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| 1 | Microstructure and Mechanical Properties of 06Cr13Ni4Mo Steel Treated by Quenching–Tempering–Partitioning Process显示文摘A heat treatment process,quenching-tempering-partitioning(Q-T-P),has been applied to a low carbon martensitic stainless steel 06Cr13Ni4 Mo.By using this process,ultrafine reversed austenite can be obtained at room temperature.The microstructures of the reversed austenite and the martensite matrix were characterized by transmission electron microscopy(TEM) and energy dispersive spectroscopy(EDS)in detail.The results show that the ultrafine reversed austenite is enriched in Ni resulting in the austenite stability at room temperature.Two new types of nano-scale carbide precipitates are found in the martensite matrix.Detailed analysis suggests that the two nano-scale precipitates can be identified asω phase and λ phase carbides,respectively.The orientation relationship between the ω phase and matrix is[011]α//[2110]ω and(211)α//(0110)ω,while that between the λ phase precipitate and matrix is[011]α//[0001]λ and(200)α//(1210)λ.For the present steel,the ultrafine reversed austenite and carbide precipitates obtained by Q-T-P treatment provide a good combination of high strength and toughness. | Yuanyuan Song Jingping Cui Lijian Rong | 2016 | Journal of Materials Science & Technology2016，32，2： | 8 |
| 2 | Optimal Reinsurance Under Distortion Risk Measures and Expected Value Premium Principle for Reinsurer显示文摘This paper discusses optimal reinsurance strategy by minimizing insurer's risk under one general risk measure:Distortion risk measure.The authors assume that the reinsurance premium is determined by the expected value premium principle and the retained loss of the insurer is an increasing function of the initial loss.An explicit solution of the insurer's optimal reinsurance problem is obtained.The optimal strategies for some special distortion risk measures,such as value-at-risk(VaR) and tail value-at-risk(TVaR),are also investigated. | ZHENG Yanting CUI Wei YANG Jingping | 2015 | Journal of Systems Science & Complexity2015，28，1： | 3 |
| 3 | Revisiting mesoscale eddy genesis mechanism of nonlinear advection in a marginal ice zone显示文摘A three-dimensional(3-D) ocean model is coupled with a two-dimensional(2-D) sea ice model, to revisit a nonlinear advection mechanism, one of the most important mesoscale eddy genesis mechanisms in the marginal ice zone. Two-dimensional ocean model simulations suggest nonlinear advection mechanism is more important when the water gets shallower. Instead of considering the ocean as barotropic fluid in the 2-D ocean model, the 3-D ocean model allows the sea ice to affect the current directly in the surface layer via ocean-ice interaction. It is found that both mesoscale eddy and sea surface elevation are sensitive to changes in a water depth in the 3-D simulations. The vertical profile of a current velocity in 3-D experiments suggests that when the water depth gets shallower, the current move faster in each layer, which makes the sea surface elevation be nearly inverse proportional to the water depth with the same wind forcing during the same time. It is also found that because of the vertical motion, the magnitude of variations in the sea surface elevation in the 3-D simulations is very small,being only 1% of the change in the 2-D simulations. And it seems the vertical motion to be the essential reason for the differences between the 3-D and 2-D experiments. | DAI Haijin CUI Jian YU Jingping | 2017 | Acta Oceanologica Sinica2017，36，11： | 2 |
| 4 | LncRNA MIR31HG controls the proliferation and metastasis of gastric cancer by c-CBL-mediated degradation of β-catenin显示文摘LncMIR31HG acts as a host gene for miR-31,also known as LncHIFCAR(long non-coding HIF-1 co-activating RNA),whose deregulation has been reported to promote the development of various human cancers,including lung cancer,colorectal cancer,etc.1,2 However,the biological functions and molecular mechanisms of MIR31HG in gastric cancer are unclear. | Wen Peng Jiayi Zhang Shenghao Wang Feng Wang a Kun Wang Rui Geng Xiangfei Ding Jingping Zhang Biao Li Xiaoxue Ke Muhan Lü Hongjuan Cui | 2023 | Genes & Diseases2023，10，3： | 1 |
