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| 1 | Minimally invasive procedures on the lumbar spine显示文摘Degenerative disease of the lumbar spine is a common and increasingly prevalent condition that is often implicated as the primary reason for chronic low back pain and the leading cause of disability in the western world. Surgical management of lumbar degenerative disease has historically been approached by way of open surgical procedures aimed at decompressing and/or stabilizing the lumbar spine. Advances in technology andsurgical instrumentation have led to minimally invasive surgical techniques being developed and increasingly used in the treatment of lumbar degenerative disease. Compared to the traditional open spine surgery, minimally invasive techniques require smaller incisions and decrease approach-related morbidity by avoiding muscle crush injury by self-retaining retractors, preventing the disruption of tendon attachment sites of important muscles at the spinous processes, using known anatomic neurovascular and muscle planes, and minimizing collateral soft-tissue injury by limiting the width of the surgical corridor. The theoretical benefits of minimally invasive surgery over traditional open surgery include reduced blood loss, decreased postoperative pain and narcotics use, shorter hospital length of stay, faster recover and quicker return to work and normal activity. This paper describes the different minimally invasive techniques that are currently available for the treatment of degenerative disease of the lumbar spine. | Branko Skovrlj Jeffrey Gilligan Holt S Cutler Sheeraz A Qureshi | 2015 | World Journal of Clinical Cases2015，3，1： | 8 |
| 2 | Classification and terminology of plant epicuticular waxes显示文摘 | WILHELM BARTHLOTT CHRISTOPH NEINHUIS DAVID CUTLER FRIEDRICH DITSCH IRIS MEUSEL INGE THEISEN HILTRUD WILHELMI | 1998 | Botanical Journal of the Linnean Society1998，，3： | 3 |
| 3 | Remembering your A, B, C’s: Alzheimer’s disease and ABCA1显示文摘The function of ATP binding cassette protein A1(ABCA1)is central to cholesterol mobilization.Reduced ABCA1 expression or activity is implicated in Alzheimer’s disease(AD)and other disorders.Therapeutic approaches to boost ABCA1 activity have yet to be translated successfully to the clinic.The risk factors for AD development and progression,including comorbid disorders such as type2 diabetes and cardiovascular disease,highlight the intersection of cholesterol transport and inflammation.Upregulation of ABCA1 can positively impact APOE lipidation,insulin sensitivity,peripheral vascular and blood-brain barrier integrity,and anti-inflammatory signaling.Various strategies towards ABCA1-boosting compounds have been described,with a bias toward nuclear hormone receptor(NHR)agonists.These agonists display beneficial preclinical effects;however,important side effects have limited development.In particular,ligands that bind liver X receptor(LXR),the primary NHR that controls ABCA1 expression,have shown positive effects in AD mouse models;however,lipogenesis and unwanted increases in triglyceride production are often observed.The longstanding approach,focusing on LXRβvs.LXRa selectivity,is over-simplistic and has failed.Novel approaches such as phenotypic screening may lead to small molecule NHR modulators that elevate ABCA1 function without inducing lipogenesis and are clinically translatable. | Cutler T.Lewandowski Megan S.Laham Gregory R.J.Thatcher | 2022 | Acta Pharmaceutica Sinica B2022，12，3： | 3 |
| 4 | Increasing prevalence of nonalcoholic steatohepatitis as an indication for liver transplantation显示文摘 | R. Cutler Quillin Gregory C. Wilson Jeffrey M. Sutton Dennis J. Hanseman Flavio Paterno Madison C. Cuffy Ian M. Paquette Tayyab S. Diwan E. Steve Woodle Daniel E. Abbott Shimul A. Shah | 2014 | Surgery2014，，4： | 2 |
