|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | 2型糖尿病患者非酒精性脂肪肝患病率的回顾性分析显示文摘收集550名T2DM住院患者的临床、肝脏彩超及生化指标检查资料,统计非酒精性脂肪肝(NAFL)的患病率,并分析其危险因素。发现T2DM中NAFL的患病率为42%,且男性患病率明显高于女性(P<0.05)。BMI、WC、FIns、TG、HOMA-IR是T2DM合并NAFL的独立危险因素。 | 赵琳 Dina Shrestha 张霞 王颖 孙崴 张萍 苏本利 | 2008 | 中国糖尿病杂志2008，16，9： | 36 |
| 2 | Mesenchymal stem cells and the neuronal microenvironment in the area of spinal cord injury显示文摘Cell-based technologies are used as a therapeutic strategy in spinal cord injury(SCI). Mesenchymal stem cells(MSCs), which secrete various neurotrophic factors and cytokines, have immunomodulatory, anti-apoptotic and anti-inflammatory effects, modulate reactivity/phenotype of astrocytes and the microglia, thereby promoting neuroregeneration seem to be the most promising. The therapeutic effect of MSCs is due to a paracrine mechanism of their action, therefore the survival of MSCs and their secretory phenotype is of particular importance. Nevertheless, these data are not always reported in efficacy studies of MSC therapy in SCI. Here, we provide a review with summaries of preclinical trials data evaluating the efficacy of MSCs in animal models of SCI. Based on the data collected, we have tried(1) to establish the behavior of MSCs after transplantation in SCI with an evaluation of cell survival, migration potential, distribution in the area of injured and intact tissue and possible differentiation;(2) to determine the effects MSCs on neuronal microenvironment and correlate them with the efficacy of functional recovery in SCI;(3) to ascertain the conditions under which MSCs demonstrate their best survival and greatest efficacy. | Yana O.Mukhamedshina Olga A.Gracheva Dina M.Mukhutdinova Yurii A.Chelyshev Albert A.Rizvanov | 2019 | Neural Regeneration Research2019，14，2： | 25 |
| 3 | Noninvasive investigations for non alcoholic fatty liver disease and liver fi brosis显示文摘Non-alcoholic fatty liver disease (NAFLD) includes a spectrum of diseases that have insulin resistance in common and are associated with metabolic conditions such as obesity, type 2 diabetes mellitus, and dyslipidemia. NAFLD ranges from simple liver steatosis, which follows a benign course, to nonalcoholic steatohepatitis (NASH), a more severe entity, with necroinflmmation and f ibrosis, which can progress to cryptogenic cirrhosis and end-stage liver disease. Liver biopsy remains the gold standard for evaluating the degree of hepatic necroinflammation and f ibrosis; however, several noninvasive investigations, such as serum biomarkers, have been developed to establish the diagnosis and also to evaluate treatment response. These markers are currently neither available in all centers nor validated in extensive studies. Examples include high-sensitivity C reactive protein and plasma pentraxin 3, which are associated with extensive liver f ibrosis in NASH. Interleukin-6 correlates with inflammation, and cytokeratin-18 represents a marker of hepatocyte apoptosis (prominent in NASH and absent in simple steatosis). Tissue polypep-tide specif ic antigen seems to have a clinical utility in the follow-up of obese patients with NASH. | Carmen Fierbinteanu-Braticevici Ion Dina Ana Petrisor Laura Tribus Lucian Negreanu Catalin Carstoiu | 2010 | World Journal of Gastroenterology2010，16，38： | 21 |
