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33篇 您的检索式:作者名="Wayne MA"
    题名 作者 年代 出处 被引量
1Adiponectin resistance and vascular dysfunction in the hyperlipidemic state显示文摘胰岛素在脉管的氮的氧化物生产的刺激起一个重要作用,与两个短术语(vasomotility 和 anti-thrombotic 效果) 并且长期(光滑的肌肉房间生长和迁居抑制) 受益。对胰岛素的损害 vasodilatory 反应,脉管的胰岛素抵抗(红外) 的特点,为循环 pathophysiology 有重要含意。在 adipokines 和红外之间的一个协会在糖尿病患者和 nondiabetic 状态被观察了。Adiponectin (APN ) 是知道刺激骨胳的肌肉的敏化胰岛素的 adipokine 丰满的酸(FA ) 氧化和还原剂类脂化合物累积。在在脉管的功能规定的 APN 和胰岛素之间的潜在的串音的最近的示范特别地有趣。观察在以后的类脂化合物累积长期高脂肪(HF ) 节食并且可以处于肥胖的状态把脉管的反应归结为 APN,一个病理学的状态作为 APN 抵抗称为。这评论在 endothelial 功能的维护强调胰岛素敏感和 APN 活动的重要性。它在 hyperlipidemic 探索在脉管的红外和 APN 抵抗之间的关系病理学的状况,新陈代谢的症候群的代表。脉管的胰岛素和 APN 抵抗的调查为在新陈代谢的症候群影响的个人的治疗学的指向提供脉管的 pathophysiology,而且一个机会的不仅更好的理解。Rong LI Wayne Bond LAU Xin Liang MA 2010Acta Pharmacologica Sinica2010,31,10:9
2Novel synthesis of fly-ash-derived Cu-loaded SAPO-34 catalysts and their use in selective catalytic reduction of NO with NH3显示文摘A combined acid–alkali hydrothermal method was used to prepare fly ash–derived SAPO-34 molecular sieves from a thermal power plant in Inner Mongolia(China).The specific surface area of the prepared fly-ash-derived SAPO-34 molecular sieves was 579 m^2 g^-1,the total pore volume was about 0.27 cm^3 g^-1,and the pore size was 0.56 nm;the molar ratios of Al2O3:P2O5:SiO2 were 1:0.86:0.45.Cu/SAPO-34 catalysts were prepared by impregnation of low-cost fly-ash-derived SAPO-34 molecular sieves as a support and tested in selective catalytic reduction with NH3(NH3-SCR).Powder X-ray diffraction(XRD),N2 adsorption–desorption,X-ray photoelectron spectroscopy(XPS),H2 temperatureprogrammed reduction(H2-TPR),NH3 temperature-programmed desorption(NH3-TPD),electron paramagnetic resonance(EPR),nuclear magnetic resonance(NMR),X-ray fluorescence analysis(XRF)and scanning electron microscopy(SEM)were used for catalyst characterization and investigation of the relationships between the catalyst structure and the catalytic activity.The actual silica:alumina ratio of the molecular sieves did not increase with increasing Cu loading,indicating that increasing the Cu loading does not change the original structure of the SAPO-34 molecular sieves.The XRF and NMR results showed that replacement by Cu results in more Si islands.The molecular sieve acidity decreased because of the increased number of Si islands.The NH3-TPD results showed that for the Cu/SAPO-34 catalysts there was a low correlation between the low-temperature activity and the amount of acidic sites.SCR activity is closely related to the location of Cu.The 4.47 Cu/SAPO-34 catalyst has the highest isolated Cu2+showed the highest NH3-SCR activities(>90%)at 250–350℃.This work opens up new avenues for recycling fly ash formed in coal-fired power plants(reducing environmental pollution)and developing low-cost SCR catalysts for NOx pollution control.Ge Li Baodong Wang Qi Sun Wayne Qiang Xu Ziran Ma Hongyan Wang Daojun Zhang Jiali Zhou 2019Green Energy & Environment2019,4,4:6
