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| 1 | Importance of nutrition in inflammatory bowel disease显示文摘Inflammatory bowel disease (IBD) results from the interaction between an individual's immune response and precipitant environmental factors, which generate an anomalous chronic inflammatory response in those who are genetically predisposed. Various feeding practices have been implicated in the origin of IBD based on epidemiological observations in developed countries, but we do not have solid evidence for the etiological role played by specific food types. IBD is associated with frequent nutritional deficiencies, the pattern and severity of which depends on the extent, duration and activity of the inflammation. Nutritional support allows these deficiencies in calories, macro-and micro-nutrients to be rectified. Enteral nutrition is also a primary therapy for IBD, especially for Crohn's disease, as it allows the inflammatory activity to be controlled, kept in remission, and prevents or delays the need for surgery. Nutritional support is especially important in childhood IBD as an alternative to pharmacological treatment. This report discusses the complex relationship between diet and IBD. | Alfredo José Lucendo Livia Cristina De Rezende | 2009 | World Journal of Gastroenterology2009，15，17： | 13 |
| 2 | Anemia in inflammatory bowel disease: A neglected issue with relevant effects显示文摘Anemia,a common complication associated with inflammatory bowel disease(IBD),is frequently overlooked in the management of IBD patients.Unfortunately,it represents one of the major causes of both decreased quality of life and increased hospital admissions among this population.Anemia in IBD is pathogenically complex,with several factors contributing to its development.While iron deficiency is the most common cause,vitamin B12and folic acid deficiencies,along with the effects of pro-inflammatory cytokines,hemolysis,drug therapies,and myelosuppression,have also been identified as the underlying etiology in a number of patients.Each of these etiological factors thus needs to be identified and corrected in order to effectively manage anemia in IBD.Because the diagnosis of anemia in IBD often presents a challenge,combinations of several hematimetric and biochemical parameters should be used.Recent studies underscore the importance of determining the ferritin index and hepcidin levels in order to distinguish between iron deficiency anemia,anemia due to chronic disease,or mixed anemia in IBD patients.With regard to treatment,the newly introduced intravenous iron formulations have several advantages over orally-administerediron compounds in treating iron deficiency in IBD.In special situations,erythropoietin supplementation and biological therapies should be considered.In conclusion,the management of anemia is a complex aspect of treating IBD patients,one that significantly influences the prognosis of the disease.As a consequence,its correction should be considered a specific,first-line therapeutic goal in the management of these patients. | Danila Guagnozzi Alfredo J Lucendo | 2014 | World Journal of Gastroenterology2014，20，13： | 12 |
| 3 | 肝的 osteodystrophy : 为在长期的肝的考虑的一件重要的事疾病显示文摘 Hepatic osteodystrophy (HO) is the generic term defining the group of alterations in bone mineral metabolism found in patients with chronic liver disease. This paper is a global review of HO and its main pathophysiological, epidemiological and therapeutic aspects. Studies examining the most relevant information concerning the prevalence, etiological factors, diagnostic and therapeutic aspects involved in HO were identified by a systematic literature search of the PubMed database. HO generically defines overall alterations in bone mineral density (BMD) (osteoporosis or osteopenia) which appear as a possible complication of chronic liver disease. The origin of HO is multifactorial and its etiology and severity vary in accordance with the underlying liver disease. Its exact prevalence is unknown, but different studies estimate that it could affect from 20% to 50% of patients. The reported mean prevalence of osteoporosis