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86篇 您的检索式:作者名="Meuth"
    题名 作者 年代 出处 被引量
1An established preadipose cell line and its differentiation in culture显示文摘GREEN H MEUTH M 1974Cell1974,3,:1
2Correlation of Tchannel coding geneexpression,IT,and the low threshold Ca《'2+》 spike in the thalamus of a rat model ofabsence epilepsy显示文摘Broicher T Kanyshkova T Meuth P 2008Mol Cell Neurosci2008,39,:1
3Methylational urinalysis:a prospective study of bladder cancer patients and age stratified benign controls显示文摘Yates DR Rehman I Meuth M 2006Oncogene2006,250,13:1
4Eupus erythematosus tu- midus is a separate subtype of cutaneous lupus erythematosus显示文摘Schmitt V Meuth AM Amle S 2010British Journal of Dermatology2010,162,:1
5The neuroprotective impact of the leak potassium channel TASK1 on stroke development in mice 显示文摘Meuth SG Kleinschnitz C Broicher T 2009Neurobiol Dis2009,33,1:1
6Detrimental contribution of the immuno-inhibitor B7-H1 to rabies virus encephalitis显示文摘Lafon M Megret F Meuth SG 2008J Immunol2008,180,11:1
7Roduction of protein-tyrosine phosphatase- 1B increases insulin signaling in FAO hepatoma cells 显示文摘Clampit JE Meuth JL Smith HT 2003Biochem biophs Commun2003,300,2:1
8Inorganic phosphate accelerates the migration of vascular smooth muscle cells : evidence for the involvement of miR-223 显示文摘Rangrez AY M' Baya-Moutoula E Metzinger-Le Meuth V 2012PLoS One2012,7,47:1
9Design of digital correlation systems for low-intensity precision photon spectroscopic measurements显示文摘Hoppe B Meuth H Engels M 2001IEE Proc-Circuits Devices Syst2001,148,:1
10Chkl and p21 cooperate to prevent apoptosis during DNA replication fork stress显示文摘Rodriguez R Meuth M 2006Mol Biol Cell2006,17,1:1
11Chkl and p21 cooperate to prevent apop- tosis during DNA replication fork stress显示文摘Rodriguez R Meuth M 2006Mol Biol Cell2006,17,1:1
12Reduced apoptotic response to camptothecin in CHO cells deficient in XRCC3 显示文摘Hinz JM Helleday T Meuth M 2003Carcinogenesis2003,24,2:1
13Numerically controlled oscillators with hybrid function generators显示文摘Janiszewski I Hoppe B Meuth H 2002IEEE Transactions on Ultrasonics Ferroelectrics and Frequency Control2002,49,7:1
14CD8+ T cells and neuronal damage:direct and collateral mechanisms of cytotoxicity and impaired electrical excitability显示文摘Melzer N Meuth SG Wiendl H 0,,01:1
15The next-generation sphingosine-1 receptor modulator BAF312(siponimod) improves cortical network functionality in focal autoimmune encephalomyelitis显示文摘Autoimmune diseases of the central nervous system(CNS) like multiple sclerosis(MS) are characterized by inflammation and demyelinated lesions in white and grey matter regions. While inflammation is present at all stages of MS, it is more pronounced in the relapsing forms of the disease, whereas progressive MS(PMS) shows significant neuroaxonal damage and grey and white matter atrophy. Hence, disease-modifying treatments beneficial in patients with relapsing MS have limited success in PMS. BAF312(siponimod) is a novel sphingosine-1-phosphate receptor modulator shown to delay progression in PMS. Besides reducing inflammation by sequestering lymphocytes in lymphoid tissues, BAF312 crosses the blood-brain barrier and binds its receptors on neurons, astrocytes and oligodendrocytes. To evaluate potential direct neuroprotective effects, BAF312 was systemically or locally administered in the CNS of experimental autoimmune encephalomyelitis mice with distinct grey-and white-matter lesions(focal experimental autoimmune encephalomyelitis using an osmotic mini-pump). Ex-vivo flow cytometry revealed that systemic but not local BAF312 administration lowered immune cell infiltration in animals with both grey and white matter lesions. Ex-vivo voltage-sensitive dye imaging of acute brain slices revealed an altered spatio-temporal pattern of activation in the lesioned cortex compared to controls in response to electrical stimulation of incoming white-matter fiber tracts. Here, BAF312 administration showed partial restore of cortical neuronal circuit function. The data suggest that BAF312 exerts a neuroprotective effect after crossing the blood-brain barrier independently of peripheral effects on immune cells. Experiments were carried out in accordance with German and EU animal protection law and approved by local authorities(Landesamt für Natur, Umwelt und Verbraucherschutz Nordrhein-Westfalen;87-51.04.2010.A331) on December 28, 2010.Petra Hundehege Manuela Cerina Susann Eichler Christian Thomas Alexander M. Herrmann Kerstin Goel Thomas Müntefering Juncal Fernandez-Orth Stefanie Bock Venu Narayanan Thomas Budde Erwin-Josef Speckmann Heinz Wiendl Anna Schubart Tobias Ruck Sven G. Meuth 2019Neural Regeneration Research2019,14,11:1
16Differential regulation of HCN channel isoform expression in tha-lamic neurons of epileptic and non-epileptic rat strains显示文摘Kanyshkova T Meuth P Bista P 2012Neurobiol Dis2012,45,1:1
17Numerically controlled oscillators with hybrid function generators显示文摘Janiszew ski I Hoppe B Meuth H 2002IEEE Trans Ferroelectrics and Frequency Control2002,49,7:1
18The contribution of TWIK-related acid-sensitive K+-containing channels to the function of dorsal lateral geniculate thalamocortical relay neurons 显示文摘Meuth SG Aller MI Munsch T 2006Mol Phannacol2006,69,:1
19Blocking of bradykinin receptor B1 protects from focal closed head injury in mice by reducing axonal damage and astroglia activation Journal of Cerebral Blood Flow and Metabo- lism显示文摘Albert-Weissenberger C Stetter C Meuth S 2012Jounat Cerebral Blood Flow & Metabolism2012,32,9:1
20miR-143 and miR-145: molecular keys to switch the phenotypeof vascular smooth muscle cells 显示文摘Rangrez AY Massy ZA Metzinger-Le Meuth V 2011Circ Cardiovasc Genet2011,4,2:1
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