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| 1 | Nebulette mutations are as- sociated with dilated cardiomyopathy and endocardial fibroelastosis 显示文摘 | Purevjav E Varela J Morgado M | 2010 | J Am Coil Cardiol2010，56，18： | 1 |
| 2 | Nebulette mutations are associated with dilated cardiomyopathy and endocardial fibroelastosis显示文摘 | PUREVJAV VARELA J MORGADO M | 2010 | Am Coll Cardiol J2010，56，18： | 1 |
| 3 | α-1-Syntrophin Mutation and the Long-QT Syndrome: A Disease of Sodium Channel Disruption显示文摘 | Geru Wu Tomohiko Ai Jeffrey J. Kim Bhagyalaxmi Mohapatra Yutao Xi Zhaohui Li Shahrzad Abbasi Enkhsaikhan Purevjav Kaveh Samani Michael J. Ackerman Ming Qi Arthur J. Moss Wataru Shimizu Jeffrey A. Towbin Jie Cheng Matteo Vatta | 2008 | Circulation: Arrhythmia and Electrophysiology2008，，3： | 1 |
| 4 | Dissection of Z-disc myopalladin gene network involved in the development of restrictive cardiomyopathy using system genetics approach显示文摘AIM To investigate the regulation of Myopalladin(Mypn) and identify its gene network involved in restrictive cardiomyopathy(RCM).METHODS Gene expression values were measured in the heart of a large family of BXD recombinant inbred(RI) mice derived from C57BL/6J and DBA/2J. The proteomics data were collected from Mypn knock-in and knock-out mice. Expression quantitative trait locus(eQ TL) mapping methods and gene enrichment analysis were used to identify Mypn regulation,gene pathway and co-expression networks.RESULTS A wide range of variation was found in expression of Mypn among BXD strains. We identified upstream genetic loci at chromosome 1 and 5 that modulate the expression of Mypn. Candidate genes within these loci include Ncoa2,Vcpip1,Sgk3,and Lgi2. We also identified 15 sarcomeric genes interacting with Mypn and constructed the gene network. Two novel members of this network(Syne1 and Myom1) have been confirmed at the protein level. Several members in this network are already known to relate to cardiomyopathy with some novel genes candidates that could be involved in RCM. CONCLUSION Using systematic genetics approach,we constructed Mypn co-expression networks that define the biological process categories within which similarly regulated genes function. Through this strategy we have found several novel genes that interact with Mypn that may play an important role in the development of RCM. | Qingqing Gu Uzmee Mendsaikhan Zaza Khuchua Byron C Jones Lu Lu Jeffrey A Towbin Biao Xu Enkhsaikhan Purevjav | 2017 | World Journal of Cardiology2017，9，4： | 1 |
| 5 | Myocardial Fas ligand expression increases susceptibility to AZT-induced cardiomyopathy显示文摘 | PUREVJAV E NELSON D P VARELA J J | 2007 | Cardiovasc Toxicol2007，7，4： | 1 |
| 6 | The development of a powerful Mongolian cyclone on 14-15 March 2021:Eddy energy analysis显示文摘2021年3月14-15日,强蒙古气旋引起的大范围强沙尘天气袭击了蒙古国和中国北方地区.本文从高频涡动能量学的角度分析了这一超强气旋的发展过程.2021年3月初,加强的极涡向中亚伸入,并与横跨东亚和东北亚的一个大型脊协同作用,由此形成了从中亚到贝加尔湖地区的强大斜压带.在这一有利的低频环流条件下,3月13日一个移动性小槽越过萨彦岭后触发了蒙古气旋.850 hPa涡旋动能的下传在气旋的加强和成熟阶段起到了关键作用.而这一机制主要由冷锋后侧的冷空气下沉过程完成.一旦气旋开始快速发展,锋面气旋波机制就变得至关重要.我们强调,高频涡动有效位能是首先从时间平均有效势能中提取能量,然后从低频有效位能中汲取能量而剧烈增长的,这正是该超强气旋的鲜明特征.“北冷南暖”的近地面温度气候异常型为时间平均有效位能的增多和向高频涡动有效位能的转换提供了条件,而“西北冷东南暖”的温度异常型则有利于低频有效位能的增加和向高频涡动有效位能的转换.分析结果表明,高低频波之间的相互作用对蒙古气旋的增强也很重要. | Cholaw Bueh Anran Zhuge Zuowei Xie Mei Yong Gomboluudev Purevjav | 2022 | Atmospheric and Oceanic Science Letters2022，15，6： | 1 |
| 7 | Molecular stud-in two Japanese childrenwith different phenotypes of glu-taric acidemia type II 显示文摘 | Purevjav E Kimura M Takusa Y | 2002 | Eur J Clin Invest2002，32，9： | 1 |
| 8 | Molecular study of electron transfer flavnprotein alpha-subunit deficiency in two Japanese chil- dren with different phenotypes of glutaric acidemia type II显示文摘 | Purevjav E Kimura M Takusa Y | 2002 | Ear J Clin Invest2002，32，9： | 1 |
| 9 | Crystal Structure of the TLR4-MD-2 Complex with Bound Endotoxin Antagonist Eritoran显示文摘 | Ho Min Kim Beom Seok Park Jung-In Kim Sung Eun Kim Judong Lee Se Cheol Oh Purevjav Enkhbayar Norio Matsushima Hayyoung Lee Ook Joon Yoo Jie-Oh Lee | 2007 | Cell2007，，5： | 1 |
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