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6篇 您的检索式：作者名="Yoshihiro Fujimura"

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		题名	作者	年代	出处	被引量
	1	Snake venom proteases affecting hemostasis and thrombosis显示文摘	Taei Matsui Yoshihiro Fujimura Koiti Titani	2000	Biochimica et Biophysica Acta2000，1447，：	1
	2	Paucity of CD34-positive cells and increased expression of high-mobility group box 1 in coronary thrombus with type 2 diabetes mellitus显示文摘	Atsushi Yamashita Kensaku Nishihira Yunosuke Matsuura Takashi Ito Kouichi Kawahara Kinta Hatakeyama Teruto Hashiguchi Ikuro Maruyama Hideo Yagi Masanori Matsumoto Yoshihiro Fujimura Kazuo Kitamura Yoshisato Shibata Yujiro Asada	2012	Atherosclerosis2012，，：	1
	3	Pivotal role of ADAMTS13 function in liver diseases显示文摘	Masahito Uemura Yoshihiro Fujimura Saiho Ko Masanori Matsumoto Yoshiyuki Nakajima Hiroshi Fukui	2010	International Journal of Hematology2010，，1：	1
	4	Snake venom protease affecting hemostasis and thrombosis 显示文摘	TAEI MATSUI YOSHIHIRO FUJIMURA KOITI TITANI	2000	Biochim Biophys Acta2000，1477，：	1
	5	Comparison of the effects of omeprazole and famotidine in treatment of upper abdominal symptoms in patients with reflux esophagitis显示文摘	Hiroaki Kusunoki Masayasu Kusaka Soichiro Kido Ryo Yamauchi Yoshinori Fujimura Yasuyuki Watanabe Michio Kobori Hiroto Miwa Toshihiko Tomita Yongmin Kin Kazutoshi Hori Nobuo Tano Kenji Sugimoto Yoshihiro Nakamura Kazuma Fujimoto Noriko Oza Aki Matsunobu Na	2009	Journal of Gastroenterology2009，，4：	1
	6	Relevance of ADAMTS13 to liver transplantation and surgery显示文摘A disintegrin-like and metalloproteinase with thrombospondin type-1 motifs 13(ADAMTS13) specifically cleaves unusually-large von Willebrand factor(VWF) multimers under high shear stress,and down-regulates VWF function to form platelet thrombi.Deficiency of plasma ADAMTS13 activity induces a life-threatening systemic disease,termed thrombotic microangiopathy(TMA) including thrombotic thrombocytopenic purpura(TTP).Children with advanced biliary cirrhosis due to congenital biliary atresia sometimes showed pathological features of TMA,with a concomitant decrease of plasma ADAMTS13 activity.Disappearance of their clinical findings of TTP after successful liver transplantation suggested that the liver is a major organ producing plasma ADAMTS13.In situ hybridization analysis showed that ADAMTS13 was produced by hepatic stellate cells.Subsequently,it was found that ADADTS13 was not merely responsible to development of TMA and TTP,but also related to some kinds of liver dysfunction after liver transplantation.Ischemia-reperfusion injury and acute rejection in liver transplant recipients were often associated with marked decrease of ADAMTS13 and concomitant formation of unusually large VWF multimers without findings of TMA/TTP.The similar phenomenon was observed also in patients who underwent hepatectomy for liver tumors.Imbalance between ADAMTS13 and VWF in the hepatic sinusoid might cause liver damage due to microcirculatory disturbance.It can be called as 'local TTP like mechanism' which plays a crucial role in liver dysfunction after liver transplantation and surgery.	Saiho Ko Hisanao Chisuwa Masanori Matsumoto Yoshihiro Fujimura Eiji Okano Yoshiyuki Nakajima	2015	World Journal of Hepatology2015，7，13：	0

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