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13篇 您的检索式：作者名="Groschner"

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		题名	作者	年代	出处	被引量
	1	Mitochondrial Ca2+ uptake 1(MICU1) and mitochondrial Ca2+ uniporter(MCU) contribute to metabolism-secretion coupling in clonal pancreatic β-cells显示文摘	Alam MR Groschner LN Parichatikanond W		0，，41：	1
	2	Protein kinase c mediates dual modulationof L-type Ca2+channels in human vascular smooth muscle显示文摘	Schuhmann K Groschner K	1994	FEBSLett1994，341，：	1
	3	显示文摘	Teubl M Groschner K Kohlwein SD	1999	J Biol C hem1999，274，29：	1
	4	Identification of a rare subset of adipose tissue-resident progenitor cells, which express CD133 and TRPC3 as a VEGF-regulated Ca 2+ entry channel显示文摘	Michael Poteser Annarita Graziani Petra Eder Ameli Yates Heinrich M?chler Christoph Romanin Klaus Groschner	2008	FEBS Letters2008，，18：	1
	5	Role of K ATP Channels in Glucose-Regulated Glucagon Secretion and Impaired Counterregulation in Type 2 Diabetes显示文摘	Quan Zhang Reshma Ramracheya Carolina Lahmann Andrei Tarasov Martin Bengtsson Orit Braha Matthias Braun Melissa Brereton Stephan Collins Juris Galvanovskis Alejandro Gonzalez Lukas N. Groschner Nils J.G. Rorsman Albert Salehi Mary E. Travers Jonathan N. W	2013	Cell Metabolism2013，，6：	1
	6	Two ways to feel the pressure: an endothelial Ca^2+ entry channel with dual mechanosensitivity 显示文摘	Groschner K	2002	Cardiovasc Res2002，53，：	1
	7	Intracellular alkalinization augments α 1 -adrenoceptor-mediated vasoconstriction by promotion of Ca 2+ entry through the non-L-type Ca 2+ channels显示文摘	Ichiro Wakabayashi Hidehisa Masui Klaus Groschner	2001	European Journal of Pharmacology2001，，2：	1
	8	Trp proteins form store-operated cation channels in human vascular endothelial cells显示文摘	Groschner K Hingel S Lintschinger B		0，，1：	1
	9	Evidence for a role of Trp proteins in the oxidative stress-induced membrane conductances of porcine aortic endothelial cells显示文摘	Balzer M Lintschinger B Groschner K		0，，02：	1
	10	The endocannabi- noid N-arachidonoyl glycine (NAGIy) inhibits store-operated Ca^2 + entry by preventing STIM1-Orail interaction 显示文摘	Deak AT Groschner LN Alam MR	2013	J Cell Sei2013，126，4：	1
	11	Optopharmacological control of TRPC channels by coumarin-caged lipids is associated with a phototoxic membrane effect显示文摘Photouncaging of second messengers has been successfully employed to gain mechanistic insight of cellular signaling pathways. One of the most enigmatic processes of ion channel regulation is lipid recognition and lipid-gating of TRPC channels, which represents pivotal mechanisms of cellular Ca^(2+) homeostasis. Recently, optopharmacological tools including caged lipid mediators became available, enabling an unprecedented level of temporal and spatial control of the activating lipid species within a cellular environment. Here we tested a commonly used caged ligand approach for suitability to investigate TRPC signaling at the level of membrane conductance and cellular Ca^(2+) handling. We report a specific photouncaging artifact that is triggered by the cage structure coumarin at UV illumination. Electrophysiological characterization identified a light-dependent membrane effect of coumarin. UV light(340 nm) as used for photouncaging, initiated a membrane conductance specifically in the presence of coumarin as low as 30 μmol L^(-1) concentrations. This conductance masked the TRPC3 conductance evoked by photouncaging, while TRPC-mediated cellular Ca^(2+) responses were largely preserved. The observed light-induced membrane effects of the released caging moiety may well interfere with certain cellular functions, and prompt caution in using coumarin-caged second messengers in cellular studies.	Oleksandra Tiapko Bernadett Bacsa Gema Guedes de la Cruz Toma Glasnov Klaus Groschner	2016	Science China(Life Sciences)2016，59，8：	1
	12	Na^+/Ca^2+ exchange facilitates Ca^2+-dependent activation of endothelial nitric-oxide synthase显示文摘	Tenbl M Groschner K Kohlwein SD	1999	J Biol Chem1999，274，29：	1
	13	钙池操纵的钙内流通路病理生理学中涌现的信号转导概念显示文摘钙池操纵的钙内流(SOCE)参与了体内许多重要的生理过程,如心脏节律、肌肉收缩、受精后卵子激活、免疫调控等等。本书是SOCE研究前沿的一个论文集。	Klaus Groschner(等) 魏玉保	2013	国外科技新书评介2013，，3：	0

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