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| 1 | Autophagy Opposes p53-Mediated Tumor Barrier to Facilitate Tumorigenesis in a Model of PALB2-Associated Hereditary Breast Cancer显示文摘 | Yanying Huo Hong Cai Irina Teplova Christian Bowman-Colin Guanghua Chen Sandy Price Nicola Barnard Shridar Ganesan Vassiliki Karantza Eileen White Bing Xia | 2013 | Cancer Discovery2013，，8： | 2 |
| 2 | Critical review of elastic and plastic thermal contact conductance models and comparison with experiment显示文摘 | SHRIDAR M R YOVANOVICH M M | 1994 | AIAA Journal of Thermophysics and Heat Transfer1994，8，4： | 1 |
| 3 | Genetic analysis of BRCA1 function in a defined tumor cell line 显示文摘 | Ralph S Shridar G Katerina V | 1999 | Mol Cell1999，4，： | 1 |
| 4 | Interaction of the Fanconi Anemia Proteins and BRCA1 in a Common Pathway显示文摘 | Irene Garcia-Higuera Toshiyasu Taniguchi Shridar Ganesan M.Stephen Meyn Cynthia Timmers James Hejna Markus Grompe Alan D D’Andrea | 2001 | Molecular Cell2001，，2： | 1 |
| 5 | Elastoplastic contact conduc- tance model for isotropic conforming rough surfaces and com- parison with experiments 显示文摘 | Shridar M R Yovanovich M M | 1966 | Joumal of Heat Transfer1966，118，2： | 1 |
| 6 | Single-stage hybrid coronary revascularization with long-term follow-up<sup> † </sup>显示文摘 | Corey Adams Daniel J.P. Burns Michael W.A. Chu Philip M. Jones Kumar Shridar Patrick Teefy William J. Kostuk Wojciech B. Dobkowski Jonathan Romsa Bob Kiaii | 2014 | European Journal of Cardio-Thoracic Surgery2014，，3： | 1 |
| 7 | Elastoplastic contact conductance model for isotropic conforming rough surfaces and comparison with experiments显示文摘 | Shridar M R Yovanovich M M | | 0，，02： | 1 |
| 8 | Elastoplastic contact conductance model for isotropic conforming rough surfaces and comparison with experiments显示文摘 | Shridar M R Yovanovich M M | 1966 | Journal of Heat Transfer1966，118，2： | 1 |
| 9 | Critical review of elastic and plastic thermal contact conductance models and comparison with experiment显示文摘 | Shridar M R Yovanovich M M | 1994 | AIAA Journal of Thermophysics and Heat Transfer1994，8，4： | 1 |
| 10 | Photonic crystals:Imaging by Flat Lens Using Negative Refraction显示文摘 | Parimi P V Lu W T Vodo P Shridar S | 2003 | Nature2003，426，： | 1 |
| 11 | Metallic species in ambient particulate matter at rural and urban location of Delhi 显示文摘 | Shridar V Khillare PS Agarwal T | 2010 | Journal of Hazardous Materials2010，175，： | 1 |
| 12 | Elastoplastic contact conductance model for isotropic conforming rough surfaces and comparison with experiments显示文摘 | SHRIDAR M R YOVANOVICH M M | 1966 | Journal of Heat Transfer1966，118，2： | 1 |
| 13 | BRCA1,PARP,and 53BP1:conditional synthetic lethality and synthetic viability显示文摘BRCA1 plays a critical role in the regulation of homologous recombination(HR)-mediated DNA double-strand break repair.BRCA1-deficient cancers have evolved to tolerate loss of BRCA1 function.This renders them vulnerable to agents,such as PARP inhibitors,that are conditionally‘synthetic lethal’with their underlying repair defect.Recent studies demonstrate that BRCA1-deficient cells may acquire resistance to these agents by partially correcting their defect in HR-mediated repair,either through reversion mutations in BRCA1 or through‘synthetic viable’loss of 53BP1.These findings and their clinical implications will be reviewed in this article. | Amal Aly Shridar Ganesan | 2011 | Journal of Molecular Cell Biology2011，3，1： | 0 |
| 14 | Loss of the BRCA1-PALB2 interaction accelerates p53-associated tumor development in mice显示文摘The BRCA1-PALB2-BRCA2 axis,or the BRCA pathway,plays key roles in genome stability maintenance and suppression of breast and several other cancers.Due to frequent p53 mutations in human BRCA1 breast cancers and mouse mammary tumors from Brcal,Brca2 and Palb2 conditional knockout models,it is often thought that p53 inactivation accelerates BRCA1/2 and PALB2-associated tumorigenesis.Here,we studied tumor development in mice with a mutation in Palb2 that disengages the PALB2-BRCA1 interaction in different Trp53 backgrounds.Rather than mammary tumors,Palb2 and Trp53 compound mutant mice developed,with greatly reduced latencies,lymphomas and sarcomas that are typically associated with germline Trp53 inactivation.Whole exome sequencing failed to identify any significant differences in genomic features between the same tumor types of Trp53 single mutant and Palb2;Trp53 compound mutant mice.These results suggest that loss of the BRCA pathway accelerates p53-associated tumor development,possibly without altering the fundamental tumorigenic processes. | Amar H.Mahdi Yanying Huo Ying Chen Pier Selenica Anchal Sharma Elise Merritt Nicola Barnard Chang Chan Shridar Ganesan Jorge S.Reis-Filho Britta Weigelt Subhajyoti De Bing Xia | 2022 | Genes & Diseases2022，9，3： | 0 |
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