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| 1 | 欧洲新生儿呼吸窘迫综合征防治指南-2010版显示文摘呼吸窘迫综合征(RDS)是由于肺表面活性物质(PS)缺乏及肺结构发育不成熟所致,多见于早产儿,自然病程为生后当时或很快发病,并在生后2d内进行性恶化,如不及时治疗,因进行性缺氧和呼吸衰竭而死亡,存活者,在生后2—4d病情开始改善。胎龄越小,RDS发生率越高,2006年EuroNeoStat的数据显示:胎龄23~25周的早产儿RDS发生率为91%, | Sweet DG Carnielli V Greisen G Hallman M Ozek E Plavka R Saugstad OD Simeoni U Speer CP Halliday HL 袁琳(翻译) 陈超(审校) | 2011 | 中华儿科杂志2011，49，1： | 137 |
| 2 | 欧洲新生儿呼吸窘迫综合征防治共识指南:2016版显示文摘新生儿呼吸窘迫综合征(RDS)是早产儿的重要疾病,尽管治疗手段不断成熟,低龄早产儿存活率逐渐增高,但支气管肺发育不良(BPD)发病率也随之升高,其中部分原因是减少了出生后激素的使用.2006年以来,来自欧洲许多国家的新生儿专家每3年一次回顾最新文献,就RDS或有RDS风险早产儿的防治达成共识,立志于改善欧洲新生儿的预后.欧洲RDS防治指南2007年开始发布,2010年和2013年进行了更新,期间获得了欧洲围产医学学会的大力支持.RDS是由于肺表面活性物质(PS)缺乏导致生后早期出现呼吸窘迫,典型临床表现随着防治手段的改进发生了巨大变化. | DavidG.Sweet Virgilio Carnielli Gorm Greisen Mikko Hallman Eren Ozek Richard Plavka Ola Didrik Saugstad Umberto Simeoni Christian P. Speer Maximo Vento Gerard H. A. Visser Henry L. Halliday | 2017 | 中华儿科杂志2017，55，3： | 90 |
| 3 | 欧洲早产儿呼吸窘迫综合征防治共识指南(2013版)显示文摘本次指南更新包括了自2010年以来Cochrane系统评价和医学文献中的最新证据,并采纳了一种新的证据评估分级系统(GRADE)。以往关于早期肺表面活性物质和持续气道正压通气治疗的推荐目前拥有了更坚定的循证依据。对产房复苏、稳定生命体征等内容进行了较大扩充。生命体征稳定后的氧疗目前仍存在一定的争议,但是在获得更多的证据之前,血氧饱和度的目标范围不应低于90%,支持治疗对于超早早产儿仍然极为重要。指南对其他注意事项进行了缩减。 | Sweet D Carnielli V Greisen G Hallman M Ozek E Plavka R Saugstad OD Simeoni U Speer CP Halliday HL 王敏婕 袁琳 陈超 | 2014 | 中华儿科杂志2014，52，10： | 34 |
| 4 | 欧洲新生儿呼吸窘迫综合征防治共识显示文摘呼吸窘迫综合征(RDS)是由于肺表面活性物质(PS)缺乏及肺结构发育不成熟所致,多见于早产儿,自然病程为出生当时或生后很快发病,并在生后2d内进行性恶化,如不及时治疗,因进行性缺氧和呼吸衰竭而死亡。存活者出生后2—4d病情开始改善。RDS临床表现早期出现呼吸窘迫如紫绀、呻吟、吸凹和呼吸急促,然后进一步发展为呼吸衰竭,血气分析结果可提示呼吸衰竭的严重性。胸部X线典型的毛玻璃样改变和支气管充气征可以确诊。Vermont Oxford新生儿协作网对RDS的定义为: | Sweet D Bevilacqua G Carnielli V Greisen G Plavka R Didrik Saugstad O Simeoni U Speer CP Valls-I-Soler A Halliday H 袁琳(译) 陈超(译) 杜立中(校) | 2008 | 中华儿科杂志2008，46，1： | 17 |
| 5 | ICSI治疗男科不育对后代的影响显示文摘采用睾丸组织中分离到单精子进行卵胞浆内单精子注射(ICSI)治疗梗阻性和非梗阻性无精子症患者,或使用射出的精子治疗弱精子患者的方法一出现就引起人们的关注,人们担心,与传统的体外受精和自然受精相比,该技术可能对后代造成不利影响。ICSI适用于体外受精法不能治疗的男性不育,同时,总的说来也没有表现出任何比体外受精不利的影响。然而,目前关于用ICSI治疗男性不育结果的研究还很少,或者说研究的样本还远远不足以说明问题。从目前ICSI疗法中有限的精子来源和不育症病因信息来看,ICSI似乎没有增加后代先天畸形的风险,但是,新生和遗传染色体异常几率略有增加。关于生长和神经发育方面的相关研究也很少,仅有个别研究结果表明父亲患有不育症对孩子的生长和神经发育没有任何的不良影响。用于ICSI的精子来源对后代的早期生活没有很大影响,但是对于隔代和表观遗传方面的影响还有待研究。如果男性不育主要是由Y染色体缺失引起,那么要选择合适的时间将这一信息告知他的男性后代,以确保他的生殖健康和生育计划能够得以优化。 | Jane Halliday | 2012 | Asian Journal of Andrology2012，14，1： | 11 |
| 6 | 额颞叶痴呆显示文摘一、前言
额颞叶痴呆(frontotemporal dementia,FFD)是一组临床痴呆症候群,由前额叶和颞叶变性引起,其病理表现与阿尔茨海默病(AD)不同,病理上称之为额颞叶变性(frontotemporal lobe degeneration,FTLD)。以往临床上也称为皮克病(Pick disease)。 | 黄越 Glenda Halliday 陈生弟 John Hodges | 2008 | 中华神经科杂志2008，41，12： | 4 |
