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| 1 | Engineering of an FGF-proteoglycan fusion protein with heparin-independent,mitogenic activity显示文摘 | Atsuko Yoneda Masahiro Asada Yuko Oda | 2000 | Nature Biotechnology2000，18，6： | 1 |
| 2 | 1α,25(OH) 2 -Dihydroxyvitamin D 3 /VDR protects the skin from UVB-induced tumor formation by interacting with the β-catenin pathway显示文摘 | Yan J. Jiang Arnaud E. Teichert Frankie Fong Yuko Oda Daniel D. Bikle | 2013 | Journal of Steroid Biochemistry and Molecular Biology2013，，： | 1 |
| 3 | Vitamin D and calcium signaling in epidermal stem cells and their regeneration显示文摘Epidermal stem cells(SCs)residing in the skin play an essential role for epidermal regeneration during cutaneous wound healing.Upon injury,distinct epidermal SCs residing in the interfollicular epidermis and/or hair follicles are activated to proliferate.Subsequently,SCs and progeny migrate,differentiate and restore the epidermis.We review a role of the vitamin D signaling through its receptor of vitamin D receptor(Vdr)in these processes.Vdr conditional knockout(cKO)mouse skin experiences a delay in wound re-epithelialization under low dietary calcium conditions,stimulating our efforts to examine a cooperative role of Vdr with calcium signaling through the calcium sensing receptor in the epidermis.We review the role of vitamin D and calcium signaling in different processes essential for injury induced epidermal regeneration during cutaneous wound repair.First,we discuss their roles in self-renewal of epidermal SCs through β-catenin signaling.Then,we describe epidermal remodeling,in which SCs and progeny migrate and differentiate to restore the epidermis,events controlled by the Ecadherin mediated adherens junction signaling.Finally,we discuss the potential mechanisms for vitamin D and calcium signaling to regulate injury induced epidermal regeneration mutually and interdependently. | Yuko Oda Daniel D Bikle | 2020 | World Journal of Stem Cells2020，12，7： | 1 |
| 4 | Identification of ghrelin in the house musk shrew ( Suncus murinus ): cDNA cloning, peptide purification and tissue distribution显示文摘 | Yuko Ishida Satoshi Sakahara Chihiro Tsutsui Hiroyuki Kaiya Ichiro Sakata Sen-ichi Oda Takafumi Sakai | 2009 | Peptides2009，，5： | 1 |
| 5 | The role of the calcium-sensing receptor in epidermal differentiation显示文摘 | Chia-Ling Tu Yuko Oda Laszlo Komuves Daniel D Bikle | 2003 | Cell Calcium2003，，3： | 1 |
| 6 | Naked DNA in cells:An inducer of major histocompatibility complex molecules to evoke autoimmune responses?显示文摘The major histocompatibility complex(MHC) is the exclusive chaperone that presents intracellular antigens,either self or foreign to T cells.Interestingly,aberrant expression of MHC molecules has been reported in various autoimmune target tissues such as thyroid follicular cells in Grave's disease.Herein,we review the discovery of an unexpected effect of cytosolic doublestranded DNA(ds DNA),despite its origins,to induce antigen processing and presenting genes,including MHC molecules,in non-immune cells.Moreover,we highlight several recent studies that suggest cell injury endows thyroid epithelial cells with a phenotype of mature antigen presenting cells by inducing multiple antigen processing and presenting genes via releasing genomic DNA fragments into the cytosol.We discuss the possibility that such cytosolic ds DNA,in naked form without binding to histone proteins,might be involved in the development of cell damage-triggered autoimmune responses.We also discuss the possible molecular mechanism by which cytosolic ds DNA can induce MHC molecules.It is reasonable to speculate that cytosolic ds DNA-induced MHC class Ⅰ is partially due to an autocrine/paracrine effect of type Ⅰ interferon(IFN).While the mechanism of cytosolic ds DNA-induced MHC class Ⅱ expression appears,at least partially,distinct from that mediated by IFN-γ.Further in-depth are required to clarify this picture. | Yuqian Luo Aya Yoshihara Kenzaburo Oda Yuko Ishido Naoki Hiroi Koichi Suzuki | 2016 | World Journal of Translational Medicine2016，5，1： | 0 |
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