| 5 | Neuroinflammation and subarachnoid hemorrhage: a revised look at the literature显示文摘A key topic for aneurysmal subarachnoid hemorrhage is neuroinflammation.Neuroinflammation can predispose to aneurysm formation and rupture.Neuroinflammation can also result from the blood breakdown products after aneurysm rupture.Recent evidence has shown that perpetual neuroinflammation can contribute to vasospasm and hydrocephalus.Targeting neuroinflammation is a novel mechanism for preventing subsequent neurologic sequalae.In this review,we highlight the pathophysiology of aneurysm formation,the neuroinflammatory surge after rupture including the involved cytokines,and ultimately tie in the contributory clinical relevance.In the last sections,we look at the pre-clinical data and novel avenues for further discovery.This paper will be a useful resource to both the clinician and scientific investigator. | Sai Sriram Christopher Cutler Mohammed Azab Ramya Reddy Rodeania Peart Brandon Lucke-Wold | 2022 | Clinical Research Communications2022，5，3： | 2 |
| 6 | Uric acid and serum antioxidant capacity: a reaction to atherosclerosis?显示文摘 | F.Javier Nieto Carlos Iribarren Myron D. Gross George W. Comstock Richard G. Cutler | 2000 | Atherosclerosis2000，，1： | 2 |
| 7 | SCH 503034, a Novel Hepatitis C Virus Protease Inhibitor, Plus Pegylated Interferon α-2b for Genotype 1 Nonresponders显示文摘 | Christoph Sarrazin Regine Rouzier Frank Wagner Nicole Forestier Dominique Larrey Samir K. Gupta Musaddeq Hussain Amrik Shah David Cutler Jenny Zhang Stefan Zeuzem | 2007 | Gastroenterology2007，，4： | 2 |
| 8 | A combined analysis of the cystic fibrosis transmembrane conductance regulator: implications for structure and diease models显示文摘 | Chen J M Cutler C Jacques C | 2001 | Mol Biol Evol2001，18，9： | 2 |
| 9 | Classification and terminology of plant epicuticular waxes显示文摘 | WILHELM BARTHLOTT CHRISTOPH NEINHUIS DAVID CUTLER FRIEDRICH DITSCH IRIS MEUSEL INGE THEISEN HILTRUD WILHELMI | 1998 | Botanical Journal of the Linnean Society1998，，3： | 2 |
| 10 | Oxygen radical absorbance capacity assay for antioxidants显示文摘 | Cao G A lessio HM Cutler RG | 1993 | Free Radic Biol Med1993，14，3： | 2 |
| 11 | Colonoscopic miss rates of adenomas determined by back-to-back colonoscopies显示文摘 | DK Rex CS Cutler GT Lemmel EY Rahmani DW Clark DJ Helper GA Lehman DG Mark | 1997 | Gastroenterology1997，，： | 2 |
| 12 | ACCORD试验中血压干预的原理和设计显示文摘ACCORD血压试验是非盲法、公开标记、随机试验,对象为4733例受试者。本文中描述ACCORD试验中血压干预的原理、设计和方法。符合血压试验纳入标准的受试者被随机分入治疗目标不同两组:收缩压<120mmHg为强化目标。 | Cushman W.C. Grimm Jr. R.H. Cutler J.A. 马超 | 2007 | 世界核心医学期刊文摘(心脏病学分册)2007，，11： | 2 |
| 13 | Effect of solidification parameters on the microstructures and properties of CMSX-10显示文摘 | B.C. Wilson E.R. Cutler G.E. Fuchs | 2007 | Materials Science & Engineering A2007，，1： | 2 |
| 14 | High-does therapy with single autologous transplantation versus chemotherapy for newly diagnosed multiple myeloma: A systematic review and meta-analysis of randomized controlled trials显示文摘 | Koreth J Cutler CS Djulbegovic B | 2007 | Biol Blood Marrow Transplant2007，13，2： | 1 |
| 15 | Characterization of frequency stability显示文摘 | Barnes J A Chi A R Cutler L S | 1971 | IEEE Transactions on Instrumentation and Measurement1971，20，2： | 1 |
| 16 | Heightened gingival inflammation and attachment loss in type 2 diabetics with hyperlipidemia显示文摘 | Cutler CW Machen RL Jotwani R | 1999 | J Periodontol1999，70，11： | 1 |
| 17 | Can the neuropathic Pain Scale discriminate between non-neuropathic and neuropathic pain 显示文摘 | Fishbain DA Lewis JE Cutler R | 2008 | Pain Medicine2008，9，2： | 1 |
| 18 | A simple method of preparing plant samples for PCR显示文摘 | WANG H QI M CUTLER A J | 1993 | Nucleic Acids Research1993，21，： | 1 |
| 19 | Indentation fracture response and damage resistance of Al2O3-ZrO2 compositesstrengthened by transformation-induced residual stresses显示文摘 | Hansen J J Cutler R A Shetty D K | 1988 | Journal of the American Ceramic Society1988，171，12： | 1 |
| 20 | Sinterable sialon powder by reaction of clay with carbon and nitrogen显示文摘 | LEE J G CUTLER I B | 1979 | Am Ceram Soc Bull1979，58，9： | 1 |
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