| 4 | Then and now: The progress in hepatitis B treatment over the past 20 years显示文摘The ultimate goals of treating chronic hepatitis B(CHB)is prevention of hepatocellular carcinoma(HCC)and hepatic decompensation.Since the advent of effective antiviral drugs that appeared during the past two decades,considerable advances have been made not only in controlling hepatitis B virus(HBV)infection,but also in preventing and reducing the incidence of liver cirrhosis and HCC.Furthermore,several recent studies have suggested the possibility of reducing the incidence of recurrent or new HCC in patients even after they have developed HCC.Currently,six medications are available for HBV treatment including,interferon and five nucleoside/nucleotide analogues.In this review,we will examine the antiviral drugs and the progresses that have been made with antiviral treatments in the field of CHB. | Dina Halegoua-De Marzio Hie-Won Hann | 2014 | World Journal of Gastroenterology2014，20，2： | 19 |
| 5 | Seminal plasma anti-Müllerian hormone level correlates with semen parameters but does not predict success of testicular sperm extraction (TESE)显示文摘瞄准:估计精液的血浆 anti-Muellerian 在肥沃、不肥沃的男性的荷尔蒙(AMH ) levalrelationships。方法:84 个男盒子被学习并且把组划分了成四:肥沃的 normozoospermia (n = 16 ) , oligoastheno-teratozoospermia (n = 15 ) ,妨碍的精子缺乏(OA )(n = 13 ) 并且非妨碍的精子缺乏(NOA )(n = 40 ) 。Conventionalsemen 分析为所有情况被做。阴囊的活体检视在 NOA 情况中为阴囊的精子抽取(TESE ) 用完了组织病理学说和新鲜织物检查。NOA 组根据 TESE 结果被细分进失败的 TESE (n = 19 ) 并且成功的 TESE (n = 21 ) 。Seminalplasma AMH 被酶估计连接免疫吸着剂试金(ELISA ) 和浆液小囊的刺激荷尔蒙(FSH ) 仅仅由放射性免疫测定(RIA ) 在 NOA 情况中被估计。结果:Meanseminal AMH 比在有意义的 oligoasthenoteratozoospermia 在肥沃的组是显著地更高的(41.5 ± 10.9 pmol/L 对 30.5 ± 10.3 pmol/L, P <
0.05 ) 。精液的 AMH 没在任何 OA 病人被检测。精液的 AMH 与阴囊的体积断然被相关( r = 0.329 , P =0.005 ),精子计数( r = 0.483 , P = 0.007 ),精子活动性百分比( r = 0.419 , P = 0.021 )并且否定地与精子反常形式百分比( r =-0.413, p = 0.023 )。不重要的关联与年龄是明显的(r =-0.155, P = 0.414 ) 并且血浆 FSH (r =-0.014, P = 0.943 ) 。在 NOA 情况中,精液的 AMH 在 23/40 情况中是可检测的,他们中的 14 个是成功的 TESE (57.5%) 并且在 17/40 情况中是无法发现的,他们中的 10 个是失败的 TESE (58.2%) 。结论:精液的 plasmaAMH 是在所有 OA 盒子中是不在的一个绝对阴囊的标记。然而,精液的 AMH 在 NOA 情况中为成功的阴囊的精子检索有差的可预测性。 | Taymour Mostafa Medhat K. Amer Guirgis Abdel-Malak Taha Abdel Nsser Wael Zohdy Shedeed Ashour Dina El-Gayar Hosam H. Awad | 2007 | Asian Journal of Andrology2007，9，2： | 17 |
| 6 | Histopathology of nonalcoholic fatty liver disease显示文摘Histological analysis of liver biopsies remains a standard against which other methods of assessment for the presence and amount of hepatic injury due to nonalcoholic fatty liver disease(NAFLD) are measured.Histological evaluation remains the sole method of distinguishing steatosis from advanced forms of NAFLD,i.e.nonalcoholic steatohepatitis(NASH) and fibrosis.Included in the lesions of NAFLD are steatosis,lobular and portal inflammation,hepatocyte injury in the forms of ballooning and apoptosis,and fibrosis.However,patterns of these lesions are as distinguishing as the lesions themselves.Liver injury in adults and children due to NAFLD may have different histological patterns.In this review,the rationale for liver biopsy,as well as the histopathological lesions,the microscopically observable patterns of injury,and the differential diagnoses of NAFLD and NASH are discussed. | Elizabeth M Brunt Dina G Tiniakos | 2010 | World Journal of Gastroenterology2010，16，42： | 16 |
| 7 | New technologies to investigate the brain-gut axis显示文摘Functional gastrointestinal disorders are commonly encountered in clinical practice, and pain is their commonest presenting symptom. In addition, patients with these disorders often demonstrate a heightened sensitivity to experimental visceral stimulation, termed visceral pain hypersensitivity that is likely to be important in their pathophysiology. Knowledge of how the brain processes sensory information from visceral structures is still in its infancy. However, our understanding has been propelled by technological imaging advances such as functional Magnetic Resonance Imaging, Positron Emission To-mography, Magnetoencephalography, and