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| 1 | NOx Emission Reduction Technology for Marine Engine Based on Tier-Ⅲ:A Review显示文摘The development of maritime trade has greatly promoted the development of diesel engines.However,with the increasingly serious environmental problems,more and more attention has been paid to the exhaust emissions of high-power marine diesel engines.The restrictions on SOx have been implemented globally,and the limitation of the NO,will be the next priority.This paper illustrates(a)Principle and research progress of NOx emissions-reduction technology of marine diesel engine;(b)Summary of advantages and disadvantages among various reduction technologies and their reduction effects;(c)The application effect of mainstream technology on board.Firstly,since exhaust gas recirculation(EGR)can achieve Tier-Ⅲ directly from Tier-Ⅰ without considering the increased fuel consumption.It is deemed as the most promising technology to reduce emissions by controlling combustion condition.However,EGR has shortcomings of excessive increase in ftiel consumption and generation of waste water,which need to be solved immediately.Secondly,selective catalytic reduction(SCR)is the most effective and straightforward means to achieve Tier-Ⅲ.Despite of the continuous optimization of SCR unit volume,the problem of scrap catalyst seriously limits its wide application.How to match the supercharger more efficiently is a key factor in choosing between high and low pressure SCR.Thirdly,nature gas(NG)engines are capable of achieving a reduction in NOx,but in order to meet the requirements of Tier-Ⅲ,it still needs to be assisted by other technologies.The emissions of hydrocarbon(HC)and CO in NG engines are huge defects that must be solved.Lastly,technologies such as the Miller cycle,Two-stage supercharging and mixed-water combustion can also reduce emissions but were rarely used alone.These technologies can be combined with EGR,SCR and NG engines to optimize the enginesJ economy and emission characteristics. | LU Xiuwei GENG Peng CHEN Yunyue | 2020 | Journal of Thermal Science2020，29，5： | 2 |
| 2 | The impact of UV treatment on microbial control and DBPs formation in full-scale drinking water systems in northern China显示文摘To manage potential microbial risks and meet increasingly strict drinking water health standards,UV treatment has attracted increasing attention for use in drinking water systems in China.However,the effects of UV treatment on microbial control and disinfection byproducts(DBPs)formation in real municipal drinking water systems are poorly understood.Here,we collected water samples from three real drinking water systems in Beijing and Tianjin to investigate the impacts of UV treatment on microbial control and DBP formation.We employed heterotrophic plate count(HPC),flow cytometry(FCM),quantitative PCR analysis,and high-throughput sequencing to measure microorganisms in the samples.Different trends were observed between HPC and total cell count(measured by FCM),indicating that a single indicator could not reflect the real degree of biological re-growth in drinking water distribution systems(DWDSs).A significant increase in the 16S rRNA gene concentration was observed when the UV system was stopped.Besides,the bacterial community composition was similar at the phylum level but differed markedly at the genera level among the three DWDSs.Some chlorine-resistant bacteria,including potential pathogens(e.g.,Acinetobacter)showed a high relative abundance when the UV system was turned off.It can be concluded that UV treatment can mitigate microbial re-growth to some extent.Finally,UV treatment had a limited influence on the formation of DBPs,including trihalomethanes,haloacetic acids,and nitrogenated DBPs.The findings of this study may help to understand the performance of UV treatment in real drinking water systems. | Xiuwei Ao Zhongyun Chen Simiao Li Chen Li Zedong Lu Wenjun Sun | 2020 | Journal of Environmental Sciences2020，32，1： | 2 |
| 3 | Fabrication and Properties of Graphene Oxide/Sulfonated Polyethersulfone Layer-by-layer Assembled Polyester Fiber Composite Proton Exchange Membranes显示文摘 | HUANG Lihua HE Yao JIN Lying HOU Xiuwei MIAO Luyang LU Changli | 2018 | Chemical Research in Chinese Universities2018，34，2： | 2 |
| 4 | Cerebral small vessel disease caused by PLOD3 mutation:Expanding the phenotypic spectrum of lysyl hydroxylase-3 deficiency显示文摘Introduction:Pathogenic variants inPLOD3,encoding lysyl hydroxylase-3(LH3),can cause a hereditary connective tissue disorder that has rarely been reported.It is a multi-system disease,presenting with craniofacial dysmorphisms,skeletal and eye manifestations,sensorineural hearing loss,and variable skin manifestations.Severe central nervous system involvement has not been reported.Case presentation:A 10-month-old girl was admitted with development delay and clustered epileptic spasms.Hypertelorism,an upturned nose,and low-set ears were noted in physical examination.Cerebral magnetic resonance imaging showed multiple intracranial malacias and bleeding foci,extensive abnormal signals in the white matter,and obvious brain atrophy,which was consistent with cerebral small vessel disease(SVD).Electroencephalography suggested hypsarrhythmia.The vertebrae were flattened.The distal end of the metacarpal bone in the left hand was irregular.She was diagnosed with West syndrome.Whole-exome sequencing revealed a novel homozygous variant of c.12161218delCTC(p.L406del)inPLOD3,which was found to be inherited from her heterozygous parents.Conclusion:We report a patient with pathogenicPLOD3 mutation who presented with cerebral SVD.This report expands the phenotypic spectrum of LH3 deficiency. | Ji Zhou Weixing Feng Xiuwei Zhuo Wenting Lu Junling Wang Fang Fang Xiaohui Wang | 2022 | Pediatric Investigation2022，6，3： | 0 |
| 5 | Absent in melanoma 2 attenuates proliferation and migration and promotes apoptosis of human colorectal cancer cells by activating P38MAPK signaling pathway显示文摘Colorectal cancer(CRC)stands among the top prevalent cancers worldwide and holds a prominent position as a major contributor to cancer-related mortality globally.Absent in melanoma 2(AIM2),a constituent of the interferoninducible hematopoietic interferon-inducible nuclear antigens with 200 amino acid repeats protein family,contributes to both cancer progression and inflammasome activation.Despite this understanding,the precise biological functions and molecular mechanisms governed by AIM2 in CRC remain elusive.Consequently,this study endeavors to assess AIM2’s expression levels,explore its potential antitumor effects,elucidate associated cancer-related processes,and decipher the underlying signaling pathways in CRC.Our findings showed a reduced AIM2 expression in most CRC cell lines.Elevation of AIM2 levels suppressed CRC cell proliferation and migration,altered cell cycle by inhibiting G1/S transition,and induced cell apoptosis.Further research uncovered the participation of P38 mitogen-activated protein kinase(P38MAPK)in AIM2-mediated modulation of CRC cell apoptosis and proliferation.Altogether,our achievements distinctly underscored AIM2’s antitumor role in CRC.AIM2 overexpression inhibited proliferation and migration and induced apoptosis of CRC cells via activating P38MAPK signaling pathway,indicating AIM2 as a prospective and novel therapeutic target for CRC. | ZHI ZHANG XIAOSONG LI YING ZHANG HAO ZHU ZHENGUO QIAO YANG LU XIUWEI MI HUIHUA CAO GENHAI SHEN SONGBING HE | 2024 | Oncology Research2024，32，2： | 0 |
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