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| 1 | Y chromosome microdeletions in azoospermic patients with Klinefelter's syndrome显示文摘瞄准:与 Klinefelter 的症候群(KFS ) 在 azoospermic 病人学习 Y 微删除的出现。方法:血和精液样品与 KFS 从 azoospermic 病人被收集(n = 14 ) 并且证明富饶的人的一个控制组(n = 13 ) 。精液分析根据世界卫生组织被做() 指南。血样品为 karyotyping,荧光灯的原位杂交(鱼) 和由放射性免疫测定刺激荷尔蒙(FSH ) 的血浆滤泡的测量被处理。决定 Y 微删除, 16 顺序的聚合酶链反应(PCR ) 标注了地点(圣) ,三基因(DFFRY, XKRY 和 RBM1Y ) 在孤立的 genomic DNA 上被执行。阴囊的好针渴望细胞学(FNAC ) 在选择情况中被做。结果:跨越一个 and AZFb 部位与 KFS 在 14 个 azoospermic 病人中的四个被发现的精子缺乏因素(AZF ) 的 Y 微删除。染色质组型和鱼分析表明显示出 Y 微删除的四个盒子,三个盒子有一 47, XXY/46, XY 染色体的模式和一个盒子有一 46, XY/47, XXY/48, XXXY/48, XXYY 染色体的模式。有 Y 微删除的一件样品的阴囊的 FNAC 揭示了形态学的 Sertoli 房间唯一的类型。然而,没有 Y 微删除在任何 13 个肥沃的人被观察。有 KFS 的所有病人提高了血浆 FSH 层次。结论:有 KFS 的病人可以怀有 Y 微删除并且为这些屏蔽应该是他们的诊断病情的检查的部分,特别地在那些考虑帮助了繁殖技术。 | Anurag Mitra Rima Dada Rajeev Kumar Narmada Prasad Gupta Kiran Kucheria Satish Kumar Gupta | 2006 | Asian Journal of Andrology2006，8，1： | 8 |
| 2 | Induction of Neutrophil Extracellular DNA Lattices by Placental Microparticles and IL-8 and Their Presence in Preeclampsia显示文摘 | Anurag Kumar Gupta Paul Hasler Wolfgang Holzgreve Stefan Gebhardt Sinuhe Hahn | 2006 | Human Immunology2006，，11： | 2 |
| 3 | Aplication of cytology and molecular biology in diagnosing premalignant or malignant oral lesions显示文摘 | Ravi Mehrotra Anurag Gupta Mamta singh | 2006 | Molecular Cancer2006，5，11： | 1 |
| 4 | Endophthalmitis after Pars Plana Vitrectomy显示文摘 | Allen Y.H. Hu Jean-Louis Bourges Sumit P. Shah Anurag Gupta Christine R. Gonzales Scott C.N. Oliver Steven D. Schwartz | 2009 | Ophthalmology2009，，7： | 1 |
| 5 | Frequent loss of Dab2 protein and infrequent promoter hypermethylation in breast cancer显示文摘 | Sarangadhara Appala Raju Bagadi Chandra Prakash Prasad Anurag Srivastava Rajinder Prashad Siddarth Datta Gupta Ranju Ralhan | 2007 | Breast Cancer Research and Treatment2007，，3： | 1 |
| 6 | Do Hedge Funds Have Enough Capital? A Value at Risk Approach 显示文摘 | Anurag Gupta Bing Liang | 2003 | Journal of Financial Eco- momics2003，29，： | 1 |
| 7 | Pol- yacrylamide-Based Biocompatible Nanoplatform Enhances the Tumor Uptake, PET/fluorescence Imaging and Anticancer Activity of a Chlorophyll Analog显示文摘 | Anurag Gupta lShouyan Wang Aimee Marko | 2014 | Theranostics2014，4，6： | 1 |
| 8 | Performance analysis of Time Warp with multiple homogeneous processors 显示文摘 | Anurag Gupta Ian F Akyildiz Richard M Fujimoto | 1991 | IEEE Transaction on Software Engineering (S0098-5589)1991，17，10： | 1 |
| 9 | Activated endothelial cells induce neutrophil extracellular traps and are susceptible to NETosis-mediated cell death显示文摘 | Anurag Kumar Gupta Manjunath B. Joshi Maria Philippova Paul Erne Paul Hasler Sinuhe Hahn Therese J. Resink | 2010 | FEBS Letters2010，，： | 1 |
| 10 | Neuroprotective effects of an adenoviral vector expressing the glucose transporter:a detailed description of the mediating cellular events显示文摘 | Anurag Gupta Dora Y Sapolsky | 2001 | Brain Res2001，908，1： | 1 |
| 11 | Distributed fair scheduling in a wireless LAN显示文摘 | NITIN VAIDYA ANURAG DUGAR SEEMA GUPTA | 2005 | IEEE Transactions on Mobile Computing2005，4，6： | 1 |
| 12 | Moral Hazard and Adverse Se- lection in the Originate-to-Distribute Model of Bank Credit 显示文摘 | Antic Berndt Anurag Gupta | 2009 | Journal of Monetary Economics2009，5，56： | 1 |
