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| 1 | The role of associating liver partition and portal vein ligation for staged hepatectomy in the management of patients with colorectal liver metastasis显示文摘Associating liver partition and portal vein ligation for staged hepatectomy(ALPPS)approach emerged as a promising surgical strategy for rapid and large hypertrophy of the future liver remnant(FLR)when a major liver resection is necessary.Colorectal liver metastasis(CRLM)is their main indication.However,the promising results published so far,are very difficult to interpret since they usually focus on the technique and not on the underlying disease.Moreover,they are usually made up of complex populations,which received different chemotherapy schemes,with the ALPPS technical variations implemented over time and without consistent long-term follow-up results as well.Whereby,its role in CRLM should be analyzed as carefully as possible to indicate and select the best candidates who will benefit the most from this approach.We conducted a computerized search using PubMed and Google Scholar for reports published so far,using mesh headings and keywords related to the ALPPS and CRLM. | Juan Glinka Victoria Ardiles Juan Pekolj Eduardo de Santibañes Martin de Santibañes | 2020 | Hepatobiliary Surgery and Nutrition2020，9，6： | 5 |
| 2 | Intraoperative Management and Repair of Bile Duct Injuries Sustained during 10,123 Laparoscopic Cholecystectomies in a High-Volume Referral Center显示文摘 | Juan Pekolj Fernando A. Alvarez Martin Palavecino Rodrigo Sánchez Clariá Oscar Mazza Eduardo de Santiba?es | 2013 | Journal of the American College of Surgeons2013，，5： | 3 |
| 3 | Molecular mechanisms underlying the neuroprotection of environmental enrichment in Parkinson’s disease显示文摘Parkinson’s disease is the most common movement disorder,affecting about 1%of the population over the age of 60 years.Parkinson’s disease is characterized clinically by resting tremor,bradykinesia,rigidity and postural instability,as a result of the progressive loss of nigrostriatal dopaminergic neurons.In addition to this neuronal cell loss,Parkinson’s disease is characterized by the accumulation of intracellular protein aggregates,Lewy bodies and Lewy neurites,composed primarily of the proteinα-synuclein.Although it was first described almost 200 years ago,there are no disease-modifying drugs to treat patients with Parkinson’s disease.In addition to conventional therapies,non-pharmacological treatment strategies are under investigation in patients and animal models of neurodegenerative disorders.Among such strategies,environmental enrichment,comprising physical exercise,cognitive stimulus,and social interactions,has been assessed in preclinical models of Parkinson’s disease.Environmental enrichment can cause structural and functional changes in the brain and promote neurogenesis and dendritic growth by modifying gene expression,enhancing the expression of neurotrophic factors and modulating neurotransmission.In this review article,we focus on the current knowledge about the molecular mechanisms underlying environmental enrichment neuroprotection in Parkinson’s disease,highlighting its influence on the dopaminergic,cholinergic,glutamatergic and GABAergic systems,as well as the involvement of neurotrophic factors.We describe experimental pre-clinical data showing how environmental enrichment can act as a modulator in a neurochemical and behavioral context in different animal models of Parkinson’s disease,highlighting the potential of environmental enrichment as an additional strategy in the management and prevention of this complex disease. | Tamara Andrea Alarcón Sarah Martins Presti-Silva Ana Paula Toniato Simões Fabiola Mara Ribeiro Rita Gomes Wanderley Pires | 2023 | Neural Regeneration Research2023，18，7： | 2 |