Electroen-cephalography (EEG). Numerous human studies have non-invasively demonstrated the complexity involved in functional pain processing, and highlighted a number of subcortical and cortical regions involved. This review will focus on the neurophysiological pathways (primary afferents, spinal and supraspinal transmission), brain- imaging techniques and the influence of endogenous and psychological processes in healthy controls and patients suffering from functional gastrointestinal dis- orders. Special attention will be paid to the newer EEG source analysis techniques. Understanding the pheno- typic differences that determine an individual's response to injurious stimuli could be the key to understanding why some patients develop pain and hyperalgesia in response to inflammation/injury while others do not. For future studies, an integrated approach is required incorporating an individual's psychological, autonomic, neuroendocrine, neurophysiological, and genetic prof ile to def ine phenotypic traits that may be at greater risk of developing sensitised states in response to gut in? am- mation or injury. | Abhishek Sharma Dina Lelic Christina Brock Peter Paine Qasim Aziz | 2009 | World Journal of Gastroenterology2009，15，2： | 15 |
| 8 | 绩效工资的非线性影响效应及其结构优化——基于前景理论的视角显示文摘绩效工资在实践中表现出的不适和错位引发人们对传统理性分析范式的反思。文章将员工心理计量过程纳入研究视野,运用前景理论修正并推演出不同心理框架交织下绩效工资的作用机制,运用曲线拟合检验其非线性影响效应,并在此基础上对绩效工资的结构优化进行系统仿真。研究表明,绩效工资对防御型任务有正向影响,但受心理计量折扣的影响增量递减;对促进型任务有'S形'非线性影响,当比例控制在30%左右时有助于抑制对伦理动机和成就动机的挤出效应,提高激励效果。 | 马君 Van Dijk Dina | 2013 | 财经研究2013，39，4： | 15 |
| 9 | Increased resistance of drought by Trichoderma harzianum fungal treatment correlates with increased secondary metabolites and proline content显示文摘Plant secondary metabolites play vital role in plant stress response. In this study we investigated whether root colonization of tomato(Solanum lycopersicum) infected by Trichoderma harzianum leads to alterations in the biosynthesis of secondary plant metabolites including phytohormones and osmolyte proline under drought stress. Exposure of tomato to drought caused a drastic decline in plant growth and physiological parameters. Tomato inoculated with T. harzianum showed increased root and shoot growth and chlorophyll pigments as compared to uninoculated controls as well as drought stressed plants. Proline and total soluble protein content was increased in plants inoculated with T. harzianum under both normal as well as drought conditions. An obvious increase in phenol and flavonoid content was observed due to T. harzianum. In addition, T. harzianum inoculated plants maintained higher levels of growth regulators indole acetic acid, indole butyric acid, and gibberellic acid under drought stress. Improved secondary metabolites which play an important role in plant stress tolerance by T. harzianum may have coordinately worked for bringing the growth regulation by protecting membranes from reactive oxygen species(ROS) and enhance plant growth through accessing more nutrients by root system. | Mona S.Alwhibi Abeer Hashem Elsayed Fathi Abd_Allah Abdulaziz A.Alqarawi Dina Wafi K.Soliman Stephan Wirth Dilfuza Egamberdieva | 2017 | Journal of Integrative Agriculture2017，16，8： | 15 |
