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		题名	作者	年代	出处	被引量
	1	Microbiome as mediator: Do systemic infections start in the gut?显示文摘The intestinal microbiome is emerging as a crucial mediator between external insults and systemic infections. New research suggests that our intestinal microorganisms contribute to critical illness and the development of non-gastrointestinal infectious diseases.Common pathways include a loss of fecal intestinal bacterial diversity and a disproportionate increase in toxogenic bacterial species. Therapeutic interventions targeting the microbiome- primarily probiotics- have yielded limited results to date. However, knowledge in this area is rapidly expanding and microbiome-based therapy such as short-chain fatty acids may eventually become a standard strategy for preventing systemic infections in the context of critical illness.	Melissa Latorre Suneeta Krishnareddy Daniel E Freedberg	2015	World Journal of Gastroenterology2015，21，37：	5
	2	When combination therapy isn't working: Emerging therapies for the management of inflammatory bowel disease显示文摘Although antagonists of tumor necrosis factor have resulted in major therapeutic benefits in inflammatory bowel disease, the magnitude and durability of response are variable. Similar to previously available drugs such as 5-aminosalicylates and immunomodulators, the therapeutic effect is not universal leaving many people searching for options. The development of newer agents has benefited from advances in the understanding of the pathophysiology of the disease. Uncontrolled activation of the acquired immune system has an important role, and lymphocytes, cytokines, and adhesion molecules are broadly targeted for therapeutic intervention. There is increasing evidence of an important role of the innate immune system and the intestinal epithelium, and the therapeutic paradigm is also shifting from immunosuppression to the reinforcement of the intestinal barrier, and modification of the disease process. In this review, we explore the limitation of current therapy as well as mechanisms of actions of new drugs and the efficacy and adverse events from data from clinical trials.	Suneeta Krishnareddy Arun Swaminath	2014	World Journal of Gastroenterology2014，20，5：	4
	3	Toll-Like Receptor-4 Promotes the Development of Colitis-Associated Colorectal Tumors显示文摘	Masayuki Fukata Anli Chen Arunan S. Vamadevan Jason Cohen Keith Breglio Suneeta Krishnareddy David Hsu Ruliang Xu Noam Harpaz Andrew J. Dannenberg Kotha Subbaramaiah Harry S. Cooper Steven H. Itzkowitz Maria T. Abreu	2007	Gastroenterology2007，，6：	1
	4	Distinguishing patients with celiac disease by quantitative analysis of videocapsule endoscopy images显示文摘	Edward J. Ciaccio Christina A. Tennyson Suzanne K. Lewis Suneeta Krishnareddy Govind Bhagat Peter H.R. Green	2010	Computer Methods and Programs in Biomedicine2010，，1：	1
	5	Phytochrome regulation of branching in arabidopsis显示文摘	Finlayson S A Krishnareddy S R Kebrom T H		0，，04：	1
	6	Cox-2 is regulated by Toil-like receptor-4 (TLR4) signaling:role in proliferation and apoptosis in the intestine显示文摘	Fukata M Chen A Klepper A Krishnareddy S Vamadevan A S Thomas L S	2006	Gastroenterology2006，131，：	1
	7	Soft rot inciting : Pectobacterium carotovorurn ( Erwinia carotovora) is unlikely to be transmitted as a latent pathogen in micropropagated banana显示文摘	THOMAS P GOPLAKRISHNAN C KRISHNAREDDY M	2011	Plant Cell Tissue and Organ Cult2011，105，3：	1

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