ranges from 13%-60% in chronic cholestasis to 20% in chronic viral hepatitis and 55% in viral cirrhosis. Alcoholic liver disease is not always related to osteo-penia. HO has been commonly studied in chronic cholestatic disease (primary biliary cirrhosis and primary sclerosing cholangitis). Several risk factors and pathogenic mechanisms have been associated with the loss of BMD in patients with chronic liver disease. However, little information has been discovered in relationship to most of these mechanisms. Screening for osteopenia and osteoporosis is recommended in advanced chronic liver disease. There is a lack of randomized studies assessing specific management for HO. | Germán López-Larramona Alfredo J Lucendo Sonia González-Castillo José M Tenias | 2011 | World Journal of Hepatology2011，3，12： | 9 |
| 4 | Colorectal cancer surveillance in patients with inflammatory bowel disease: What's new?显示文摘Several studies assessing the incidence of colorectal cancer (CRC) in inflammatory bowel disease (IBD) patients have found an increased risk globally estimated to be 2 to 5 times higher than for the general population of the same age group. The real magnitude of this risk, however, is still open to debate. Research is currently being carried out on several risk and protective factors for CRC that have recently been identified in IBD patients. A deeper understanding of these factors could help stratify patient risk and aid specialists in choosing which surveillance program is most efficient. There are several guidelines for choosing the correct surveillance program for IBD patients; many present common characteristics with various distinctions. Current recommendations are far from perfect and have important limitations such as the fact that their efficiency has not been demonstrated through randomized controlled trials, the limited number of biopsies performed in daily endoscopic practice, and the difficulty in establishing the correct time to begin a given surveillance program and maintain a schedule of surveillance. That being said, new endoscopic technologies should help by replacing random biopsy protocols with targeted biopsies in IBD patients, thereby improving the efficiency of surveillance programs.However, further studies are needed to evaluate the cost-effectiveness of introducing these techniques into daily endoscopic practice. | Danila Guagnozzi Alfredo J Lucendo | 2012 | World Journal of Gastrointestinal Endoscopy2012，4，4： | 6 |
| 5 | Small bowel video capsule endoscopy in Crohn’s disease: What have we learned in the last ten years?显示文摘Since its introduction in 2001, capsule endoscopy (CE) has become the most important advance in the study of small bowel disease, including Crohn's disease (CD). This technique has been demonstrated to be superior to all other current forms of radiological investigation in detecting mucosal abnormalities of small bowel nonstricturing CD. CE has proven to be extremely useful in diagnosing CD in patients with inconclusive findings from ileocolonoscopy and x-ray-based studies. Almost half of all patients with CD involving the ileum also present lesions in proximal intestinal segments, with the small bowel being exclusively involved in up to 30% of all CD cases. Despite the widespread use of CE, several questions concerning the utility of this technique remain unanswered. The lack of commonly agreed diagnostic criteria for defining CD lesions with the aid of CE may have had an influence on the variation in diagnostic re sults for CE reported in the literature. The utility of CE in monitoring CD and in guiding therapy has also been proposed. Furthermore, CE could be a useful second-line technique for patients with an established diagn osis of CD and unexplained symptoms. Finally, as no thres hold for CD diagnosis has been agreed upon, a se verity scale of mucosal disease activity has notbeen universally followed. None of the available activity indexes based on CE findings has been independently validated. This article discusses several cutting-edge aspects of the usefulness of CE in CD 10 years after its introduction as a sensible method to study the small intestine. | Alfredo J Lucendo Danila Guagnozzi | 2011 | World Journal of Gastrointestinal Endoscopy2011，3，2： | 4 |