| 4 | Nontreponemal tests in the diagnosis of neurosyphilis: an evaluation of the Venereal Disease Research Laboratory (VDRL) and the Rapid Plasma Reagin (RPR) tests显示文摘 | Castro R Prieto ES da Luz Martins Pereira F | 2008 | J Clin Lab Anal2008，22，4： | 1 |
| 5 | Parkin,a gene implicated in autosomal recessive juvenile parkinsonism,is a candidate tumor suppressor gene on chromosome 6q25-q27显示文摘 | Cesari R Martin ES Calin GA | 2003 | Proc Natl Acad Sci USA2003，100，10： | 1 |
| 6 | Peripheral Blood Mononuclear Cells Immunophenotyping in Pulmonary Tuberculosis Patients before and after Treatment显示文摘 | Warly Barcelos Olindo Assis Martins‐Filho Tania Mara Pinto Dabés Guimar?es Márcio Hamilton Prostzner Oliveira Silvana Spíndola‐de‐Miranda Beatriz Nascimento Carvalho Vicente de Paulo Coelho Peixoto Toledo | 2006 | Microbiology and Immunology2006，，： | 1 |
| 7 | C3G mediated suppression of malignant transformation involves activation of PP2A phosphatases at the subcnrtical actin cytoskeleton 显示文摘 | Martin ES Santos E Guerrero C | 2007 | Exp Cell Res2007，313，18： | 1 |
| 8 | The BCSC-1 locus at chromosome 11 q23-q24 is a candidate tumor suppressor gene 显示文摘 | Martin ES Cesari R Pentimalli F | 2003 | Proe Natl Acad Sci USA2003，100，11： | 1 |
| 9 | Confirmation of a clini- cal diagnosis of Prader - Willi syndrome by fluorescence in situ hy- bridization (FISH) 显示文摘 | Joseph A Martin ES McConnack BJ | 1995 | Del Med J1995，67，12： | 1 |
| 10 | Apoptosis as a possible cause of gradual devel-opment of complete heart block and fatal arrhythmias associated with absence of the ANnode sinus node and internodal pathways显示文摘 | James TN Martin ES Willis III PW | 1996 | Circulation1996，9，3： | 1 |
| 11 | The BCSC-1 locus at chromosome 11q23-q24 is a candidate tumor suppressor gene显示文摘 | Martin ES Cesari R Pentimalli F | 2003 | PNAS2003，100，11： | 1 |
| 12 | Nontreponemal tests in the diagnosis of neurosyphilis: an evaluation of the Venereal Disease Research Laboratory (VDRL) and the Rapid Plasma Reagin (RPR) lests显示文摘 | Castro R Prieto ES da Luz Martins Pereira F | 2008 | J Clin Lab Anal2008，22，4： | 1 |
| 13 | The BCSC-1 locus at chromosome 1 l q23-q24 is a candidate tumor suppressor gene 显示文摘 | Martin ES Cesari R Pentimalli F | 2003 | Proc Natl Acad Sci USA2003，100，11： | 1 |
| 14 | The BCSC-1 locus atchromosome 11 q23-q24 is a candidate tumor suppressor gene显示文摘 | Martin ES Cesari R Pentimalli F | 2003 | Proc Natl Acaid Sci USA2003，100，11： | 1 |
| 15 | Assessing arsenic, cadmium, and lead contents in major crops in Brazil for food safety purposes显示文摘 | Ana Paula Branco Corguinha Guilherme Amaral de Souza Veridiana Cardoso Gon?alves Camila de Andrade Carvalho Willian Eduardo Amaral de Lima Fábio Aurélio Dias Martins Celso Hideto Yamanaka Eros Artur Bohac Francisco Luiz Roberto Guimar?es Guilherme | 2014 | Journal of Food Composition and Analysis2014，，： | 1 |
| 16 | The BCSC-1 locus at chromosome 11q23-q24 is a candidate tumor suppressor gene显示文摘 | Martin ES Cesari R Pentimalli F et a1 | 2003 | PNAS2003，100，11： | 1 |
| 17 | Prevalence and associated factors of subclinical hypereortisolism in patients with resistant hypertension显示文摘 | Martins LC Muxfeldt ES Salles GF | | 0，，5： | 1 |
| 18 | Transposable elementsgenerate novel spatial patterns of gene expression in Antirrhinummajus 显示文摘 | Coen ES Carpenter R Martin C | 1986 | Cell1986，47，2： | 1 |
| 19 | Improving axonal growth and functional recovery after experimental spinal cord injury by neutralizing myelin associated inhibitors显示文摘 | Fouad K Dietz V Martin ES | 2001 | Brain Research Review2001，，36： | 1 |
| 20 | Evaluation of Hepatobiliary Excretion and Enterobiliary Reflux in Rats with Biliary Obstruction Submitted to Bilioduodenal or Biliojejunal Anastomosis显示文摘 | Robson Azevedo Dutra José Sebasti?o Santos Whemberton Martins Araújo Marcus Vinicius Sim?es Aline Nazareth Paiva Paix?o-Becker Luciano Neder | 2008 | Digestive Diseases and Sciences2008，，4： | 1 |
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