3Autoantibody against angiotensin AT1 receptor from preeclamptic patients enhances collagen-induced human platelet aggregation显示文摘Hypercoagulability,血小板激活,和 thrombocytopenia 是 preeclampsia 的主要特征,但是他们的负责的内在的分子的机制仍然保持阴暗。最近的研究证明了对血管收缩素 II 类型 1 受体(AT1-AA ) 的自身抗体为 preeclampsia 组成一个新奇风险因素。然而,在血小板激活的 AT1-AA 和在 preeclampsia 的 hypercoagulability 的角色从来没被调查过。在现在的学习,我们决定了 AT1-AA 是否在 vitro 支持血小板聚集,并且把潜在的位于 \O 下面机制。AT1-AA 被连接酶的 immunosorbent 试金检测。在从 preeclamptic 病人净化的免疫球蛋白 G 部分以后,积极 sera 被加到从健康志愿者,血小板聚集和细胞内部的 Ca2+ 孤立的血小板层次被检测。AT1-AA 显著地在 vitro 提高了导致骨胶原的血小板聚集,效果由 AT1 受体对手 losartan 堵住了。另外, AT1-AA 在整个实验增加了并且维持导致骨胶原的 cytosolic 钙集中。我们第一次证明 AT1-AA 显著地在 vitro 通过血管收缩素类型 1 受体激活支持导致骨胶原的血小板聚集,潜在地经由增加的细胞内部的 Ca2+ 集中,作为一个潜在的贡献者支持 AT1-AA 到 preeclampsia 的 hypercoagulable 状态。Kehua Bai Ke Wang Xiaoyu Li Jie Wang Jie Zhang LiSong Jin Wang Suli Zhang Wayne Bond Lau Xinliang Ma Huirong Liu 2013Acta Biochimica et Biophysica Sinica2013,45,9:2
4End-tidal carbon dioxide and outcome of out-of-hospital cardiac arrest显示文摘Levine RL Wayne MA Miller CC 1997N Engl J Med1997,337,:1
5Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic mice显示文摘Hui Su Yuexing Yuan Xiao-Ming Wang Wayne Bond Lau Yajing Wang Xiaoliang Wang Erhe Gao Walter J. Koch Xin-Liang Ma 2013Basic Research in Cardiology2013,,1:1
6Essential Role of Caveolin-3 in Adiponectin Signalsome Formation and Adiponectin Cardioprotection显示文摘Yajing Wang Xiaoliang Wang Jean-Fran?ois Jasmin Wayne Bond Lau Rong Li Yuexin Yuan Wei Yi Kurt Chuprun Michael P. Lisanti Walter J. Koch Erhe Gao Xin-Liang Ma 2012Arteriosclerosis Thrombosis and Vascular Biology2012,,4:1
7Clinical Outcomes With the Epicholorohydrin-Modified Porcine Aortic Heart Valve: A 15-Year Follow-Up显示文摘Xufeng Wei Wei Yi Wensheng Chen Xinliang Ma Wayne Bond Lau Hongbing Wang Dinghua Yi 2010The Annals of Thoracic Surgery2010,,5:1
8AMP-Activated Protein Kinase Deficiency Enhances Myocardial Ischemia/Reperfusion Injury but Has Minimal Effect on the Antioxidant/Antinitrative Protection of Adiponectin显示文摘Yajing Wang Erhe Gao Ling Tao Wayne Bond Lau Yuexin Yuan Barry J. Goldstein Bernard L. Lopez Theodore A. Christopher Rong Tian Walter Koch Xin-Liang Ma 2009Circulation2009,,6:1
9“Know Diabetes by Heart”: role of adipocytecardiomyocyte communications显示文摘Cardiovascular disease is the leading cause of morbidity in patients with diabetes mellitus.In 2019,the American Heart Association and the American Diabetes Association(along with industry leaders)launched the groundbreaking collaborative initiative“Know Diabetes by Heart™”to reduce cardiovascular deaths in type 2 diabetic patients.The molecular basis linking diabetes with cardiovascular complications has not yet been fully defined.Recent clinical and experimental studies strongly suggest that adipocyte dysfunction and subsequent pathological communications between adipocyte and cardiomyocytes play important roles in diabetic cardiac injury.This perspective article will review recent development concerning adipocyte-cardiomyocyte communications,and identify the most critical questions remain to be answered in this filed.Yajing Wang Wayne Bond Lau Xinliang Ma 2021Medical Review2021,1,1:1
