G Shimamoto M DeRose R Elliott A Colombero H.-L Tan G Trail J Sullivan E Davy N Bucay L Renshaw-Gegg T.M Hughes D Hill W Pattison P Campbell S Sander G Van 1997Cell1997,,2:6
8Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation显示文摘D.L Lacey E Timms H.-L Tan M.J Kelley C.R Dunstan T Burgess R Elliott A Colombero G Elliott S Scully H Hsu J Sullivan N Hawkins E Davy C Capparelli A Eli Y.-X Qian S Kaufman I Sarosi V Shalhoub G Senaldi J Guo J Delaney W.J Boyle 1998Cell1998,,2:4
9Efficacy of boceprevir, an NS3 protease inhibitor, in combination with peginterferon alfa-2b and ribavirin in treatment-naive patients with genotype 1 hepatitis C infection (SPRINT-1): an open-label, randomised, multicentre phase 2 trial显示文摘Paul Y Kwo Eric J Lawitz Jonathan McCone Eugene R Schiff John M Vierling David Pound Mitchell N Davis Joseph S Galati Stuart C Gordon Natarajan Ravendhran Lorenzo Rossaro Frank H Anderson Ira M Jacobson Raymond Rubin Kenneth Koury Lisa D Pedicone Clifford 2010The Lancet2010,,9742:3
10澳洲淡水龙虾(Cherax quadricarinatus)商业饲料中鸡肉粉替代鱼粉研究显示文摘水产动物红螯螯虾(Cherax quadricarinatus),俗称澳洲淡水龙虾,在热带和亚热带区域养殖发展十分迅速。由于饲料工业处于初级发展期,红螯螯虾营养需求与各种原料消化吸收率数据极其有限,生产饲料一般并未按照螯虾的特殊要求来单独设计配方。考虑到近期鱼粉价格上涨,使用类似鸡肉粉等替代蛋白源显得更为经济。本研究评价螯虾幼虾投喂使用鸡肉粉替代鱼粉的商业饲料生长效果。红螯螯虾幼虾(0.45±0.01) g暂养在户外圆池(容积24 600 L),密度为12.5尾/m2。养殖池水源为水库抽提,养殖期间保持流水。小龙虾投喂6种饲料,蛋白为26%,粗脂肪为7%,每个处理4个重复,在同等环境条件下生长良好。基础饲料中的鱼粉按比例被鸡肉粉替代,鱼粉含量递减为15%、12%、9%、6%、3%和0,而鸡肉粉含量递增为7.8%、10.5%、13.2%、15.8%、18.5%和21.2%,等氮等能饲料。经过56 d养殖试验,小龙虾所有试验组的存活率大于83%,增长11.3 g以上,处理组间没有显著差异。基于以上试验结果,可以得出结论,鸡肉粉适合作为小龙虾饲料中鱼粉的替代物。刘训华 何志刚 Saoud,I P Rodgers,L J Davis,D A 2019饲料与畜牧2019,0,6:2
11Bacteria on the rampage 显示文摘Davies J 1996Nature1996,383,:2
12Mathematical modeling of the extrusion of 6061/A12O3/20p composite 显示文摘CHEN W C DAVIES C H J SAMARASEKERA I V 1996Matallurgical and Materials Transactions1996,,27:2
13Stress hormone and blood glucose response follow acute stroke in the elderly显示文摘O Neill PA Davies I Fullerton K J 1991Stroke1991,22,7:2
14Age at diagnosis of type 2 diabetes and cardiovascular risk factor profile:A pooled analysis显示文摘BACKGROUND The diagnosis of type 2 diabetes(T2D)in younger adults,an increasingly common public health issue,is associated with a higher risk of cardiovascular complications and mortality,which may be due to a more adverse cardiovascular risk profile in individuals diagnosed at a younger age.AIM To investigate the association between age at diagnosis and the cardiovascular risk profile in adults with T2D.METHODS A pooled dataset was used,comprised of data from five previous studies of adults with T2D,including 1409 participants of whom 196 were diagnosed with T2D under the age of 40 years.Anthropometric and blood biomarker measurements included body weight,body mass index(BMI),waist circumference,body fat percentage,glycaemic control(HbA1c),lipid profile and blood pressure.Univariable and multivariable linear regression models,adjusted for diabetes duration,sex,ethnicity and smoking status,were used to investigate the association between age at diagnosis and each cardiovascular risk factor.RESULTS A higher proportion of participants diagnosed with T2D under the age of 40 were female,current smokers and treated with glucose-lowering medications,compared to participants diagnosed later in life.Participants diagnosed with T2D under the age of 40 also had higher body weight,BMI,waist circumference and body fat percentage,in addition to a more adverse lipid profile,compared to participants diagnosed at an older age.Modelling results showed that each one year reduction in age at diagnosis was significantly associated with 0.67 kg higher body weight[95%confidence interval(CI):0.52-0.82 kg],0.18 kg/m^(2) higher BMI(95%CI:0.10-0.25)and 0.32 cm higher waist circumference(95%CI:0.14-0.49),after adjustment for duration of diabetes and other confounders.Younger age at diagnosis was also significantly associated with higher HbA1c,total cholesterol,low-density lipoprotein cholesterol and triglycerides.CONCLUSION The diagnosis of T2D earlier in life is associated with a worse cardiovascular risk factor profile,compared to those diagnosed later in life.Mary M Barker Francesco Zaccardi Emer M Brady Gaurav S Gulsin Andrew P Hall Joseph Henson Zin ZinHtike Kamlesh Khunti Gerald P McCann Emma L Redman David R Webb Emma G Wilmot Tom Yates Jian Yeo Melanie J Davies Jack A Sargeant 2022World Journal of Diabetes2022,13,3:2
15Photosynthesis of nine pioneers Macaranga species from Borneo in relation to life history 显示文摘Davies S J 1998Ecology1998,79,:2
16The role of ammonium polyacrylate in dispersing concentrated alumina suspensions 显示文摘Davies J 2000J Eur Ceram Soc2000,20,10:2
17Fiber grating sensors显示文摘Kersey A D Davis M A Patrick H J 0,,:2
18Causes of encephalitis and differences in their clinical presentations in England: a multicentre, population-based prospective study显示文摘Julia Granerod Helen E Ambrose Nicholas WS Davies Jonathan P Clewley Amanda L Walsh Dilys Morgan Richard Cunningham Mark Zuckerman Ken J Mutton Tom Solomon Katherine N Ward Michael PT Lunn Sarosh R Irani Angela Vincent David WG Brown Natasha S Crowcroft 2010The Lancet Infectious Diseases2010,,12:2
19Biliary-colonic fistula caused by cholecystectomy bile duct injury显示文摘Biliary-colonic fistula is a rare complication after laparoscopic cholecystectomy. We present a case of post-cholecystectomy iatrogenic biliary injury that resulted in a fistula between the common hepatic duct and large bowel. Magnetic resonance cholangiopancreatography provided good visualization of injury even with concurrent normal level of alkaline phosphatase. Radiologic findings and surgical management of this condition are discussed in detail.Francisco Igor B Macedo Victor J Casillas James S Davis Joe U Levi Danny Sleeman 2013Hepatobiliary & Pancreatic Diseases International2013,12,4:2
20Bacteria on the rampage显示文摘Davies J 0,,:2
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