| 13 | Ability of microvolt T-wave alternans to modify risk assessment of ventricular tachyarrhythmic events: A meta-analysis显示文摘 | Anurag Gupta Donald D. Hoang Leah Karliner Jeffrey A. Tice Paul Heidenreich Paul J. Wang Mintu P. Turakhia | 2012 | American Heart Journal2012，，： | 1 |
| 14 | Moral hazard and adverse selection in the originate-to-distribute model of bank credit显示文摘 | Antje Berndt Anurag Gupta | 2009 | Journal of Monetary Economics2009，，5： | 1 |
| 15 | Analysis of plasma elastase levels in early and late onset preeclavapsia 显示文摘 | Anurag Kumar Gupta AE Stefan Gebhardt | 2006 | Arch Gynecol Obstet2006，273，： | 1 |
| 16 | Neuroprotective effects of an adenoviral vector expressing the glucose transporter:a detailed description of the mediating cellular events显示文摘 | Anurag Gupta Dora Y Sapolsky | 2001 | Brain Res2001，908，1： | 1 |
| 17 | 基于质量源于设计理念研究不同地区余甘子的关键性状显示文摘目的探讨质量源于设计(QbD)工具在评估印度5个不同地区余甘子地理变异中的应用。方法在本实验中,采用简化四次模型的Box-Behnken设计,运行105次,结合使用Design Expert软件进行随机响应面映射。本研究使用了三种不同的浸提方法(索氏浸提、浸渍浸提和超声浸提)以及三种溶剂[蒸馏水、甲醇和水-甲醇混合物(50:50 v/v)]。采用气相色谱-质谱法(GC-MS)测定并分析余甘子的抗氧化活性、总黄酮含量(TFC)和总酚含量(TPC),以鉴定其主要成分。结果QbD叠加图显示余甘子的提取值不低于30%w/w,2,2-二苯基-1-三硝基苯肼(DPPH)不低于60%mcg/mL(微克每毫升),TFC不低于75 mg QE/g(毫克槲皮素当量每克干原料),TPC不低于80 mg GAE/g(毫克没食子酸当量每克干原料)。此外,(GC-MS)数据证实了余甘子的生物活性存在变异。结论本研究模型能较好的描述浸提值、DPPH、TFC和TPC的变化。QbD方法可能倾向于在浸提过程中优先考虑彻底性,最终提高浸提产品的质量。 | Grishm Rohilla Priya Masand Pooja Dhama Anurag Sunil Gupta Alok Sharma | 2023 | Digital Chinese Medicine2023，6，3： | 0 |
| 18 | Loss of LAT1 sex-dependently delays recovery after caerulein-induced acute pancreatitis显示文摘BACKGROUND The expression of amino acid transporters is known to vary during acute pancreatitis(AP)except for LAT1(slc7a5),the expression of which remains stable.LAT1 supports cell growth by importing leucine and thereby stimulates mammalian target of rapamycin(mTOR)activity,a phenomenon often observed in cancer cells.The mechanisms by which LAT1 influences physiological and pathophysiological processes and affects disease progression in the pancreas are not yet known.AIM To evaluate the role of LAT1 in the development of and recovery from AP.METHODS AP was induced with caerulein(cae)injections in female and male mice expressing LAT1 or after its knockout(LAT1 Cre/LoxP).The development of the initial AP injury and its recovery were followed for seven days after cae injections by daily measuring body weight,assessing microscopical tissue architecture,mRNA and protein expression,protein synthesis,and enzyme activity levels,as well as by testing the recruitment of immune cells by FACS and ELISA.RESULTS The initial injury,evaluated by measurements of plasma amylase,lipase,and trypsin activity,as well as the gene expression of dedifferentiation markers,did not differ between the groups.However,early metabolic adaptations that support regeneration at later stages were blunted in LAT1 knockout mice.Especially in females,we observed less mTOR reactivation and dysfunctional autophagy.The later regeneration phase was clearly delayed in female LAT1 knockout mice,which did not regain normal expression of the pancreas-specific differentiation markers recombining binding protein