| 10 | 圆二色光谱、红外光谱法解析羊乳和牛乳β-酪蛋白结构及性质差异显示文摘羊乳β-酪蛋白比牛乳β-酪蛋白更容易被婴幼儿消化吸收,主要原因是二者结构的不同。目前对牛乳β-酪蛋白结构的研究较多,但对羊乳β-酪蛋白的结构以及羊乳和牛乳β-酪蛋白结构差异的研究还鲜有报道。蛋白质二级结构的信息可由光谱获得,其中圆二色光谱是利用蛋白质分子中具有光学活性的生色基团对左、右平面圆偏振光吸收不同,对蛋白质结构进行表征的方法,可以测定溶液状态下的蛋白质样品,使蛋白质构象更接近其生理状态,而且具有快速简便,对构象变化灵敏等优点;红外光谱则是利用蛋白质分子在振动过程中不同化学键或官能团对红外光吸收频率不同,对蛋白质结构进行表征的方法,可以测定固体状态下的蛋白质样品,具有扫描速度快、分辨率高、可测波长范围广、不易受蛋白质样品的分子大小和外界条件影响等优点。圆二色光谱和红外光谱已被广泛应用于蛋白质构象的研究中,但是结合使用这两种方法分析β-酪蛋白结构的研究还鲜有报道。因此,该研究采用圆二色光谱和红外光谱比较羊乳和牛乳β-酪蛋白的结构特点,并利用分光光度法对二者的巯基含量及溶解性进行了分析,从功能性质方面的不同对两种蛋白结构的差异进行更好的说明。圆二色光谱测得羊乳和牛乳β-酪蛋白二级结构中主要以无规卷曲为主,但羊乳β-酪蛋白的无规卷曲含量(50.2%±0.16%)显著高于牛乳β-酪蛋白(43.8%±0.14%),其有序结构中α-螺旋含量(2.7%±0.21%)、β-折叠含量(15.3%±0.08%)显著低于牛乳β-酪蛋白(4.3%±0.13%, 19.5%±0.12%),β-转角含量分别为31.8%±0.11%和32.4%±0.09%,差异不显著;红外光谱测得羊乳β-酪蛋白二级结构中α-螺旋、β-折叠、β-转角含量分别比牛乳β-酪蛋白低18%~20%, 9%~10%, 0.6%~1%,无规卷曲含量比牛乳β-酪蛋白高17%~19%。对两种蛋白功能性质的研究表明,羊乳β-酪蛋白与牛乳β-酪蛋白表面巯基含量基本一致19~20μmol·g^-1,但羊乳β-酪蛋白总巯基含量[(28.35±0.13)μmol·g^-1]显著低于牛乳β-酪蛋白[(46.72±0.21)μmol·g^-1];羊乳β-酪蛋白与牛乳β-酪蛋白的等电点较为接近(pH为4~5),且在等电点附近前者的溶解性低于后者,而远离等电点时前者溶解性则高于后者。研究结果说明与牛乳β-酪蛋白相比,羊乳β-酪蛋白分子的无序性和柔韧性更高,胶束内部结构更加的柔软疏松。 | 李萌 王娟 魏子凯 康佳欣 张玲 Tabys Dina 刘宁 张爽 | 2020 | 光谱学与光谱分析2020，40，3： | 12 |
| 11 | Pathophysiology of chronic pancreatitis显示文摘Chronic pancreatitis(CP)is an inflammatory disease of the pancreas characterized by progressive fibrotic destruction of the pancreatic secretory parenchyma.Despite the heterogeneity in pathogenesis and involved risk factors,processes such as necrosis/apoptosis,inflammation or duct obstruction are involved.This fibrosing process ultimately leads to progressive loss of the lobular morphology and structure of the pancreas,deformation of the large ducts and severe changes in the arrangement and composition of the islets.These conditions lead to irreversible morphological and structural changes resulting in impairment of both exocrine and endocrine functions.The prevalence of the disease is largely dependent on culture and geography.The etiological risk-factors associated with CP are multiple and involve both genetic and environmental factors.Throughout this review the M-ANNHEIM classification system will be used,comprising a detailed description of risk factors such as:alcohol-consumption,nicotineconsumption,nutritional factors,hereditary factors,efferent duct factors,immunological factors and miscellaneous and rare metabolic factors.Increased knowledge of the different etiological factors may encourage the use of further advanced diagnostic tools,which potentially will help clinicians to diagnose CP at an earlier stage.However,in view of the multi factorial disease and the complex clinical picture,it is not surprising that treatment of patients with CP is challenging and often unsuccessful. | Christina Brock Lecia Mφller Nielsen Dina Lelic Asbjφrn Mohr Drewes | 2013 | World Journal of Gastroenterology2013，19，42： | 11 |
| 12 | E2F transcription factors and digestive system malignancies: How much do we know?显示文摘E2F family of transcription factors regulates various cellular functions related to cell cycle and apoptosis. Its individual members have traditionally been classified into activators and repressors, based on in vitro studies. However their contribution in human cancer is more complicated and difficult to predict. We review current knowledge on the expression of E2Fs in digestive system malignancies and its clinical implications for patient prognosis and treatment. E2F1, the most extensively studied member and the only one with prognostic value, exhibits a tumor-suppressing activity in esophageal, gastric and colorectal adenocarcinoma, and in hepatocellular carcinoma (HCC), whereas in pancreatic ductal adenocarcinoma and esophageal squamous cell carcinoma may function as a tumorpromoter. In the latter malignancies, E2F1 immunohistochemical expression has been correlated with higher tumor grade and worse patient survival, whereas in esophageal, gastric and colorectal adenocarcinomas is a marker of increased patient survival. E2F2 has only been studied in colorectal cancer, where its role is not considered significant. E2F4's role in colorectal, gastric and hepatic carcinogenesis is