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| 1 | Further compatibility tests of the entomopathogenic fungus Lecanicillium muscarium with conventional insecticide products for control of sweetpotato whitefly, Bemisia tabaci on poinsettia plants显示文摘为鸡蛋的控制和甘薯白人的第二个中间形态阶段为 24 h 跟随直接暴露到杀虫药剂 Majestik,水花油, Agri-50E, Savona 和天王卫四的 entomopathogenic 真菌 Lecanicillium muscarium 的孢子萌芽上的效果飞, Bemisia tabaci,被决定。到 Agri-50E 和天王卫四的暴露被可接受的孢子萌芽跟随。L 的传染性率。一品红叶子上的 muscarium 也面对杀虫药剂的干燥残余被调查。B 的控制的没有重要有害效果 on the levels。tabaci 与用于没有残余的叶子的真菌相比被记录。有真菌的化学药品 Savona,水花油和 Majestik 的顺序的申请都生产了第二中间形态 B 的死亡。超过 90% 的 tabaci。有 L 的这些化学药品的加入。muscarium 进为 B 的综合防治法节目。tabaci 被讨论。 | Andrew G. S. Cuthbertson Lisa F. Blackburn Phil Northing Weiqi Luo Raymond J. C. Cannon Keith F. A. Waiters | 2008 | Insect Science2008，15，4： | 5 |
| 2 | 蜂胶对成纤维细胞、单核巨噬细胞系基质金属蛋白酶MMP-9表达的抑制作用显示文摘目的探讨蜂胶对糖尿病伤口愈合的作用及其机制。方法采用荧光电泳法,检测不同葡萄糖浓度(5、25mmol/L)和不同蜂胶浓度(10、50、100、200μg/ml)处理的体外培养的人成纤维细胞及单核巨噬细胞系的基质金属蛋白酶MMP9蛋白的表达。结果高浓度葡萄糖(25mmol/L)状态下,MMP9表达增加,但与低浓度(5mmol/L)比较,差异无统计学意义。蜂胶可显著降低MMP9的表达,并呈剂量依赖性(P<0.05)。结论高糖能增加单核巨噬细胞系及成纤维细胞MMP9的表达,可能会影响糖尿病患者伤口的愈合。蜂胶可抑制这些细胞MMP9的表达,有益于细胞基质的积聚,从而有益于糖尿病患者伤口的愈合。 | 贾军宏 Min Danqing Lisa Luo DK Yue SV McLennan | 2006 | 解放军医学杂志2006，31，6： | 2 |
| 3 | Biomarkers and Mechanisms of FANCD2 Function显示文摘 | Henning Willers Lisa A. Kachnic Chen-Mei Luo Li Li Martin Purschke Kerstin Borgmann Kathryn D. Held Simon N. Powell Lisa Wiesmuller | 2008 | Journal of Biomedicine and Biotechnology2008，，： | 1 |
| 4 | Telaprevir Twice Daily is Noninferior to Telaprevir Every 8 hrs for Patients with Chronic Hepatitis C显示文摘 | Maria Buti Kosh Agarwal Yves Horsmans William Sievert Ewa Janczewska Stefan Zeuzem Lisa Nyberg Robert S. Brown Christophe Hezode Mario Rizzetto Raymundo Parana Sandra De Meyer Ralph De Masi Donghan Luo Kirk Bertelsen James Witek | 2013 | Gastroenterology2013，，： | 1 |
| 5 | A Biological Global Positioning System: Considerations for Tracking Stem Cell Behaviors in the Whole Body显示文摘 | Shengwen Calvin Li Lisa May Ling Tachiki Jane Luo Brent A. Dethlefs Zhongping Chen William G. Loudon | 2010 | Stem Cell Reviews and Reports2010，，2： | 1 |
| 6 | SARS-CoV-2 infection causes immunodeficiency in recovered patients by downregulating CD19 expression in B cells via enhancing B-cell metabolism显示文摘The SARS-CoV-2 infection causes severe immune disruption.However,it is unclear if disrupted immune regulation still exists and pertains in recovered COVID-19 patients.In our study,we have characterized the immunephe no type of B cells from 15 recovered COVID-19 patients,and found that healthy controls and recovered patients had similar B-cell populations before and after BCR stimulation,but the frequencies of PBC in patients were significantly increased when compared to healthy controls before stimulation.However,the percentage of unswitched memory B cells was decreased in recovered patients but not changed in healthy controls upon BCR stimulation.Interestingly,we found that CD19 expression was significantly reduced in almost all the B-cell subsets in recovered patients.Moreover,the BCR signaling and early B-cell response were disrupted upon BCR stimulation.Mechanistically,we found that the reduced CD19 expression was caused by the dysregulation of cell metabolism.In conclusion,we found that SARS-CoV-2 infection causes immunodeficiency in recovered patients by downregulating CD19 expression in B cells via enhandng B-cell metabolism,which may provide a new intervention target to cure COVID-19. | Yukai Jing Li Luo Ying Chen Lisa S.Westerberg Peng Zhou Zhiping Xu Andres A.Herrada Chan-Sik Park Masato Kubo Heng Mei Yu Hu Pamela Pui-Wah Lee Bing Zheng Zhiwei Sui Wei Xiao Quan Gong Zhongxin Lu Chaohong Liu | 2021 | Signal Transduction and Targeted Therapy2021，6，10： | 0 |
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