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13篇 您的检索式:作者名="Yaw J"
    题名 作者 年代 出处 被引量
1Limit Load of Buried Pipelines in Inelastic Soil Medium显示文摘Yaw J C Shue Y C 0,,5:1
2Role of angiogenisis in hepatitis and hepatcoellular carcinoma显示文摘Chen - Guo K Hong - Yaw C Chung - Chou J 1999Hepto Gastroenterology1999,46,:1
3Breast cancer burden in Maiduguri,North eastern Nigeria显示文摘Nggada HA Yawe KD Abdulazeez J 2008Breast J2008,14,3:1
4Inheritance of fresh seed dormancy in groundnut显示文摘Yaw J Richard A Osei S 2008African Jour- nal of Biotechnology2008,7,4:1
5Differential roles of angiogenic chemokines in endothelial progenitor cell-induced angiogenesis显示文摘Isabella Kanzler Nancy Tuchscheerer Guy Steffens Sakine Simsekyilmaz Simone Konschalla Andreas Kroh David Simons Yaw Asare Andreas Schober Richard Bucala Christian Weber Jürgen Bernhagen Elisa A. Liehn 2013Basic Research in Cardiology2013,,1:1
6Waste minimization by process modifyeation显示文摘Hopper J R Yaws C kilo T C Vickhailak M 1993Waste Management1993,13,3:1
7Hydrocarbons : water solubility data 显示文摘Yaws C L Yang H C Hopper J R 1990Chem Eng1990,97,17:1
8Calculat- ing solubility Henry's law constants for gases in water 显示文摘YAWS C L HOPPER J R WANG X M 1999Chemical Engineering1999,106,6:1
9Calculating Solubility & Henry's Law Constants for Gases in Water 显示文摘Yaws C L Hopper J R Wang X 1999Chem Eng1999,6,:1
10Correlation constants for chemical compounds 24: Gas thermal conductivity, Gas viscosity,Vapor pressure 显示文摘Carl L Yaws Miller J M 1976Chem Eng1976,11,:1
11Differential roles of angiogenic chemokines in endothelial progenitor cell-induced angiogenesis显示文摘Isabella Kanzler Nancy Tuchscheerer Guy Steffens Sakine Simsekyilmaz Simone Konschalla Andreas Kroh David Simons Yaw Asare Andreas Schober Richard Bucala Christian Weber Jürgen Bernhagen Elisa A. Liehn 2013Basic Research in Cardiology2013,,1:1
12IKKβ binds NLRP3 providing a shortcut to inflammasome activation for rapid immune responses显示文摘Dear Editor,A rapid immune response to signals released from pathogens and injuries is critical for maintaining tissue integrity and restoring homeostasis.This response is largely mediated by the concerted action of pattern recognition receptors(PRRs).Such cooperativity has been described for Toll-like receptors(TLRs)and NACHT,LRR,and pyrin domain-containing protein 3(NLRP3),but the underlying molecular mechanisms remain incompletely understood.Inflammasomes are multi-protein complexes defined by a cytosolic innate immune sensor,usually a PRR,which recruits the adaptor molecule apoptosis-associated speck-like protein containing a caspase-recruitment domain(ASC)to activate the effector caspase-1 leading to the release of matured IL-1βand IL-18.Active caspase-1 further cleaves gasdermin D(GSDMD)allowing the N-terminal domain of GSDMD(GSDMD-N)to form pores in the plasma membrane,thus facilitating the release of matured IL-1βand IL-18.Pore-forming GSDMD-N further induces pyroptosis,an inflammatory form of cell death.Yaw Asare Margarita Shnipova LukaŽivković Christina Schlegl Federica Tosato Arailym Aronova Markus Brandhofer Laura Strohm Nathalie Beaufort Rainer Malik Christian Weber Jürgen Bernhagen Martin Dichgans 2022Signal Transduction and Targeted Therapy2022,7,11:0
13Tracing the failing heart: dual genetic fate mapping for target identification显示文摘In a recent study published in Nature Genetics,1 Han and colleagues employed a dual genetic lineage tracing approach in combination with a model of heart failure(HF)to identify a key sub-population of endocardium-derived fibroblasts,which gives rise to excessive myofibroblast formation in a Wnt pathway-mediated manner and contributes to cardiac fibrosis.1 The identified fibroblast sub-population and Wnt signaling pathway could be novel targets for the therapy of HF.Improvements in risk management and cardiovascular inter-vention have significantly reduced age-specific cardiovascular disease-related mortality.In contrast,the rate of HF-related hospitalisations has increased over the past decades and HF is a disease with highest social and economic cost in industrialized countries.HF is defined as impairment of the heart’s blood pumping function and its severity is assessed based on ejection fraction.HF often occurs after myocardial infarction(MI),with infarct size and quality of ventricular remodeling after MI influencing ventricular dysfunctions.Causes of HF also include hypertension,atrial fibrillation,and cardiomyopathies(Fig.1).Especially elderly patients with recurrent MI and comorbidities are increasingly presenting with signs of HF.Yaw Asare Christian Stoppe Jürgen Bernhagen 2023Signal Transduction and Targeted Therapy2023,8,9:0
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