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128篇 您的检索式：作者名="Phillips RJ"

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	题名	作者	年代	出处	被引量
1	The stromal de- rived factor-i/ CXCL12- CXC chemokine receptor 4 bio- logical axis in non-small cell lung cancer metastases显示文摘	Phillips RJ Burdick MD Lutz M	2003	Am J Respir Crit Care Med2003，167，12：	1
2	Anatomical and pathological considerations in percutaneous vertebroplasty and kyphoplasty:a reappraisal of the vertebral venous system显示文摘	Groen RJ du Toit DF Phillips FM	2004	Spine (Phila Pa 1976)2004，29，13：	1
3	Establishing a quality man- agement system for unit use testing based on NCCLS proposed guideline (EP18 P)显示文摘	Whitley RJ Santrach PJ Phillips DL	2001	Clin Chim Acta2001，307，12：	1
4	The SDF-1/CXCL12/CXCR4 biological axis in non-small cell lung cancer metastases显示文摘	Belperio JA Phillips RJ Burdick MD	2004	Chest2004，125，5：	1
5	The stromal derived receptor 4 biological axis in non-small cell lung cancer metastases 显示文摘	Phillips RJ Burdick MD Lutz M	2003	Am J Respir Cfit Care Med2003，167，12：	1
6	显示文摘	Phillips RJ Burdiek MD Hong K	2004	J Clin Invest2004，114，3：	1
7	Establishing a quality management system for unit use testing based on NCCLS proposed guideline(EP18-P)显示文摘	Whifley RJ Santrach PJ Phillips DL	2001	Clin Chim Acta2001，307，12：	1
8	Electroencephalographic and psychometric differences between boys with and without attention deficit hyperactivity disorder(ADHD): a pilot study 显示文摘	Cox DJ Kovatchev BP Morris JB Jr Phillips C Hill RJ Merkel L	1998	Appl Psychophysiol Biofeedback1998，23，3：	1
9	The stromal derived factor-1/CXCL12-CXC chemokine receptor 4 biological axis in non-smallcell lung cancer metastases显示文摘	Phillips RJ Burdick MD Lutz M	2003	Am J Respir Crit Care Med2003，167，12：	1
10	The influence of maternal BMI and age in twin pregnancies on insulin resistance in the offspring显示文摘	Phillips DI Fagard R	2002	Diabetes Care2002，25，12：	1
11	CXC chemokinesin angiogenesis of cancer显示文摘	Strieter RM Belperio JA Phillips RJ	2004	Semin Cancer Biol2004，14，：	1
12	Suppression of LPS-in- duced interferon-y and nitric oxide in splenic lymphocytes by select estrogen-regulated MieroRNAs: a novel mechanism of immune modulation显示文摘	Dai RJ Phillips RA Zhang Y	2008	Blood2008，112，：	1
13	ldentiflcation of a novel blocker of IkB kinase that enhances cellular apoptosis and inhibits cellular invasion through suppression of NF-κB regulated gene products显示文摘	Jimi E Phillips RJ Rincon M	2005	J Immunol2005，174，11：	1
14	Detection of macrolide resistant Mycoplasma pneumoniae in England,September 2014 to September 2015显示文摘	Brown RJ Macfarlane-Smith L Phillips S	2015	Euro Surveill2015，20，30：	1
15	Three-dimensional computed tomography of congenital nasal anomalies显示文摘	Schlosser RJ Faust RA Phillips CD	2002	In t J Pediatr Otorhinolaryngol2002，65，2：	1
16	The SDF-1/CXCR4 Biological Axis in Non-small Cell Lung Cancer Metastases显示文摘	Belperio JA Phillips RJ Burdick MD	2004	Chest2004，125，：	1
17	I kappa B-beta regulates the persistent response in a biphasic activation of NF-kappa B显示文摘	Thompson JE Phillips RJ Erdjument BH	1995	Cell1995，80，：	1
18	IκB -β regulates the persistent response inabiphasic activation of NF-κB显示文摘	Thompson JE Phillips RJ Erdjument Bromage H	1995	Cell1995，80，4：	1
19	Multifocal ' tapcte ' papillary fibroelastoma 显示文摘	Law KB Phillips KR Cusimano RJ	2009	J Clin Pathol2009，62，：	1
20	Steatotic livers are susceptible to normothermic ischemiareperfusion injury from mitochondrial Complex-Ⅰ?dysfunction显示文摘AIM: To assess the effects of ischemic preconditioning(IPC, 10-min ischemia/10-min reperfusion) on steatotic liver mitochondrial function after normothermic ischemia-reperfusion injury(IRI).METHODS: Sixty male Sprague-Dawley rats were fed8-wk with either control chow or high-fat/high-sucrose diet inducing > 60% mixed steatosis. Three groups(n = 10/group) for each dietary state were tested:(1) the IRI group underwent 60 min partial hepatic ischemia and 4 h reperfusion;(2) the IPC group underwent IPC prior to same standard IRI; and(3) sham underwent t h e s a m e s u r g e r y w i t h o u t I R I o r I P C. H e p a t i c mitochondrial function was analyzed by oxygraphs. Mitochondrial Complex-Ⅰ, Complex-Ⅱ enzyme activity, serum alanine aminotransferase(ALT), and histological injury were measured.RESULTS: Steatotic-IRI livers had a greater increase in ALT(2476 ± 166 vs 1457 ± 103 IU/L, P < 0.01) and histological injury following IRI compared to the lean liver group. Steatotic-IRI demonstrated lower Complex-Ⅰ?activity at baseline [78.4 ± 2.5 vs 116.4 ± 6.0 nmol/(min.mg protein), P < 0.001] and following IRI [28.0 ± 6.2 vs 104.3 ± 12.6 nmol/(min.mg protein), P < 0.001]. Steatotic-IRI also demonstrated impaired Complex-Ⅰ?function post-IRI compared to the lean liver IRI group. Complex-Ⅱ activity was unaffected by hepatic steatosis or IRI. Lean liver mitochondrial function was unchanged following IRI. IPC normalized ALT and histological injury in steatotic livers but had no effect on overall steatotic liver mitochondrial function or individual mitochondrial complex enzyme activities. CONCLUSION: Warm IRI impairs steatotic liver Complex-Ⅰ?activity and function. The protective effects of IPC in steatotic livers may not be mediated through mitochondria.	Michael JJ Chu Rakesh Premkumar Anthony JR Hickey Yannan Jiang Brett Delahunt Anthony RJ Phillips Adam SJR Bartlett	2016	World Journal of Gastroenterology2016，22，19：	1

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