| 5 | A novel long-term intravenous combined with local treatment with human amnion-derived mesenchymal stem cells for a multidisciplinary rescued uremic calciphylaxis patient and the underlying mechanism显示文摘Calciphylaxis is a rare disease characterized histologically by microvessel calcification and microthrombosis,with high mortality and no proven therapy.Here,we reported a severe uremic calciphylaxis patient with progressive skin ischemia,large areas of painful malodorous ulcers,and mummified legs.Because of the worsening symptoms and signs refractory to conventional therapies,treatment with human amnion-derived mesenchymal stem cells(hAMSCs)was approved.Preclinical release inspections of hAMSCs,efficacy,and safety assessment,including cytokine secretory ability,immunocompetence,tumorigenicity,and genetics analysis in vitro,were introduced.We further performed acute and long-term hAMSC toxicity evaluations in C57BL/6 mice and rats,abnormal immune response tests in C57BL/6 mice,and tumorigenicity tests in neonatal Balbc-nu nude mice.After the preclinical research,the patient was treated with hAMSCs by intravenous and local intramuscular injection and external supernatant application to the ulcers.When followed up to 15 months,the blood-based markers of bone and mineral metabolism improved,with skin soft tissue regeneration and a more favorable profile of peripheral blood mononuclear cells.Skin biopsy after 1-month treatment showed vascular regeneration with mature noncalcified vessels within the dermis,and 20 months later,the re-epithelialization restored the integrity of the damaged site.No infusion or local treatment-related adverse events occurred.Thus,this novel long-term intravenous combined with local treatment with hAMSCs warrants further investigation as a potential regenerative treatment for uremic calciphylaxis due to effects of inhibiting vascular calcification,stimulating angiogenesis and myogenesis,anti-inflammatory and immune modulation,multidifferentiation,re-epithelialization,and restoration of integrity. | Lianju Qin Jing Zhang Yujie Xiao Kang Liu Yugui Cui Fangyan Xu Wenkai Ren Yanggang Yuan Chunyan Jiang Song Ning Xiaoxue Ye Ming Zeng Hanyang Qian Anning Bian Fan Li Guang Yang Shaowen Tang Zhihong Zhang Juncheng Dai Jing Guo Qiang Wang Bin Sun Yifei Ge Chun Ouyang Xueqiang Xu Jing Wang Yaoyu Huang Hongqing Cui Jing Zhou Meilian Wang Zhonglan Su Yan Lu Di Wu Jingping Shi Wei Liu Li Dong Yinbing Pan Baiqiao Zhao Ying Cui Xueyan Gao Zhanhui Gao Xiang Ma Aiqin Chen Jie Wang Meng Cao Qian Cui Li Chen Feng Chen Youjia Yu Qiang Ji Zhiwei Zhang Mufeng Gu Xiaojun Zhuang Xiaolin Lv Hui Wang Yanyan Pan Ling Wang Xianrong Xu Jing Zhao Xiuqin Wang Cuiping Liu Ningxia Liang Changying Xing Jiayin Liu Ningning Wang | 2022 | Journal of Molecular Cell Biology2022，14，2： | 1 |
| 6 | Advances in Biomarkers for Multiple Myeloma显示文摘Multiple myeloma(MM)is the second most common malignancy in hematology.MM is characterized by the malignant proliferation of plasma cells in the bone marrow,accompanied by the secretion of monoclonal immunoglobulin,mainly occurring in the elderly.The clinical manifestations of MM include renal dysfunction,bone destruction,infection,anemia,hemorrhage,hypercalcemia,and hyperviscosity syndrome.The recent discovery of biomarkers related to the diagnosis or prognosis of MM provides an important basis for the diagnosis and treatment of MM.This paper reviews the research progress of biomarkers expressed in tissues and peripheral blood at home and abroad. | Chao Yang Ji Qi Tianhong Jia Jingping Ran Wenxuan Cui | 2023 | Proceedings of Anticancer Research2023，7，1： | 0 |
| 7 | DNA damage chemotherapeutic drugs suppress basal-like breast cancer growth by down-regulating the transcription of the FOXO1-KLF5 axis显示文摘Most basal-like breast cancers(BLBCs)have a poor prognosis and high crossover with triple-negative breast cancers(TNBCs).However,approximately 50%of TNBC patients develop chemoresistance.^(1) Dexamethasone reportedly induces Krüppel-like factor 5(KLF5)and can cause docetaxel and cisplatin resistance in TNBC.A super-enhancer can maintain the transcription of KLF5. | Qiuxia Cui Jian Sun Jingping Yuan Juanjuan Li Chuanyu Yang Guangshi Du Chengang Zhou Pu Qiu Jidong Gao Yuanqi Zhang Dewei Jiang Ceshi Chen | 2024 | Genes & Diseases2024，11，1： | 0 |
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