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| 1 | Cerebral microbleeds are associated with worse cognitive function: The Rotterdam Scan Study显示文摘 | M.M.F. Poels M.A. Ikram A. van der Lugt A. Hofman W.J. Niessen G.P. Krestin M.M.B. Breteler M.W. Vernooij | 2012 | Neurology2012，，5： | 4 |
| 2 | Systemic and Hepatic Hemodynamics in Hepatosplenic Manson’s Schistosomiasis with and Without Propranolol显示文摘 | Sergio Mies Oddone Braghirolli Neto Andre Beer Carlos E.S. Baia Fernando Alfieri Leila M.M.B. Pereira Marcelo J.A. Sette Silvano Raia | 1997 | Digestive Diseases and Sciences1997，，4： | 1 |
| 3 | PARK6 is a common cause of familial parkinsonism显示文摘 | E.M. Valente F. Brancati V. Caputo E.A. Graham M.B. Davis A. Ferraris M.M.B. Breteler T. Gasser V. Bonifati A.R. Bentivoglio G. De Michele A. Dürr P. Cortelli A. Filla G. Meco B.A. Oostra A. Brice A. Albanese B. Dallapiccola N.W. Wood | 2002 | Neurological Sciences2002，，2： | 1 |
| 4 | White matter atrophy and lesion formation explain the loss of structural integrity of white matter in aging显示文摘 | M.W. Vernooij M. de Groot A. van der Lugt M.A. Ikram G.P. Krestin A. Hofman W.J. Niessen M.M.B. Breteler | 2008 | Neuroimage2008，，： | 1 |
| 5 | Insulin metabolism and the risk of Alzheimer disease: The Rotterdam Study显示文摘 | E.M.C. Schrijvers J.C.M. Witteman E.J.G. Sijbrands A. Hofman P.J. Koudstaal M.M.B. Breteler | 2010 | Neurology2010，，22： | 1 |
| 6 | 脑白质损害与痴呆的发病风险 | Prins N.D. Van Dijk E.J. Den Heijer T. M.M.B. Breteler 黄卫东 | 2005 | 世界核心医学期刊文摘(神经病学分册)2005，0，3： | 0 |
| 7 | 血压、白质损伤和颞叶内侧萎缩之间的联系 | Den Heijer T. Launer L.J. Prins N.D. M.M.B. Breteler 邓剑平 | 2005 | 世界核心医学期刊文摘(神经病学分册)2005，0，6： | 0 |
| 8 | 普通人群中帕金森综合征和帕金森病的发病率:鹿特丹研究 | De Lau L.M.L. Giesbergen P.C.L.M. De RijkM.C. M.M.B. Breteler 姚庆和 | 2005 | 世界核心医学期刊文摘(神经病学分册)2005，0，3： | 0 |
| 9 | 脂蛋白相关磷脂酶A2水平与痴呆风险相关显示文摘Objective: High levels of the inflammatory marker lipoproteinassociated phospholipase A2 (Lp-PLA2) have been proposed to be a predictor of coronary heart disease and stroke. Because both inflammation and vascular disease are associated with dementia, the objective of the present study was to examine the association between Lp-PLA2 and the risk of dementia. Methods: Within the Rotterdam Study, a population-based prospective cohort study, we performed a case-cohort study. Of the 6,713 participants at risk for dementia, a random sample of 1,742 individuals was drawn. During follow-up (mean, 5.7 years), 302 incident dementia cases were identified. Cox proportional hazard models were used to estimate the association of Lp-PLA2 and dementia. Results: We found that subjects with higher levels of Lp-PLA2 had an increased risk of dementia. Compared with the first quartile of Lp-PLA2, ageand sex-adjusted hazard ratios (HRs; 95% confidence interval [CI]) for dementia for the second, third, and fourth quartiles were 1.19 (0.78- 1.81), 1.15 (0.74- 1.79), and 1.56 (1.03- 2.37), respectively (p value for trend 0.04). Additional adjustment for cardiovascular and inflammatory factors did not change the estimates. Interpretation: This is the first study to our knowledge that shows that Lp-PLA2 is associated with the risk of dementia independent of cardiovascular and inflammatory factors and provides evidence for a potential role of Lp-PLA2 in identifying subjects at risk for dementia. | Van Oijen M Van Der Meer I.M Hofman A M.M.B. Breteler 周永 | 2006 | 世界核心医学期刊文摘(神经病学分册)2006，2，5： | 0 |
| 10 | 鹿特丹地区膳食脂肪酸和帕金森病发病风险关系的研究 | De Lau L.M.L. Bornebroek M. Witteman J.C.M. M.M.B. Breteler 史利利 | 2005 | 世界核心医学期刊文摘(神经病学分册)2005，0，10： | 0 |
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