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| 1 | Intraoperative cell salvage with autologous transfusion in liver transplantation显示文摘Liver transplant(LT) is the primary treatment for patients with end-stage liver disease. About 25000 LTs are performed annually in the world. The potential for intraoperative bleeding is quite variable. However, massive bleeding is common and requires blood transfusion. Allogeneic blood transfusion has an immunosuppressive effect and an impact on recipient survival, in addition to the risk of transmission of viral infections and transfusion errors, among others.Techniques to prevent excessive bleeding or to use autologous blood have been proposed to minimize the negative effects of allogeneic blood transfusion.Intraoperative reinfusion of autologous blood is possible through previous selfdonation or blood collected during the operation. However, LT does not normally allow autologous transfusion by prior self-donation. Hence, using autologous blood collected intraoperatively is the most feasible option. The use of intraoperative blood salvage autotransfusion(IBSA) minimizes the perioperative use of allogeneic blood, preventing negative transfusion effects without negatively impacting other clinical outcomes. The use of IBSA in patients with cancer is still a matter of debate due to the theoretical risk of reinfusion of tumor cells. However, studies have demonstrated the safety of IBSA in several surgical procedures, including LT for hepatocellular carcinoma. Considering the literature available to date, we can state that IBSA should be routinely used in LT, both in patients with cancer and in patients with benign diseases. | Marcelo A Pinto Marcio F Chedid Leo Sekine Andre P Schmidt Rodrigo P Capra Carolina Prediger Jo?o E Prediger Tomaz JM Grezzana-Filho Cleber RP Kruel | 2019 | World Journal of Gastrointestinal Surgery2019，11，1： | 16 |
| 2 | Differential susceptibility following 13-amyloid peptide-(1-40)administration in C57BL/6 and Swiss albino mice:Evidence for a dissociation between :ognitive deficits and the glutathione system respons显示文摘 | Prediger RDS Franco J L Pandolfo P | 2007 | BehavBrain Res2007，177，2： | 1 |
| 3 | Involvement of phosphoinositide 3-kinaseγ in the nenro-inflammatory response and cognitive impairments induced by 13-amyloid 1 -40 peptide in mice 显示文摘 | Passos G F Figueiredo C P Prediger R D | 2010 | Brain Behav Immun2010，24，3： | 1 |
| 4 | Genetic deletion or antagonism of kinin B(1) and B(2) receptors improves cognitive deficits in a mouse model of Alzheimer's disease显示文摘 | Prediger RD Medeiros R Pandolfo P Duarte FS Passos GF Pesquero JB | | 0，，： | 1 |
| 5 | Connecting TNF-alpha signaling pathways to iNOS expression in a mouse model of Alzheimer's disease:relevance for the behavioral and synaptic deficits induced by amyloid beta protein显示文摘 | Medeiros R Prediger RD Passos GF Pandolfo P Duarte FS Franco JL | | 0，，： | 1 |
| 6 | Genetic deletion or an-tagonism of Kinin B(1)and B(2)receptors improves cognitive defi-cits in a mouse model of Alzheimer's disease显示文摘 | Prediger R D Medeiros R Pandolfo P | 2008 | Neuroscience2008，151，3： | 1 |
| 7 | Palladium-catalyzed Suzuki cross-coupling of 2-haloselenophenes : synthesis of 2-arylselenophenes, 2,5 -diarylselenophenes, and 2-arylselenophenyl ketones 显示文摘 | Prediger P Moro A V Nogueira C W | 2006 | J Org Chem2006，71，10： | 1 |
| 8 | Differential susceptibility following β-amyloid peptide-(1-40) administration in C57BL/6 and Swiss albino mice: Evidence for a dissociation between cognitive deficits andtheglutathionesystemresponse显示文摘 | Prediger RDS Franco JL Pandolfo P | 2007 | BehavBrainRes2007，177，2： | 1 |
| 9 | Connecting TNF - a signaling path- ways to iNOS expression in a mouse model of Alzheimer's disease: relevance for the behavioral and synaptic deficits induced by amy- loid β protein 显示文摘 | Medeiros R Prediger RD Passos GF Pandolfo P Duarte FS Franco JL Dafre AL Di Giunta G Figueiredo CP Takahashi RN Campos MM Calixto JB | 2007 | The Journal of Neuroscience2007，27，20： | 1 |
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