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    题名 作者 年代 出处 被引量
1Long-Term Complications, Immunologic Effects, and Role of Passage for Outcome in Mesenchymal Stromal Cell Therapy显示文摘2012Biology of Blood and Marrow Transplantation2012,,4:4
2HLA-Haploidentical Bone Marrow Transplantation for Hematologic Malignancies Using Nonmyeloablative Conditioning and High-Dose, Posttransplantation Cyclophosphamide显示文摘Leo Luznik Paul V. O'Donnell Heather J. Symons Allen R. Chen M. Susan Leffell Marianna Zahurak Ted A. Gooley Steve Piantadosi Michele Kaup Richard F. Ambinder Carol Ann Huff William Matsui Javier Bola?os-Meade Ivan Borrello Jonathan D. Powell Elizabeth Ha 2008Biology of Blood and Marrow Transplantation2008,,6:4
3Clinical characteristics and survival of patients with IgD multiple myeloma显示文摘MM(multiple myeloma)accounts for about 10%of hematological malignancies and 1%of cancer.IgD myeloma is a rare type of MM.It was reported that IgD type accounts for about 1%to 2%of all patients with MM abroad,while 3%to 8.9%in domestic cases.^(1) This immunoglobulin protein was first discovered by Dr.Rowe and Fahey in 1965.^(2) Half of a century had passed,the management of IgD MM remains a challenging field.Yi Li Zhen Cai 2021Blood Science2021,3,2:3
4Guidelines for Preventing Infectious Complications among Hematopoietic Cell Transplantation Recipients: A Global Perspective显示文摘Marcie Tomblyn Tom Chiller Hermann Einsele Ronald Gress Kent Sepkowitz Jan Storek John R. Wingard Jo-Anne H. Young Michael A. Boeckh 2009Biology of Blood and Marrow Transplantation2009,,10:3
5European LeukemiaNet recommendations for the management of chronic myeloid leukemia: 2013显示文摘Michele Baccarani Michael W. Deininger Gianantonio Rosti Andreas Hochhaus Simona Soverini Jane F. Apperley Francisco Cervantes Richard E. Clark Jorge E. Cortes Fran?ois Guilhot Henrik Hjorth-Hansen Timothy P. Hughes Hagop M. Kantarjian Dong-Wook Kim Richa 2013Blood2013,,6:3
6PD-L1:expression regulation显示文摘Programmed death-ligand 1(PD-L1),expressed on the surface of tumor cells,can bind to programmed cell death-1(PD-1)on T cells.The interaction of PD-1 and PD-L1 can inhibit T-cell responses by decreasing T-cell activity and accelerating their apoptosis.Various cancers express high levels of PD-L1 and exploit PD-L1/PD-1 signaling to evade T-cell immunity,and immunotherapies targeting the PD-1/PD-L1 axis have been shown to exert remarkable anti-tumor effects;however,not all tumor patients benefit from these therapies.Therefore,study of the mechanisms regulating PD-L1 expression are imperative.In this review,we explore regulation of PD-L1 expression in the contexts of gene transcription,signaling pathways,histone modification and remodeling,microRNAs,long noncoding RNAs,and post-translational modification.Current developments in studies of agents that block PD-L1 and correlations between immunotherapies targeting PD-1/PD-L1 and PD-L1 expression are also summarized.Our review will assist in understanding of PD-L1 expression regulation and discusses the implications of reported findings in cancer diagnosis and immunotherapy.Yu-Jie Zhou Guoli Li Jiyin Wang Mengyuan Liu Zihan Wang Yu Song Xulong Zhang Xi Wang 2023Blood Science2023,5,2:3
7Interleukin-15 can replace theIL-2 signal in IL-2-dependent adult T-cell leukemia (ATL) cell linesexpression of IL-15 receptor alpha on ATL cells 显示文摘Yamada Y Sugawara K Hata T 1998Blood1998,91,11:3
8Aplastic Anemia: Pathophysiology and Treatment显示文摘Neal S. Young Andrea Bacigalupo Judith C.W. Marsh 2010Biology of Blood and Marrow Transplantation2010,,1:3
9Red cell distribution width and inflammation in patients with non-dipper hypertension显示文摘Firat ?zcan Osman Turak Akif Durak Ahmet ??leyen Fatih U?ar Zeynep Gini? Fatma U?ar Fatma Nurcan Ba?ar Sinan Aydo?du 2013Blood Pressure2013,,2:3