| 7 | DNA extraction from fresh-frozen and formalin-fixed, paraffinembedded human brain tissue显示文摘Both fresh-frozen and formalin-fixed,paraffinembedded(FFPE)human brain tissues are invaluable resources for molecular genetic studies of central nervous system diseases,especially neurodegenerative disorders.To identify the optimal method for DNA extraction from human brain tissue,we compared methods on differently-processed tissues.Fragments of LRRK2 and MAPT(257 bp and 483 bp/245 bp)were amplified for evaluation.We found that for FFPE samples,the success rate of DNA extraction was greater when using a commercial kit than a laboratory-based method(successful DNA extraction from 76%versus 33%of samples).PCR amplicon size and storage period were key factors influencing the success rate of DNA extraction from FFPE samples.In the fresh-frozen samples,the DNA extraction success rate was 100%using either a commercial kit(QIAamp DNA Micro)or a laboratorybased method(sample boiling in 0.1 mol/L NaOH,followed by proteinase K digestion,and then DNA extraction using Chelex-100)regardless of PCR amplicon length or tissue storage time.Although the present results demonstrate that PCR-amplifiable genomic DNA can be extracted from both fresh-frozen and FFPE samples,fresh brain tissue is recommended for DNA extraction in future neuropathological studies. | Jian-Hua Wang Amany Gouda-Vossos Nicolas Dzamko Glenda Halliday Yue Huang | 2013 | Neuroscience Bulletin2013，29，5： | 3 |
| 8 | European Consensus Guidelines on the Management of Neonatal Respiratory Distress Syndrome in Preterm Infants - 2013 Update显示文摘 | Sweet David G Carnielli Virgilio Greisen Gorm Hallman Mikko Ozek Eren Plavka Richard Saugstad Ola D Simeoni Umberto Speer Christian P Vento Maximo Halliday Henry L | 2013 | Neonatology2013，，4： | 3 |
| 9 | Osteopontin-CD44 Signaling in the Glioma Perivascular Niche Enhances Cancer Stem Cell Phenotypes and Promotes Aggressive Tumor Growth显示文摘 | Alexander Pietras Amanda M. Katz Elin J. Ekstr?m Boyoung Wee John J. Halliday Kenneth L. Pitter Jillian L. Werbeck Nduka M. Amankulor Jason T. Huse Eric C. Holland | 2014 | Cell Stem Cell2014，，3： | 3 |
| 10 | Can we clinically diagnose dementia with Lewy bodies yet?显示文摘Dementia with Lewy Bodies(DLB)was initially identified and confirmed primarily by pathology,but is soon to be incorporated into the Diagnostic and Statistical Manual criteria as a clinical disease entity.Despite these advances over more than 20 years,current data suggest that the sensitivity of accurate clinical diagnosis of DLB is still very low,although there is mounting evidence that supportive features may increase diagnostic accuracy.Although DLB remains easy to identify pathologically with different cellular pathologies differentiating it from other dementia syndromes,pathological identification using only Lewy body pathology has been shown to be inaccurate due to overlap with patients without dementia symptoms.A number of studies now suggest that a combination of cellular pathologies,which include α-synuclein and β-amyloid deposition as well as dopamine denervation,assist with differentiating this dementia syndrome from others.The clinical and pathological