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24篇 您的检索式：作者名="Edsfeldt"

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	题名	作者	年代	出处	被引量
1	干扰素调节因子5通过损伤胞葬作用控制动脉粥样硬化病变中的坏死核心形成显示文摘髓系细胞是动脉粥样硬化病变发展和易损斑块形成的核心。动脉吞噬细胞吸收凋亡细胞(胞葬作用)的能力受损,可促进病变生长并建立坏死核心。干扰素调节因子(interferon regulatory factor,IRF)5是髓系细胞功能的重要调节因子。该研究试图确定IRF5是否影响动脉粥样硬化病变的形成和表型。方法:通过两种互补模型,研究IRF5在动脉粥样硬化中的作用:	Seneviratne AN Edsfeldt AO Cole JE Kassiteridi C Swart M Park I Green P Khoyratty TE Saliba DG Goddard ME Sansom SN Goncalves I Krams R Udalova IA Monaco C 刘莉叶鹏	2017	中华高血压杂志2017，25，8：	9
2	High Plasma Levels of Galectin-3 Are Associated with Increased Risk for Stroke after Carotid Endarterectomy 显示文摘	Edsfeldt A Bengtsson E Asciutto G	2016	Cerebrovasc Dis2016，41，34：	1
3	Evidence supporting akey role of Lp-PLA2-generated lysophosphatidylcholine in hu-man atherosclerotic plaque inflammation显示文摘	Goncalves I Edsfeldt A Ko NY	2012	Arterioscler ThrombVasc Biol2012，32，6：	1
4	Low elastin content of carotid plaques is associated with increased risk of ipsilateral stroke显示文摘	Asciutto G Dias N V Edsfeldt A	2015	PLoS One2015，10，01：	1
5	Low elastin content of carotid plaques is associated with increased risk of ipsilateral stroke 显示文摘	Asciutto G Dias NV Edsfeldt A	2015	PLoS One2015，10，3：	1
6	Soluble urokinase plasminogen ac- :ivator receptor is associated with inflammation in the vulnerable human athero-sclerotic plaque显示文摘	Edsfeldt A Nitulescu M Grufman H	2012	Stroke2012，43，12：	1
7	Evidence supporting a key role o: Lp-PLA2-generated lysophosphatidylcholine in human atherosclerotic plaque inflammation显示文摘	GONCALVES I EDSFELDT A KO N Y	2012	Arterioscler Thromb Vasc Biol2012，32，：	1
8	Plasma levels of high-sensitive C-reactive protein do not correlate with inflammato- ry activity in carotid atherosclerotic plaques 显示文摘	Grufman H Goncalves I Edsfeldt A	2014	Intern Med2014，275，2：	1
9	Evidence Supporting a Key Role of Lp-PLA2-Generated Lysophosphatidylcholine in Human Atherosclerotic Plaque Inflammation显示文摘	Isabel Gon?alves Andreas Edsfeldt Na Young Ko Helena Grufman Katarina Berg Harry Bj?rkbacka Mihaela Nitulescu Ana Persson Marie Nilsson Cornelia Prehn Jerzy Adamski Jan Nilsson	2012	Arteriosclerosis, Thrombosis, and Vascular Biology2012，，6：	1
10	Evidence supporting a key role of Lp-PLA2-generated lysophosphatidylcholine in human atherosclerotic plaque inflammation显示文摘	GONCALVES I EDSFELDT A KO N Y	2012	Arterioscter Thromb Vasc Biol2012，32，：	1
11	Evidence for altered in-flammatory and repair responses in symptomatic arotid plaques from eld-erly patients显示文摘	Grufman H Schiopu A Edsfeldt A	2014	Atherosclerosis2014，237，1：	1
12	Treatment with beta-blockers is associat- ed with lower levels of Lp-PLA2 and suPAR in carotid plaques 显示文摘	Asciutto G Edsfeldt A Dias NV	2013	Cardiovasc Patho12013，22，6：	1
13	Plasma levels of high-sensitive C-reactive protein do not correlate with in flammatory activity in carotid atherosclerotic plaques显示文摘	Grufman H Goncalves I Edsfeldt A	2013	J Intern Med2013，7，2：	1
14	Treatment with beta-blockers is associated with lower levels of LpPLA2 and suPAR in carotid plaques 显示文摘	Asciutto G Edsfeldt A Dias NV	2013	Cardiovasc Pathol2013，22，6：	1
15	Soluble urokinase plasminogen activator receptor is associated with inflammation in the vulnerable human atherosclerotic plaque 显示文摘	Edsfeldt A Nituleseu M Grufman H	2012	Stroke2012，43，12：	1
16	Evidence supporting a key role of Lp-PLAz-generated lysophosphatidylcholine in human atherosclerotic plaque inflammation 显示文摘	Gongalves I Edsfeldt A Ko NY	2012	Arterioscler Thromb Vasc Binl2012，32，：	1
17	Evidencesupporting a key role of Lp-PLA2-generated lysophosphatidyl-choUne in human atherosclerotic plaque inflammation 显示文摘	GONCALVES I EDSFELDT A KO N Y	2012	Arteri-oscler Thromb Vase Biol2012，32，6：	1
18	Evidence supporting a key role of Lp-PLA2-generated lysophosphatidylcholine in human atherosclerotic plaque inflammation 显示文摘	Goncalves I Edsfeldt A Ko NY	2012	Arterioscler Thromb Vasc Biol2012，32，6：	1
19	Evidence supporting a keyrole of Lp-PLA2-generated lysophosphatidyloholine in human ath-erosclerotioplaque inflammation显示文摘	Goncalves I Edsfeldt A Ko NY	2012	Arterioscler Thromb Vase Bi-ol2012，32，6：	1
20	Plasma levels of highsensitive C reactive protein do not correlate with inflammatoryactivity in carotid atherosclerotic plaque 显示文摘	Grufman H Goncalves I Edsfeldt A	2014	Intern Med2014，275，2：	1

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