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15篇 您的检索式:作者名="Calì"
    题名 作者 年代 出处 被引量
1Astroglial anatomy in the times of connectomics显示文摘Corrado Calì 2017Journal of Translational Neuroscience2017,2,4:0
2Morbidly adherent placenta:evaluation of ultrasound diagnostic criteria and differentiation of placenta accreta from percreta显示文摘CalìG Giambanco L Puccio G 2013Ultrasound Obstet Gynecol2013,41,4:1
3Mitochondrial Ca2+ and neurodegeneration显示文摘Calì T Ottolini D Brini M 2012Cell Calcium2012,52,1:1
4Morbidly adherent placenta:evaluation of ultrasound diagnostic criteria and differentiation of placenta accreta from percreta显示文摘CalìG Giambanco L Puccio G 2013Ultrasound Obstet Gynecol2013,41,4:1
5Calcium in health and disease显示文摘Brini M Ottolini D CalìT 2013Met Ions Life Sci2013,13,:1
6Morbidly adherent placenta: evaluation of ultrasound diagnostic criteria and differentiation of placenta accreta from percreta显示文摘Calì G Giambanco L Puccio G 2013Ultrasound Obstet Gynecol2013,41,4:1
7Clinical and molecular characterisation of 80 patients with 5p deletion:genotype-phenotype correlation显示文摘Mainardi PC Perfumo C Calì A 2001J Med Genet2001,38,3:1
8Spotlights on immunological effects of reactive nitrogen species: When inflammation says nitric oxide显示文摘Over the last decades, nitric oxide(NO) has been definitively recognised as one of the key players involved in immunity and inflammation. NO generation was originally described in activated macrophages, which still represent the prototype of NO-producing cells. Notwithstanding, additional cell subsets belonging to both innate and adaptive immunity have been documented to sustain NO propagation by means of the enzymatic activity of different nitric oxide synthase isoforms. Furthermore, due to its chemical characteristics, NO could rapidly react with other free radicals to generate different reactive nitrogen species(RNS), which have been intriguingly associated with many pathological conditions. Nonetheless, the plethora of NO/RNS-mediated effects still remains extremely puzzling. The aim of this manuscript is to dig into the broad literature on the topic to provide intriguing insights on NO-mediated circuits within immune system. We analysed NO and RNS immunological clues arising from their biochemical properties, immunomodulatory activities and finally dealing with their impact on different pathological scenarios with far prompting intriguing perspectives for their pharmacological targeting.Andrea Predonzani Bianca Calì Andrielly HR Agnellini Barbara Molon 2015World Journal of Experimental Medicine2015,5,2:3
9Heart-kidney crosstalk and role ofhumoral signaling in critical illness显示文摘Virzì G Day S de Cal M 2014Crit Care2014,18,1:1
10Enhanced parkin levels favor ER-mitochondria crosstalk and guarantee Ca 2+ transfer to sustain cell bioenergetics显示文摘Tito Calì Denis Ottolini Alessandro Negro Marisa Brini 2013BBA - Molecular Basis of Disease2013,,4:1
11Oxidative stress:dual pathway induction in cardiorenal syndrome type 1 pathogenesis显示文摘VirzìGM Clementi A de Cal M 2015Oxid Med Cell Longev2015,2015,39:1
12Heart-kidney Crosstalk and role of humoral signaling in criticalillness显示文摘VirzìG Day S de Cal M 2014Crit Care2014,18,1:1
13Coronaviruses Hijack the LC3-I-Positive EDEMosomes, ER-Derived Vesicles Exporting Short-Lived ERAD Regulators, for Replication显示文摘Fulvio Reggiori Iryna Monastyrska Monique H. Verheije Tito Calì Mustafa Ulasli Siro Bianchi Riccardo Bernasconi Cornelis A.M. de Haan Maurizio Molinari 2010Cell Host & Microbe2010,,6:1
14GRP78 Mediates Cell Growth and Invasiveness in Endometrial Cancer显示文摘Gaetano Calì Luigi Insabato Domenico Conza Giuseppe Bifulco Luca Parrillo Paola Mirra Francesca Fiory Claudia Miele Gregory Alexander Raciti Bruno Di Jeso Giuseppe Terrazzano Francesco Beguinot Luca Ulianich 2014J Cell Physiol2014,,10:1
15Neutrophil gelatinase-associated lipocalin does not predict acute kidney injury in heart failure显示文摘BACKGROUND Acute cardiorenal syndrome type 1(CRS-1)is defined by a rapid cardiac dysfunction leading to acute kidney injury(AKI).Neutrophil gelatinaseassociated lipocalin(NGAL)is expressed on the surface of human neutrophils and epithelial cells,such as renal tubule cells,and its serum(sNGAL)and urinary have been used to predict AKI in different clinical settings.AIM To characterize CRS-1 in a cohort of patients with acute heart diseases,evaluating the potentiality of sNGAL as an early marker of CRS-1.METHODS We performed a retrospective cohort,multi-centre study.From January 2010 to December 2011,we recruited 202 adult patients admitted to the coronary intensive care unit(CICU)with a diagnosis of acute heart failure or acute coronary syndrome.We monitored the renal function to evaluate CRS-1 development and measured sNGAL levels within 24 h and after 72 h of CICU admission.RESULTS Overall,enrolled patients were hemodynamically stable with a mean arterial pressure of 92(82-107)mmHg,55/202(27.2%)of the patients developed CRS-1,but none of them required dialysis.Neither the NGAL delta value(AUC 0.40,95%CI:0.25-0.55)nor the NGAL peak(AUC 0.45,95%CI:0.36-0.54)or NGAL cutoff(≥140 ng/mL)values were statistically significant between the two groups(CRS-1 vs no-CRS1 patients).The area under the ROC curve for the prediction of CRS-1 was 0.40(95%CI:0.25-0.55)for the delta NGAL value and 0.45(95%CI:0.36-0.54)for the NGAL peak value.Finally,in multivariate analysis,the risk of developing CRS-1 was correlated with age>60 years,urea nitrogen at admission and 24 h-urine output(AUC 0.83,SE=60.5%SP=93%),while sNGAL was not significantly correlated.CONCLUSION In our population,sNGAL does not predict CRS-1,probably as a consequence of the mild renal injury and the low severity of heart disease.So,these data might suggest that patient selection should be taken into account when considering the utility of NGAL measurement as a biomarker of kidney damage.Fiorenza Ferrari Elisa Scalzotto Pasquale Esposito Sara Samoni Flavio Mistrorigo Lilia Maria Rizo Topete Massimo De Cal Grazia Maria Virzì Valentina Corradi Rossella Torregrossa Roberto Valle Stefania Bianzina Nadia Aspromonte Matteo Floris Alessandro Fontanelli Alessandra Brendolan Claudio Ronco 2020World Journal of Clinical Cases2020,8,9:3
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