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| 1 | Infectious diseases in the aftermath of monsoon flooding in Pakistan显示文摘Pakistan is ranked 9th in terms of flood-affected countries worldwide.In the summer of 2010,the northern province of Khyber-Pakhtunkhwa received more than 312 mm of rain in a 56 hour period.This resulted in over 1 600 deaths across the region.In addition,over 14 million people were directly affected by this record-breaking deluge.Flood affected regions serve as ideal breeding grounds for pathogens,leading to the spread of diseases.The poor standards of hygiene in camps set up for individuals displaced by the floods also contribute to this.It is essential that those involved in relief efforts are aware of the epidemiology of diseases that have historically seen a sudden upsurge after natural disasters.Keeping this in mind,we conducted a simple review of literature.An extensive literature search was conducted using the PubMed data base and online search engines.Articles published in the last 20 years were considered along with some historical articles where a background was required.Seven major diseases were identified to increase substantially in the aftermath of natural disasters.They were then classified into acute and sub-acute settings.Diarrhea,skin&eye infections and leptospirosis were identified in the acute setting while malaria,leishmaniasis,respiratory infections and hepatitis were identified in the sub-acute setting. | Maryam Baqir Zain A Sobani Amyn Bhamani Nida Shahab Bham Sidra Abid Javeria Farook M Asim Beg | 2012 | Asian Pacific Journal of Tropical Biomedicine2012，2，1： | 2 |
| 2 | Mechanisms mediating the effects of alcohol and HIV anti-retroviral agents on mTORC1,mTORC2 and protein synthesis in myocytes显示文摘Alcoholism and acquired immune deficiency syndrome are associated with severe muscle wasting.This impairment in nitrogen balance arises from increased protein degradation and a decreased rate of protein synthesis.The regulation of protein synthesis is a complex process involving alterations in the phosphorylation state and protein-protein interaction of various components of the translation machinery and mammalian target of rapamycin(mTOR) complexes.This review describes mechanisms that regulate protein synthesis in cultured C2C12 myocytes following exposure to either alcohol or human immunodeficiency virus antiretroviral drugs.Particular attention is given to the upstream regulators of mTOR complexes and the downstream targets which play an important role in translation.Gaining a better understanding of these molecular mechanisms could have important implications for preventing changes in lean body mass in patients with catabolic conditions or illnesses. | Ly Q Hong-Brown Abid A Kazi Charles H Lang | 2012 | World Journal of Biological Chemistry2012，3，6： | 2 |
| 3 | Candida esophagitis:Risk factors in non-HIV population in Pakistan显示文摘AIM:Candida esophagitis is a frequent infection in immunocompromised patients. This study was designed to determine its characteristics in non- human immune deficiency virus (HIV) infected patients attending a teaching hospital.METHODS: Clinical records of all patients coded by international classification of diseases 9th revision with clinical modifications'(ICD-9-CM), with candida esophagitis diagnosed by esophagogastroduodenoscopy (EGD) and histopathology over a period of 5 years were studied.RESULTS: Fifty-one patients (27 males, 24 females, range 21-77 years old and mean age 52.9 years) fulfilled the criteria (0.34% of the EGD). The common predisposing factors were carcinoma (OR 3.87, CI 1.00-14.99) and diabetes mellitus (OR 4.39, CI 1.34-14.42). The frequent clinical symptoms were retrosternal discomfort, dysphagia and epigastric abdominal pain with endoscopic appearance of scattered mucosal plaques. Another endoscopic lesion was associated with candida esophagitis in 15% patients.CONCLUSION: Carcinomas, diabetes mellitus, corticosteroid and antibiotic therapy are major risk factors for candida esophagitis in Pakistan. It is an easily managed complication that responds to treatment with nystatin. | Javed Yakoob Wasim Jafri Shahab Abid Nadeem Jafri Muhammad Islam Saeed Hamid Hasnain A Shah Akbar S Hussainy | 2003 | World Journal of Gastroenterology2003，9，10： | 2 |