overlap with the tauopathy of Alzheimer’s disease still remains to be clarified.To determine more robust and independent clinicopathological correlates from Alzheimer’s disease,longitudinal prospective studies,using specific clinical batteries on dementia patients reaching the proposed criteria for DLB,with post-mortem assessment of the multiple pathologies associated with dementia,are required.Identifying genetic causes for DLB is another approach to investigate the pathogenesis of DLB.However this approach has been hindered to date by difficulties with identifying DLB clinically.The use of novel techniques is likely to advance knowledge on the pathogenesis of DLB and assist with redefining clinical and pathologic diagnostic criteria.To achieve the goal of more accurate clinical diagnosis of DLB,breakthroughs are necessary on the pathogenesis of DLB. | Yue Huang Glenda Halliday | 2013 | Translational Neurodegeneration2013，2，1： | 2 |
| 11 | 非典型性帕金森样病显示文摘非典型性帕金森样病亦称帕金森综合征,为一组临床表现多样的疾病症候群,除了覆盖原发性帕金森病(PD)的主要临床症状[静止性震颤、肌强直、运动不能和(或)运动迟缓、姿势反射障碍外,还具有进展迅速、对左旋多巴反应不佳或其他特征性表现,如疾病早期易跌倒。 | 黄越 陈生弟 Gregor Wenning Lawrence Golbe 王少石 王拥军 赵钢 吕佩源 陈彪 Jun Yu Christine Song Andrew Lees Irene Litvan 王晓民 Glenda Halliday | 2011 | 中国现代神经疾病杂志2011，11，1： | 2 |
| 12 | Gelatinase A activation is regulated by the organization of the polymerized actin cytoskeleton显示文摘 | Tomasek JJ Halliday NL Updike DL | 1997 | J BiolChem1997，272，： | 2 |
| 13 | Loss of brainstem serotonin and substance P-containing neurons in Parkinson's disease 显示文摘 | Halliday GM Blumbergs PC Cotton RG | 1990 | Brain Res1990，510，1： | 1 |
| 14 | Rb-Sr dating of sphalerites from Tenessee and the genesis of Mississippi Valley-Type(MVT) ore deposits显示文摘 | Nakai S Halliday A N Kesler S E Jones H D | 1990 | Nature1990，346，： | 1 |
| 15 | Recent developments in inductively coupled plasma magnetic sector multiple collector mass spectrometry 显示文摘 | Halliday A N Lee D C Christensen J N Walder A J Freedman P A Jones C E Hall C M Yi W Teagle D | 1995 | Int J Mass Spec Ion Proc1995，，： | 1 |
| 16 | MR diffusion weighted imaging in a case of West Nile virus encephalitis显示文摘 | Agid R Duereux D Halliday WC | 2003 | Neurology2003，61，12： | 1 |
| 17 | Precarved tyophilized tissue for lamellar keratoplasty in recurrent pterygiuml显示文摘 | Busin M Halliday BL Arffa RC | 1986 | Am J Opht halmol1986，102，5： | 1 |
| 18 | Management of bronchopulmonary dysplasia in infants:guidelines for corticosteroid use显示文摘 | Grier DG Halliday HL | 2005 | Drugs2005，65，1： | 1 |
| 19 | Do poly- morphisms in the familial Parkinsonism genes contribute to risk for sporadic Parkinson's disease? 显示文摘 | Sutherland GT Halliday GM Silburn PA | 2009 | Mov Disord2009，24，6： | 1 |
| 20 | Core formation on Mars and differentiated asteroids 显示文摘 | Lee D C Halliday A N | 1997 | Nature1997，388，： | 1 |
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