suppressor of hairless-like protein(rbpjl)and basic helixloop-helix family member A15(mist1).Amylase mRNA and protein levels remained lower,and,strikingly,female LAT1 knockout mice presented signs of fibrosis lasting until day seven.In contrast,pancreas morphology had returned to normal in wild-type littermates.CONCLUSION LAT1 supports the regeneration of acinar cells after AP.Female mice lacking LAT1 exhibited more pronounced alterations than male mice,indicating a sexual dimorphism of amino acid metabolism. | Cristina M Hagen Eva Roth Theresia Reding Graf François Verrey Rolf Graf Anurag Gupta Giovanni Pellegrini Nadège Poncet Simone Mafalda Rodrigues Camargo | 2022 | World Journal of Gastroenterology2022，28，10： | 0 |
| 19 | Refractory hypocalcemia precipitated by dual infection with typhoid fever and hepatitis A in a patient with congenital hypoparathyroidism显示文摘We present this rare occurrence of a 17 yr old boy,a known case of congenital hypoparathyroidism, who presented with fever and jaundice for 8 days and 2 episodes of generalised tonic-clonic seizures.Premorbidly patient was on regular oral calcium supplementations with normal serum calcium levels.Investigations revealed severe hypocalcaemia(3.2 mg/dL),low 25 hydroxyvitamin D levels and hypomagnesacmia.The marked elevation of serum bilirubin was accompanied by derangement of liver enzymes.Microbiological investigations were confirmatory for both hepatitis A and typhoid fever.In spite of the aggressive management with intravenous calcium gluconate infusion,refractory hypocalcaemia persisted with recovery only after gradual decline in the bilirubin levels.We inferred that the cholestatic process produced by both acute viral hepatitis A and typhoid fever precipitated this state of refractory hypocalcaemia in the previously well preserved patient. | Suman S Karanth Ram Bhat Anurag Gupta | 2012 | Asian Pacific Journal of Tropical Medicine2012，5，8： | 0 |
| 20 | Unilateral massive hemothorax in Dengue hemorrhagic fever:A unique presentation显示文摘Dengue hemorrhagic fever is a more serious form of disease characterised by plasma leakage syndrome,thrombocytopenia and disseminated intravascular coagulation.We present a 51 year old male who presented with fever,petechiae and acute onset of breathlessness.Emergency chest rhoentogram showed a massive riglit sided pleural effusion.On insertion of intercostal drain,there was a sudden gush of blood tinged fluid suggestive of hemothorax.There was no history of trauma or bleeding tendencies.Laboratory investigations revealed a raised hematocrit and severe thrombocytopenia.Dengue IgM was surprisingly positive.After aggressive supportive management the patient gradually improved and was discharged.While bilateral pleural effusion is a known occurrence in dengue hemorrhagic fever,massive hemothorax is unheard of.We report the first case in literature ol dengue hemorrhagic fever presenting as unilateral massive hemothorax.A suspicion of dengue must also be borne in mind in cases of non-traumatic hemothorax especially in endemic areas. | Suman S Karanth Anurag Gupta Mukhyaprana Prabhu | 2012 | Asian Pacific Journal of Tropical Medicine2012，5，9： | 0 |
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