| 6 | Eosinophilic Esophagitis in Adults is Associated with IgG4 and Not Mediated by IgE显示文摘 | Frederic Clayton John C. Fang Gerald J. Gleich Alfredo J. Lucendo Jose M. Olalla Laura A. Vinson Amy Lowichik Xinjian Chen Lyska Emerson Kristen Cox Molly A. O’Gorman Kathryn A. Peterson | 2014 | Gastroenterology2014，，： | 3 |
| 7 | Eosinophilic esophagitis: Updated consensus recommendations for children and adults显示文摘 | Chris A. Liacouras Glenn T. Furuta Ikuo Hirano Dan Atkins Stephen E. Attwood Peter A. Bonis A. Wesley Burks Mirna Chehade Margaret H. Collins Evan S. Dellon Ranjan Dohil Gary W. Falk Nirmala Gonsalves Sandeep K. Gupta David A. Katzka Alfredo J. Lucendo Jo | 2011 | The Journal of Allergy and Clinical Immunology2011，，1： | 3 |
| 8 | Subepithelial collagen deposition, profibrogenic cytokine gene expression, and changes after prolonged fluticasone propionate treatment in adult eosinophilic esophagitis: A prospective study显示文摘 | Alfredo J. Lucendo ángel Arias Livia C. De Rezende Jose Luis Yagüe-Compadre Teresa Mota-Huertas Sonia González-Castillo Rubén A. Cuesta José M. Tenias Teresa Bellón | 2011 | The Journal of Allergy and Clinical Immunology2011，，： | 2 |
| 9 | Efficacy of Dietary Interventions in Inducing Histologic Remission in Patients with Eosinophilic Esophagitis: a Systematic Review and Meta-analysis显示文摘 | ángel Arias Jesús González-Cervera José M. Tenias Alfredo J. Lucendo | 2014 | Gastroenterology2014，，： | 2 |
| 10 | Four-food group elimination diet for adult eosinophilic esophagitis: A prospective multicenter study显示文摘 | Javier Molina-Infante Angel Arias Jesus Barrio Joaquín Rodríguez-Sánchez Marta Sanchez-Cazalilla Alfredo J. Lucendo | 2014 | The Journal of Allergy and Clinical Immunology2014，，： | 2 |
| 11 | Tracheal aspiration of a capsule endoscope: a new case report and literature compilation of an increasingly repor- ted complication显示文摘 | Lucendo AJ Gonzdlez-Castillo S Ferndndez-Fuente M | 2011 | Dig Dis Sci2011，56，9： | 1 |
| 12 | Eosinophilic esophagitis: Updated consensus recommendations for children and adults显示文摘 | Chris A. Liacouras Glenn T. Furuta Ikuo Hirano Dan Atkins Stephen E. Attwood Peter A. Bonis A. Wesley Burks Mirna Chehade Margaret H. Collins Evan S. Dellon Ranjan Dohil Gary W. Falk Nirmala Gonsalves Sandeep K. Gupta David A. Katzka Alfredo J. Lucendo Jo | 2011 | The Journal of Allergy and Clinical Immunology2011，，1： | 1 |
| 13 | Nonanesthesiologist- administered propofol sedation for colonoscopy issafeandeffective: a prospectiveSpanish studyover 1000 consecutiveexams显示文摘 | Lucendo A J Olveira A Friqina-Ruiz A B | 2012 | Eur J Gastroenterol Hepatol2012，24，7： | 1 |
| 14 | Montelukast was inefficient in maintaining steroid-induced remission in adult eosinophilic esophagitis 显示文摘 | Lucendo AJ De Rezende LC Jimenez-Contreras S | 2011 | Dig Dis Sci2011，56，12： | 1 |
| 15 | Montelukast was inefficient in maintaining steroid-induced re- mission in adult eosinophilic esophagitis显示文摘 | Lucendo AJ De Rezende LC Jimenez-Contreras S | 2011 | Dig Dis Sci2011，56，12： | 1 |
| 16 | The rela- tionship between proximal and distal colonic adenomas : is screening sigmoidoscopy enough in the presence of a changing epidemiology 显示文摘 | Lucendo AJ Guagnozzi D Angueira T | 2013 | Eur J Gastroenterol Hepatol2013，25，8： | 1 |
| 17 | Eosinophilic esophagitis - clinical manifestations, diagnosis, and treatment显示文摘 | Lucendo Villartn AJ | 2009 | Rev Enferm Dig2009，101，1： | 1 |
| 18 | Gran-ular cell tumors of the esophagus:report of five cases andreview of diagnostic and therapeutic techniques显示文摘 | De Rezende L Lucendo AJ Alvarez-Argüelles H | 2007 | DisEsophagus2007，，5： | 1 |
| 19 | Eosinophilic esophagitis In adults,an emergency cause of dysphagia:Description of 9 cases显示文摘 | Lucendo Villarin A J Carrion Alonso G Navarro Sanchez M | 2005 | Rev Esp Enferm Dig2005，97，4： | 1 |
| 20 | Eosinophilic gastroenteritis: ~m update显示文摘 | Lucendo A J Arias A | 2012 | Expert Rev Gastroenterol HeDatok2012，6，5： | 1 |
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