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| 1 | Fine-tune chiroptical activity in discrete chiral Au nanorods显示文摘Accurate researches on the surface plasmon resonance(SPR)-based applications of chiral plasmonic metal nanoparticles(NPs)still remain a great challenge.Herein,a series of chiral plasmonic metal NPs,e.g.,chiral Au nanorods(c-Au NRs),c-Au@Ag core–shell,and c-Au@TiO_(2) core–shell NRs,with different chiroptical activities have been produced.Plasmonic circular dichroism(PCD)bands of c-Au NRs can be precisely tailored by tuning the longitudinal SPR(LSPR)and amount of Au NRs as seeds.Besides,a shift of PCD bands within ultraviolet–visible–near infrared ray(UV–vis–NIR)region can also be achieved through the functionalization of a shell of another metal or semiconductor.Interestingly,chirality transfer from c-Au core to Ag shell leads to new PCD bands at the near-UV region.The tuning of PCD bands and chirality transfer are confirmed by our developed theoretical model.Developing chiral Au NRs-based chiral plasmonic nanomaterials with tunable chiroptical activities will be helpful to understand the structure-direct PCD and to extend circularly polarized-based applications. | Guangchao Zheng Sulin Jiao Wei Zhang Shenli Wang Qinghua Zhang Lin Gu Weixiang Ye Junjun Li Xiaochen Ren Zhicheng Zhang Kwok-yin Wong | 2022 | Nano Research2022，15，7： | 2 |
| 2 | BEAM BROADENING SYNTHESIS FOR SAR ANTENNA ARRAY BASED ON PROJECTION MATRIX ALGORITHM显示文摘A new beam broadening synthesis technique for Synthetic Aperture Radar(SAR) antenna array, namely Projection Matrix Algorithm(PMA) is presented. The theory of PMA is introduced firstly, and then the iterative renewed manner is improved to resolve the unbalance problem under amplitude and phase control. In order to validate the algorithm correct and effective, an actual engineering application example is investigated. The beam synthesis results of 1.0~4.5 times broadening under the phase only control and the amplitude and phase control using improved PMA are given. The results show that the beam directivity, the beam broadening, and the side-lobe level requirements were met. It is demonstrated that the improved PMA was effective and feasible for SAR application. | Jiao Junjun Sun Huifeng | 2014 | Journal of Electronics(China)2014，31，5： | 0 |
| 3 | Honokiol attenuates acetaminophen-induced acute liver injury by inhibiting hepatic CYP1A2 activity and improving liver mitochondrial dysfunction显示文摘Objective:Acetaminophen(APAP)overdose is a common cause of liver injury.This study aimed to investigate the protective effect of honokiol(Hon)against APAP-induced hepatotoxicity and its potential mechanism.Methods:C57BL/6 mice were administrated with Hon(10 and 30 mg/kg)after APAP(300 mg/kg)treatment.On 1.5 h and 5 h after Hon treatment,mice were sacrificed.Serum and liver were collected.And then,liver injury-related indexes,APAP metabolism-related indexes,mitochondrial respiratory chain function-related indexes,and mitochondrial membrane function-related protein expression were evaluated.Results:It was found that Hon significantly decreased serum alanine aminotransferase(ALT)/aspartate aminotransferase(AST)activity and glutathione(GSH)depletion,increased hepatic catalase(CAT)and GSH peroxidase(GSH-Px)activities,reduced hepatic MDA and 3-nitrotyrosine contents,inhibited hepatic CYP1A2 activity and APAP protein adducts(APAP-CYS)formation.Meanwhile,oxidative phosphorylation capacity of complex I and electron transfer capacity of complex IV in mitochondrial respiratory chain was increased,whereas the release of H2O2 in the mitochondria was decreased following Hon treatment.Furthermore,Hon markedly down-regulated p-JNK in both cytosol and mitochondria,and obviously inhibited the release of apoptosis inducing factor(AIF)and endonuclease G(EndoG)from mitochondria to cytosol.Conclusion:Hon alleviated APAP-induced liver injury through the following pathways:Reducing the production of APAP-CYS by inhibiting CYP1A2 activity;Ameliorating hepatic oxidative stress by increasing the levels of hepatic CAT,GSH-Px and GSH;Improving mitochondrial respiratory chain function by promoting oxidative phosphorylation capacity of complex I and electron transfer capacity of complex IV;Improving the function of mitochondrial membrane by inhibiting p-JNK and its translocation to mitochondria,thereby reducing the release of AIF and EndoG. | Xiaolei Miao Chengting Jin Jiao Liu Junjun Wang Yong Chen | 2023 | Chinese Herbal Medicines2023，15，2： | 0 |
| 4 | Tet酶调控端粒和基因组稳定性的作用和机制显示文摘Tets家族催化5-甲基胞嘧啶转变为5-羟甲基胞嘧啶。在小鼠胚胎干细胞中单独干扰Tet1或/和Tet2,伴随着端粒重组下降,导致端粒长度缩短和基因组稳定性异常。 | Jiao Yang Renpeng Guo Hua Wang Xiaoying Ye Zhongcheng Zhou Jiameng Dan Haiying Wang Peng Gong Wei Deng Yu Yin Shi Qing Mao Lingbo Wang Junjun Ding Jinsong Li David L.Keefe Meelad M.Dawlaty Jianlong Wang Guo Liang Xu 刘林 | 2017 | 科学新闻2017，19，4： | 0 |
| 5 | Tolerance Limits Under Gamma Mixtures:Application in Hydrology显示文摘In this study,the authors proposed upper tolerance limits for the gamma mixture distribution based on generalized fiducial inference,and an MCMC simulation is performed to sample from the generalized fiducial distributions.The simulation results and a real hydrological data example show that the proposed tolerance limits are more efficient. | JIAO Junjun CHENG Weihu | 2023 | Journal of Systems Science & Complexity2023，36，3： | 0 |
| 6 | Arabidopsis RNA polymerase Ⅱ C-terminal domain phosphatase-like 1 targets mitogen-activated protein kinase cascades to suppress plant immunity显示文摘Mitogen-activated protein kinase(MAPK) cascades play pivotal roles in plant defense against phytopathogens downstream of immune receptor complexes. The amplitude and duration of MAPK activation must be strictly controlled, but the underlying mechanism remains unclear. Here, we identified Arabidopsis CPL1(C-terminal domain phosphatase-like 1)as a negative regulator of microbe-associated molecular pattern(MAMP)-triggered immunity via a forward-genetic screen. Disruption of CPL1 significantly enhanced plant resistance to Pseudomonas pathogens induced by the bacterial peptide fg22. Furthermore, fg22-induced MPK3/MPK4/MPK6 phosphorylation was dramatically elevated in cpl1 mutants but severely impaired in CPL1 overexpression lines, suggesting that CPL1 might interfere with fg22-induced MAPK activation. Indeed, CPL1 directly interacted with MPK3 and MPK6, as well as the upstream MKK4 and MKK5. A firefy luciferase-based complementation assay indicated that the interaction between MKK4/MKK5 and MPK3/MPK6 was significantly reduced in the presence of CPL1. These results suggest that CPL1 plays a novel regulatory role in suppressing MAMP-induced MAPK cascade activation and MAMP-triggered immunity to bacterial pathogens. | Junjun Wei Wei Sun Xinhang Zheng Shanshan Qiu Shuangyu Jiao Kevin Babilonia Hisashi Koiwa Ping He Libo Shan Wenxian Sun Fuhao Cui | 2023 | Journal of Integrative Plant Biology2023，65，10： | 0 |
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