10Signature microRNA Expression Profile of Essential Hypertension and Its Novel Link to Human Cytomegalovirus Infection显示文摘Shuqiang Li Jianguo Zhu Weili Zhang Youren Chen Ke Zhang Laurentiu M. Popescu Xinliang Ma Wayne Bond Lau Rong Rong Xueqing Yu Bingbing Wang Yafeng Li Chuanshi Xiao Mingming Zhang Shuyan Wang Liping Yu Alex F. Chen Xinchun Yang Jun Cai 2011Circulation2011,,:1
11Standard cardiopulmonary resuscitation versus active compression- decompression cardiopulmonary resuscitation with augmentation of negative intrathoracic pressure for out-of-hospital cardiac arrest: a randomised trial 显示文摘Aufderheide TP Frascone R J Wayne MA 2011Lancet2011,377,9762:1
12Cardiac-derived adiponectin induced by long-term insulin treatment ameliorates myocardial ischemia/reperfusion injury in type 1 diabetic mice via AMPK signaling显示文摘Haifeng Pei Yan Qu Xiaoyan Lu Qiujun Yu Kun Lian Peilin Liu Wenjun Yan Jingyi Liu Yanzhuo Ma Yi Liu Chengxiang Li Weijie Li Wayne Bond Lau Haifeng Zhang Ling Tao 2013Basic Research in Cardiology2013,,1:1
13Rhox:A novel homeobox gene clister显示文摘Maclean JA Chen MA Wayne CM 2005Cell2005,120,3:1
14Triage during the week of the Sichuan earthquake: A review of utilized patient triage, care, and disposition procedures显示文摘Hu Nie Shi-Yuan Tang Wayne Bond Lau Jian-Cheng Zhang Yao-Wen Jiang Bernard L. Lopez Xin L. Ma Yu Cao Theodore A. Christopher 2010Injury2010,,5:1
15Comparison of traditional versus video laryngoscopy in out-of-hospital tracheal intubation显示文摘Wayne MA McDonnell M 2010Prehosp Emerg Care2010,14,2:1
16Standard cardiopulmonary resuscitation versus active compression decompression cardiopulmonary resuscitation with augmentation of negative intrathoracic pressure for out-of-hospital cardiac arrest: a randomized trial显示文摘Aufderheide TP Frascone RJ Wayne MA 2011Lancet2011,377,:1
17Intraosseous vascular access:devices,sites and rationale for IO use显示文摘Wayne MA 2007JEMS2007,32,10:1
18Use of end-tidal carbon dioxide to predict outcome in prehospital cardiac arrest 显示文摘Wayne MA Levine RL Miller CC 1995Ann Emerg Med1995,25,6:1
19End-tidal carbon dioxide and outcome of out-of-hospital cardiac arrest 显示文摘Levine RL Wayne MA Miller CC 1997N Engl J Med1997,337,5:1
20C1q/TNF-Related Proteins, A Family of Novel Adipokines, Induce Vascular Relaxation Through the Adiponectin Receptor-1/AMPK/eNOS/Nitric Oxide Signaling Pathway显示文摘Qijun Zheng Yuexing Yuan Wei Yi Wayne Bond Lau Yajing Wang Xiaoliang Wang Yang Sun Bernard L. Lopez Theodore A. Christopher Jonathan M. Peterson G. William Wong Shiqiang Yu Dinghua Yi Xin-Liang Ma 2011Arteriosclerosis Thrombosis and Vascular Biology2011,,11:1
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