tumor-promoting. E2F8 is strongly upregulated in human HCC, thus possibly contributing to hepatocarcinogenesis. Adenoviral transfer of E2F as gene therapy to sensitize pancreatic cancer cells for chemotherapeutic agents has been used in experimental studies. Other therapeutic strategies are yet to be developed, but it appears that targeted approaches using E2F-agonists or antagonists should take into account the tissue-dependent function of each E2F member. Further understanding of E2Fs' contribution in cellular functions in vivo would help clarify their role in carcinogenesis. | Athanasios Xanthoulis Dina G Tiniakos | 2013 | World Journal of Gastroenterology2013，19，21： | 10 |
| 13 | Vascular calcification:When should we interfere in chronic kidney disease patients and how?显示文摘Chronic kidney disease(CKD) patients are endangered with the highest mortality rate compared to other chronic diseases. Cardiovascular events account for up to 60% of the fatalities. Cardiovascular calcifications affect most of the CKD patients. Most of this calcification is related to disturbed renal phosphate handling. Fibroblast growth factor 23 and klotho deficiency were incriminated in the pathogenesis of vascular calcification through different mechanisms including their effects on endothelium and arterial wall smooth muscle cells. In addition, deficient klotho gene expression, a constant feature of CKD, promotes vascular pathology and shares in progression of the CKD. The role of gut in the etio-pathogenesis of systemic inflammation and vascular calcification is a newly discovered mechanism. This review will cover the medical history, prevalence, pathogenesis, clinical relevance, different tools used to diagnose, the ideal timing to prevent or to withhold the progression of vascular calcification and the different medications and medical procedures that can help to prolong the survival of CKD patients. | Usama Abdel Azim Sharaf El Din Mona Mansour Salem Dina Ossama Abdulazim | 2016 | World Journal of Nephrology2016，5，5： | 10 |
| 14 | Using Online Conversations to Study Word-of-Mouth Communication显示文摘 | David Godes Dina Mayzlin | 2004 | Marketing Science2004，，4： | 10 |
| 15 | New animal models for hepatitis C viral infection and pathogenesis studies显示文摘Hepatitis C virus (HCV) is a major cause of chronic live disease, cirrhosis and hepatocellular carcinoma (HCC) In man, the pathobiological changes associated wit HCV infection have been attributed to both the immun system and direct viral cytopathic effects. Until now, th lack of simple culture systems to infect and propagat the virus has hampered progress in understandin the viral life cycle and pathogenesis of HCV infection including the molecular mechanisms implicated in HCV induced HCC. This clearly demonstrates the need t develop small animal models for the study of HCV associated pathogenesis. This review describes an discusses the development of new HCV animal models t study viral infection and investigate the direct effects o viral protein expression on liver disease. | Dina Kremsdorf Nicolas Brezillon | 2007 | World Journal of Gastroenterology2007，13，17： | 9 |
| 16 | 超声波与电火花表面复合强化Ti17合金的疲劳性能(英文)显示文摘将Ti17合金经过电火花处理(EST)后再进行超声波表面处理以期提高疲劳寿命。通过扫描电子显微镜观察表面形貌及强化层结构,经X射线衍射分析表面残余应力,采用HT-100高温疲劳试验机测试350℃拉-拉疲劳寿命。结果表明,经过超声波表面处理后,试样表面粗糙度及表面残余应力降低,复合强化后试样的疲劳寿命是仅经过EST处理的两倍。经过硬质合金YG8电极EST以及超声波复合处理的试样疲劳寿命最长。 | 焦磊 乔生儒 张程煜 马侃 韩栋 Dina Gertsriken Vladimir Mazanko | 2010 | 稀有金属材料与工程2010，39，12： | 9 |
| 17 | 砖石古塔地震损伤演化的数值模拟显示文摘采用显式积分法探讨了砖石古塔的地震破坏机制.应用动力弹塑性分析法表征并求解砖砌体结构破坏的非线性问题,得到古塔在地震作用下产生变形、开裂、压溃等结果的动态过程和破坏形态.结果表明,显式积分法适用于模拟砖石古塔的震损演化过程. | 李胜才 赵有军 D’Ayala Dina 刘羽 | 2014 | 扬州大学学报(自然科学版)2014，17，4： | 9 |
