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12篇 您的检索式:作者名="Kylie E"
    题名 作者 年代 出处 被引量
1Comparison of Escherichia coli isolates implicated in human urinary tract infection and avian colibacillosis 显示文摘Kylie E Rodriguez -Siek 2005Micro2005,151,:1
2Advanced glycation end-products induce vascular dysfunction via resistance to nitric oxide and suppression of endothelial nitric oxide synthase显示文摘Aino Soro-Paavonen Wei-Zeng Zhang Kylie Venardos Melinda T Coughlan Emma Harris David CK Tong Daniella Brasacchio Karri Paavonen Jaye Chin-Dusting Mark E Cooper David Kaye Merlin C Thomas Josephine M Forbes 2010Journal of Hypertension2010,,4:1
3DNA sequence of a ColV plasmid and prevalence of selected plasmid-encoded virulence genes among avian Eschenchia coli strains 显示文摘Timothy J J Kylie E S Sara J J 2006J Bacteriol2006,188,2:1
4The integration of problem-based learning strategies in distance education显示文摘Nancy E Kylie H Betty C 1999Nurse Educator1999,24,1:1
5Glass transition related cohesion of amorphous sugar powders显示文摘KYLIE D F BRONLUND J E PATERSON A H J 2006J Food Eng2006,77,4:1
6Influence o decomposing iellyfish on the sediment oxygen demand and nutrient dynamics 显示文摘West E Welsh D Pitt Kylie 2009Hydrobiologia2009,616,:1
7Prevalence and underdiagnosis of chronic obstructive pulmonary disease among patients at risk in primary care显示文摘Hill Kylie Goldstein Roger S Guyatt Gordon H Blouin Maria Tan Wan C Davis Lori L Heels-Ansdell Diane M Erak Marko Bragaglia Pauline J Tamari Itamar E Hodder Richard Stanbrook Matthew B 2010Canadian Medical Association. Journal2010,,7:1
8Effect of recombinant human bone morphogenetic protein - 2 onmandibular distraction osteogenesis显示文摘Ozec Y Ozturk M Kyly E 2006J Craniofae Surg2006,17,1:1
9Stepwise oxidation of thiophene and its derivatives by hydrogen peroxide catalyzed by methyltrioxorhenium(VII) 显示文摘KYLIE N B JAMES H E 1996Inorg Chem1996,35,25:1
10Metastatic disease to the liver:Locoregional therapy strategies and outcomes显示文摘Secondary cancers of the liver are more than twenty times more common than primary tumors and are incurable in most cases.While surgical resection and systemic chemotherapy are often the first-line therapy for metastatic liver disease,a majority of patients present with bilobar disease not amenable to curative local resection.Furthermore,by the time metastasis to the liver has developed,many tumors demonstrate a degree of resistance to systemic chemotherapy.Fortunately,catheter-directed and percutaneous locoregional approaches have evolved as major treatment modalities for unresectable metastatic disease.These novel techniques can be used for diverse applications ranging from curative intent for small localized tumors,downstaging of large tumors for resection,or locoregional control and palliation of advanced disease.Their use has been associated with increased tumor response,increased disease-free and overall survival,and decreased morbidity and mortality in a broad range of metastatic disease.This review explores recent advances in liver-directed therapies for metastatic liver disease from primary colorectal,neuroendocrine,breast,and lung cancer,as well as uveal melanoma,cholangiocarcinoma,and sarcoma.Therapies discussed include bland transarterial embolization,chemoembolization,radioembolization,and ablative therapies,with a focus on current treatment approaches,outcomes of locoregional therapy,and future directions in each type of metastatic disease.Kylie E Zane Jordan M Cloyd Khalid S Mumtaz Vibhor Wadhwa Mina S Makary 2021World Journal of Clinical Oncology2021,12,9:0
11Emerging curative-intent minimally-invasive therapies for hepatocellular carcinoma显示文摘Hepatocellular carcinoma(HCC)is the most common cause of liver malignancy and the fourth leading cause of cancer deaths universally.Cure can be achieved for early stage HCC,which is defined as 3 or fewer lesions less than or equal to 3 cm in the setting of Child-Pugh A or B and an ECOG of 0.Patients outside of these criteria who can be down-staged with loco-regional therapies to resection or liver transplantation(LT)also achieve curative outcomes.Traditionally,surgical resection,LT,and ablation are considered curative therapies for early HCC.However,results from recently conducted LEGACY study and DOSISPHERE trial demonstrate that transarterial radio-embolization has curative outcomes for early HCC,leading to its recent incorporation into the Barcelona clinic liver criteria guidelines for early HCC.This review is based on current evidence for curativeintent loco-regional therapies including radioembolization for early-stage HCC.Kylie E Zane Paul B Nagib Sajid Jalil Khalid Mumtaz Mina S Makary 2022World Journal of Hepatology2022,14,5:0
12Spinal cord injury reprograms muscle fibroadipogenic progenitors to form heterotopic bones within muscles显示文摘The cells of origin of neurogenic heterotopic ossifications(NHOs), which develop frequently in the periarticular muscles following spinal cord injuries(SCIs) and traumatic brain injuries, remain unclear because skeletal muscle harbors two progenitor cell populations: satellite cells(SCs), which are myogenic, and fibroadipogenic progenitors(FAPs), which are mesenchymal. Lineage-tracing experiments using the Cre recombinase/Lox P system were performed in two mouse strains with the fluorescent protein Zs Green specifically expressed in either SCs or FAPs in skeletal muscles under the control of the Pax7 or Prrx1 gene promoter, respectively. These experiments demonstrate that following muscle injury, SCI causes the upregulation of PDGFRα expression on FAPs but not SCs and the failure of SCs to regenerate myofibers in the injured muscle, with reduced apoptosis and continued proliferation of muscle resident FAPs enabling their osteogenic differentiation into NHOs. No cells expressing Zs Green under the Prrx1 promoter were detected in the blood after injury, suggesting that the cells of origin of NHOs are locally derived from the injured muscle. We validated these findings using human NHO biopsies. PDGFRα+mesenchymal cells isolated from the muscle surrounding NHO biopsies could develop ectopic human bones when transplanted into immunocompromised mice, whereas CD56+myogenic cells had a much lower potential. Therefore, NHO is a pathology of the injured muscle in which SCI reprograms FAPs to undergo uncontrolled proliferation and differentiation into osteoblasts.Hsu-Wen Tseng Dorothée Girard Kylie A.Alexander Susan M.Millard Frédéric Torossian Adrienne Anginot Whitney Fleming Jules Gueguen Marie-Emmanuelle Goriot Denis Clay Beulah Jose Bianca Nowlan Allison R.Pettit Marjorie Salga François Genêt Marie-Caroline Le Bousse-Kerdilès Sébastien Banzet Jean-Pierre Lévesque 2022Bone Research2022,10,2:0
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