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169篇 您的检索式:作者名="Beug"
    题名 作者 年代 出处 被引量
1Molecular requirements for epithelial–mesenchymal transition during tumor progression显示文摘Margit A Huber Norbert Kraut Hartmut Beug 2005Current Opinion in Cell Biology2005,,5:3
2p38α controls erythroblast enucleation and Rb signaling in stress erythropoiesis显示文摘成红血球细胞的 Enucleation 对哺乳动物在终端区别期间唯一。尽管 erythroid enucleation 广泛地被学习了,包括 retinoblastoma 蛋白质(Rb ) ,仅仅一些基因被识别了调整原子挤出。它由哪个仍然保持大部分未定义发信号的分子,外来的刺激例如 erythropoietin (Epo ) ,是 transduced 导致 enucleation。这里,我们显示出那 p38α,激活 mitogen 的蛋白质 kinase (MAPK ) ,为 erythroid enucleation 被要求。在包含高 Epo 层次并且模仿的一个前 vivo 区别系统强调红血球生成, p38α在 erythroid 区别期间被激活。p38α 的损失;完全堵住主要成红血球细胞的 enucleation。而且, p38α在胎儿、贫血的压力红血球生成期间在 vivo 以一种房间自治的方式调整成红血球细胞 enucleation。显著地, p38α 的损失;导致 p21 目标 Rb 的 p21,和减少的激活的 downregulation,哪个是成红血球细胞 enucleation 的重要管理者。这研究表明那 p38α在压力红血球生成期间是为成红血球细胞 enucleation 的一个关键发信号分子。Simon M Schultze Andreas Mairhofer Dan Li Jin Cen Hartmut Beug Erwin F Wagner Lijian Hui 2012Cell Research2012,22,3:2
3Molecular requirements for epithelial- mesenchymal transition during tumor progression 显示文摘Huber MA Kraut N Beug H 2005Curr Opin Cell Biol2005,17,5:1
4Molecular requirements for epithe- lial-mesenchymal transition during tumor progression 显示文摘Huber MA Kraut N Beug H 2005Curr Opin Cell Bio12005,17,5:1
5An overview of mushroom poisonings in North America 显示文摘Beug M W 2004Mycophile2004,45,2:1
6Molecular requirements for ep ithelial-mesenchymal transition during tumor progression显示文摘Huber MA Kraut N Beug H 2005CurrOpin Cell Biol2005,17,5:1
7Molecular requirements for epithelial- mesenchyrnal transition during tumor progression 显示文摘Huber MA Kraut N Beug H 2005Current opinion in cell biology2005,17,5:1
8Molecular requirementsfor epithelial-mesenchymal transition during tumor progression 显示文摘Huber M A Kraut N Beug H 2005Curt Opin Cell Biol2005,17,5:1
9Molecular requirements for epithelial-mesenchymal transition during tumor progression显示文摘Huber MA Kraut N Beug H 2005Curr Opin Cell Biol2005,17,:1
10Investigation of program saturation in scaled interpoly dielectric floating- gate memory devices 显示文摘Beug M F Chan N Hoehr T 2009IEEE Transactions on Elec- tron Devices2009,86,8:1
11Diverse cellular and molecular mechanisms contribute to epithelial plasticity and metastasis 显示文摘Grunert S Jechlinger M Beug H 2003Nat Rev Mol Cell Boil2003,4,8:1
12Molecular requirements for epithelial- mesenchymal transition during tumor progression 显示文摘Huber MA Krant N Beug H 2005Curr Opin Cell Biol2005,17,5:1
13Molecular requirements for epithelial-mesenchymal transition during tumor progression显示文摘Huber M A Kraut N Beug H 2005Curr Opin Cell Biol2005,17,5:1
14TGF-β signaling is necessary for carcinoma cell inv asiveness and metastasis显示文摘Oft M Heider KH Beug H 1998Curr Biol1998,8,23:1
15Diverse cellular and molecular mechanisms contribute to epithelial plasticity and metastasis显示文摘Grunert S Jechlinger M Beug H 2003Nat Rev Mol Cell Biol2003,4,8:1
16Reduction of fibro- genesis by selective delivery of a Rho kinase inhibitor to hepatic stellate cells in mice 显示文摘Van Beuge MM Prakash J Lacombe M 2011J Pharmacol Exp Ther2011,337,3:1
17TGF-beta signaling is necessary for carcinoma cell invasiveness and metastasis显示文摘Oft M Heider KH Beug H 1998Curr Biol1998,8,23:1
18Pollen distribution in hemipelagic surface sediments of the South China Sea and its relation to modem vegetation distribution 显示文摘Sun Xiangjun Li Xun Beug H-J 1999Mar Geol1999,156,14:1
19An interdisciplinary approach to the care of patients with Wegener's granulomatosis:long-term outcome in 155 patients显示文摘Reinhold-Keller E Beuge N Latza U 2000Arthritis Rheum2000,43,5:1
20Epithelial-mesenchymal transition:NF-κB takes center stage显示文摘Huber MA Beug H Wirth T 2004Cell Cycle2004,3,12:1
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