10Stroke is more common than myocardial infarction in hypertension:analysis based on 11 major randomized intervention trials显示文摘Kjeldsen SE Julius S Hedner T Blood Pressure0,,:3
11Changes in amino acid neurotransmitters and cerebral blood flow in the ischemic penumbral region following middle cerebral artery occlusion in the rat:correlation with histopathology显示文摘Takagi K Ginsbern MD Globus MYT 1993J Cereb Blood Flow Metab1993,,:2
12Risk Factors for Progression from Cytomegalovirus Viremia to Cytomegalovirus Disease after Allogeneic Hematopoietic Stem Cell Transplantation显示文摘Ji Eun Jang Shin Young Hyun Yun Deok Kim Sul Hee Yoon Doh Yu Hwang Soo Jeong Kim Yuri Kim Jin Seok Kim June-Won Cheong Yoo Hong Min 2012Biology of Blood and Marrow Transplantation2012,,6:2
13Inhibition of Immature eryrthroid progenitor cell proliferation by macrophage inflammatory protein-1 alpha by interacting mainly with a C-C chemokine receptor, CCR1显示文摘Su S Mukaida N Wand J 1997Blood1997,90,2:2
14The quantification of cerebral infarction following focal ischemia in the rat:influence of strain,arterial pressure,blood glucosr concentration and age显示文摘Duverger D MacKenzine ET 1988J Cereb Blood Flow Metab1988,8,4:2
15Downregulation of antigen-presenting cell functions after administration of mitogenic anti-CD3 monoclonal antibodies in mice显示文摘Murailla E Andris F Pajak B 1999Blood1999,94,:2
16Macrophages in leukemia microenvironment显示文摘Macrophages,the important component of tissue microenvironment,play important roles in both physiological and pathological processes,including tumor and leukemia.Tumor-associated macrophages are involved in tumor proliferation,angiogenesis,invasion,metastasis,and chemotherapy resistance.In leukemia,macrophages are educated by leukemia microenvironment to obtain specific activated phenotype and participate in leukemia progression.Recent studies have shown that accumulation of macrophages in leukemia patients or mouse model is correlated with poor prognosis.Hence,increasing attentions have been paid to study the characteristics of them and to develop novel therapeutic strategies targeting macrophages against leukemia.In this article,we summarize recent development of macrophages in leukemia microenvironment.Lina Wang Guoguang Zheng 2019Blood Science2019,1,1:2
17DDIT4 mediates the proliferation-promotive effect of IL-34 in human monocytic leukemia cells显示文摘Interleukin 34(IL-34)is a cytokine that shares the receptor with colony-stimulating factor 1(CSF-1).IL-34 is involved in a broad range of pathologic processes including cancer.We previously demonstrated that IL-34 promoted the proliferation and colony formation of human acute monocytic leukemia(AMoL)cells.However,the mechanism has not been elucidated.Here,by analyzing the gene profiles of Molm13 and THP1 cells overexpressing IL-34(Molm13-IL-34 and THP1-IL-34),upregulation of the DNA damageinducible transcript 4(DDIT4)was detected in both series.Knockdown of DDIT4 effectively inhibited the proliferation,promoted apoptosis and colony formation in Molm13-IL-34 and THP1-IL-34 cells.Our results suggest that DDIT4 mediates the proliferationpromotive effect of IL-34 whereas does not mediate the promotive effect of IL-34 on colony formation in AMoL cells.Xiaoqian Lv Yuting Hu Lina Wang Dongyue Zhang Hao Wang Yibo Dai Xiaoxi Cui Guoguang Zheng 2021Blood Science2021,3,2:2
18Chronic lymphocytic leukemic B cells but not normal B cells are rescued from apoptosis by contact with normal bone marrow stromal cells显示文摘Lagneaux L Delforge D Bron D 1998Blood1998,91,7:2
19Membrane-type matrix metalloproteinase-mediated angiogenesis in a fibrin-collagen matrix 显示文摘Collen A Hanemaaijer R Lupu F 2003Blood2003,101,5:2
20Stroke is More Common than Myocardial Infarction in Hypertension: Analysis based on 11 Major Randomized Intervention Trials显示文摘Sverre E. Kjeldsen Stevo Julius Thomas Hedner Lennart Hansson 2001Blood Pressure2001,,4:2
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