| 4 | 策略将在肝移植以后减少丙肝病毒复发显示文摘 Hepatitis C virus (HCV) is a major health problem that leads to chronic hepatitis, cirrhosis and hepatocellular carcinoma, being the most frequent indication for liver transplantation in several countries. Unfortunately, HCV re-infects the liver graft almost invariably following reperfusion, with an accelerated history of recurrence, leading to 10%-30% of patients progressing to cirrhosis within 5 years of transplantation. In this sense, some groups have even advocated for not retransplanting this patients, as lower patient and graftoutcomes have been reported. However, the management of HCV recurrence is being optimized and several strategies to reduce post-transplant recurrence could improve outcomes, decrease the rate of re-transplantation and optimize the use of available grafts. Three moments may be the focus of potential actions in order to decrease the impact of viral recurrence: the pretransplant moment, the transplant environment and the post-transplant management. In the pre-transplant setting, it is not well established if reducing the pre transplant viral load affects the risk for HCV progression after transplant. Obviously, antiviral treatment can render the patient HCV RNA negative post transplant but the long-term benefit has not yet been fully established to justify the cost and clinical risk. In the transplant moment, factors as donor age, cold ischemia time, graft steatosis and ischemia/reperfusion injury may lead to a higher and more aggressive viral recurrence. After the transplant, discussion about immunosuppression and the moment to start the treatment (prophylactic, pre-emptive or once-confirmed) together with new antiviral drugs are of interest. This review aims to help clinicians have a global overview of posttransplant HCV recurrence and strategies to reduce its impact on our patients. | Ruben Ciria María Pleguezuelo Shirin Elizabeth Khorsandi Diego Davila Abid Suddle Hector Vilca-Melendez Sebastian Rufian Manuel de la Mata Javier Briceo Pedro López Cillero Nigel Heaton | 2013 | World Journal of Hepatology2013，5，5： | 2 |
| 5 | Results of percuta- neous coronary interventions in patients≥90 years of age显示文摘 | IGAL T ABID A ELI L | 2007 | Catheter Cardiovasc Interv2007，70，： | 1 |
| 6 | Postoperative analgesia after cesarean section:sublingual buprenorphine versus subcutaneous morphine显示文摘 | Abid A Benhamou D Labaille T | 1990 | Ann Fr Anesth Reanim1990，9，： | 1 |
| 7 | 显示文摘 | ROBERT A M ABID A ANDREW P D | 1999 | J Am Chem Soc1999，121，： | 1 |
| 8 | NPHS2 gene in steroid-resistant nephrotic syn- drome : prevalence, clinical course, and mutational spectrum in south- west Iranian children显示文摘 | Mubarak M Abid A | 2014 | Iran J Kidney Dis2014，8，3： | 1 |
| 9 | Synthesis and antiamoebic activity of new oximc ether derivatives containing 2-aacetylpy- ridine/2-acetylfia-ran显示文摘 | Abid M Husain K Azam A | | 0，，19： | 1 |
| 10 | On the realizability ofReynolds stress turbulence closures 显示文摘 | Speziale C G Abid R Durbin P A | 1994 | Journal of ScientificComputing1994，9，： | 1 |
| 11 | Identification of the Uridine Diphosphate Glucuronosyltransferase isoform UGT1A6 in rat brain and in primary cultures of neurons and astrocytes显示文摘 | Suleman F G Abid A Gradinaru D | 1998 | Archives of Biochemistry and Biophysics1998，358，： | 1 |
| 12 | QuantitativeMeasurement of Soot Particle Size Distribution in Pre-mixed Flames-the Burner-Stabilized Stagnation FlameApproach 显示文摘 | Abid A D Camacho J Sheen D A | 2009 | Combustion and Flame2009，156，10： | 1 |
| 13 | Identifi- cation of the uridine diphosphate glucuronosyltrans- ferase isoform UGT1A6 in rat brain and in primary cultures of neurons and astrocytes显示文摘 | Suleman FG Abid A Gradinaru D | 1998 | Arch Biochem Biophys1998，358，1： | 1 |
| 14 | Anti- coagulation in pregnancy with mechanical heart valves: 10-year experience显示文摘 | AKHTAR R P ABID A R ZAFAR H | 2007 | Asian Cardiovasc Tho- rac Ann2007，15，： | 1 |
| 15 | Construction and characterization of infectious intragenotypic and intergenotypic hepatitis C virus chimeras显示文摘 | Pietschmann T Kaul A Koutsoudakis G Shavinskaya A Kallis S Steinmann E Abid K Negro F | | 0，，： | 1 |
| 16 | Familial aggregation and excess maternal transmission of type 2 diabetes in Tunisia显示文摘 | Arfa I Abid A Malouche D | 2007 | Postgard Med J2007，83，979： | 1 |
| 17 | Sizedistribution and morphology of nascent soot in premixed ethylene flames with and without benzene doping显示文摘 | Abid A D Tolmachoff E D Phares D J | 2009 | Proceedings of the Combustion Institute2009，32，1： | 1 |
| 18 | Quantitative Measurement of Soot Particle Size Distribution in Premixed Flames-The Burner-stabilized Flame Approach显示文摘 | Abid A D Camacho J Sheen D A | | 0，，： | 1 |
| 19 | Inhibition of microparticle release triggers endothelial cell apoptosis and detachment显示文摘 | Abid Hussein MN Bing AN Sturk A | 2007 | Thromb Haemost2007，98，5： | 1 |
| 20 | On Evolution of Particle Size Distribution Functions of Incipient Soot in Premixed Ethylene-Oxygen-Argon Flames显示文摘 | Abid A D Heinz N Tolmachoff E | | 0，，4： | 1 |
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