| 18 | 加强公共卫生服务提供以推进中国全民健康覆盖显示文摘中国“基本公共卫生服务均等化”政策通过加强公共卫生体系来推进全民健康覆盖。均等化政策提高了服务的人群覆盖率,不同经济发展水平地区服务覆盖率的差距有所缩小。基本公共卫生服务的质量,特别是慢性病管理服务,是目前政策仍未完全解决的问题。均等化政策的完善需要考虑筹资水平和服务包设计更科学地匹配;合理设计提供者激励制度以促进服务质量的提高;推动基本公共卫生服务包提供和基层诊疗服务更好地整合;推进健康服务提供体系更好地整合,以及健康服务筹资制度的进一步融合。 | 袁蓓蓓(译) Dina Balabanova 高军 汤胜蓝 郭岩 | 2019 | 英国医学杂志中文版2019，22，12： | 8 |
| 19 | Liver histology in ICU patients dying from sepsis:A clinico-pathological study显示文摘AIM:To determine end-stage pathologic changes in the liver of septic patients dying in the intensive care unit. METHODS: Needle liver biopsies obtained immediately after death from 15 consecutive patients with sepsis and no underlying liver disease were subjected to routine histological examination. Liver function tests and clinical monitoring measurements were also recorded. RESULTS: Liver biochemistries were increased in the majority of patients before death. Histology of liver bi- opsy specimens showed portal inflammation in 73.3%, centrilobular necrosis in 80%, lobular inflammation in 66.7%, hepatocellular apoptosis in 66.6% and cholan- gitis/cholangiolitis in 20% of patients. Mixed hepatitic/ cholestatic type of liver injury was observed in 6/15 (40%) patients and hepatitc in 9/15 (60%). Steatosis was ob- served in 11/15 (73.3%) patients affecting 5%-80% of liver parenchyma. Among the histological features, the presence of portal inflammation in liver biopsy was as- sociated with increased hospitalization in the ICU prior death (P = 0.026). CONCLUSION: Features of hepatitis and steatosis arethe main histological findings in the liver in the majority of patients dying from sepsis. | John Koskinas Ilias P Gomatos Dina G Tiniakos Nikolaos Memos Maria Boutsikou Aspasia Garatzioti Athanasios Archimandritis Alexander Betrosian | 2008 | World Journal of Gastroenterology2008，14，9： | 8 |
| 20 | Pathological,immunohistochemical and microbiologicalal analysis of lacrimal sac biopsies in patients with chronic dacrocystitis显示文摘AIM:To analyze cases of obstruction of the nasolacrimal duct which creates a fertile environment for secondary bacterial Infection and can result in dacryocystitis,which is a constant threat to cornea and orbital soft tissue and a potential source of endophthalmitis following intraocular surgery.The majority of obstructions of the lacrimal excretory outflow system are acquired ones occurring in adulthood and involving the distal parts of the system.Acquired obstruction may be primary/idiopathic or secondary to a wide variety of infectious,inflammatory,traumatic,mechanical,toxic or neoplastic causes mimicking idiopathic inflammation.These cases are treated by dacryocystorhinostomy(DCR).METHODS:The present study was conducted to determine the histopathologic,immunohistochemical and current microbiologic characteristics of lacrimal sac specimens in patients undergoing external dacryocystorhinostomy.RESULTS:Non-specific lacrimal sac pathology was present in all 33 cases and 81.8%of the cases showed moderate chronic inflammation with a chronic inflammatory score(CIS)ranging between 4 and 6,whereas 12.12%showed severe inflammatory changes with a CIS of 7.Mild degree of inflammation was seen in6.06%with a CIS of 3.The total prevalence of grampositive,gram-negative,and culture-negative samples were 59.4%,37.5%,and 3%respectively.CONCLUSION:Non-specific chronic inflammation withfibrosis is indeed the most commonly reported histopathological finding in lacrimal sac wall biopsy specimens. | Rowayda Mahmoud Amin Faten Aly Hussein Hisham Farouk Idriss Nesrine Fathy Hanafy Dina Mohamed Abdallah | 2013 | International Journal of Ophthalmology(English edition)2013，6，6： | 7 |
您所在的机构暂未开通此部分的功能，请联系管理员