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| 1 | Diabetic retinopathy: a matter of retinal ganglion cell homeostasis显示文摘Diabetic retinopathy(DR)is the most common complication of diabetes and the leading cause of visual impairment in the working-age population in the Western world(http://gffzzc722d8cd65ac40e7hnu6pn50co9fv65xx.ffgz.tsg.suse.edu.cn/).One of the earliest events observed in DR is the impairment of retinal perfusion,caused by the constriction of retinal blood vessels,inducing biochemical and metabolic alterations,perycites/endothelial degeneration,and retina ischemia(Wong et al.,2016).Severe stages of DR include proliferative DR,caused by the abnormal growth of new vessels,and diabetic macular oedema in the central part of the retina.DR is strongly associated with a prolonged duration of diabetes,hypertension,and hyperglycaemia but it can persist even after replacement of normal glycaemic status(Wong et al.,2016). | Elisabetta Catalani Davide Cervia | 2020 | Neural Regeneration Research2020,15,7: | 9 |
| 2 | 'Cervia II Working Group Report 2006' : guidelines on diagnosis and treatment of Helicobacter pylori infection in Italy显示文摘 | Working Group of the Cervia I1 Meeting | 2007 | Dig Liver Dis2007,39,8: | 1 |
| 3 | Nitric oxide and sphingolipids control apoptosis and autophagy with a significant impact on Alzheimer's disease 显示文摘 | Cervia D Permtta C Moscheni C | 2013 | J Bid Regul Homeost Agents2013,27,2: | 1 |
| 4 | Associations of proin- flammatory cytokine levels with lipid profiles, growth, and body composition in HIV-infected children initiating or changing an- tiretroviral therapy显示文摘 | Cervia JS Chantry CJ Hughes MD | 2010 | Pediatr Infect Dis J2010,29,12: | 1 |
| 5 | Soma- tostatin receptors differentially affect spontaneous epileptiform activi- ty in mouse hippocampal slices显示文摘 | CAMMALLERI M CERVIA D LANGENEGGER D | 2004 | Eur J Neurosci2004,20,10: | 1 |
| 6 | Compensatory changes in the hippocampus of somatostatin knockout mice : upregulation of somatostatin receptor 2 and its function in the control of bursting activity and synaptic transmission显示文摘 | Cammalleri M Cervia D Dal Monte M | 2006 | Eur J Neurosci2006,23,24: | 1 |
| 7 | Interleukin 18 in the CNS 显示文摘 | Alboni S Cervia D Sugama S | 2010 | J Neuroinflammation2010,7,: | 1 |
| 8 | Five-Aminosalicylic Acid: An Update for the Reappraisal of an Old Drug显示文摘 | Cristiana Perrotta Paolo Pellegrino Eliana Moroni Clara De Palma Davide Cervia Piergiorgio Danelli Emilio Clementi Bj?rn Moum | 2015 | Gastroenterology Research and Practice2015,,: | 1 |
| 9 | Interleukin 18 in the CNS显示文摘 | Alboni S Cervia D Sugama S | 2010 | J Neuroinflammation2010,7,9: | 1 |
| 10 | Diagnosis and management of infectious esophagitis associated with human immunodeflciency virus infection显示文摘 | Zaidi SA Cervia JS | 2002 | J Int Assoc Physicians AIDS Care(Chic)2002,1,2: | 1 |
| 11 | Interleukin 18 in the CNS显示文摘 | Silvia 'Albonil Davide Cervia Shuei Sugama | 2010 | J Neuroinflammation2010,7,: | 1 |
| 12 | Molecular mechanisms of euplotin C-induced apoptosis:involvement of mitochondrial dysfunction, oxidative stress and protea- ses 显示文摘 | Cervia D Garcia-Gil M Simonetti E | 2007 | Apoptosis2007,12,8: | 1 |
| 13 | Leukocyte reduction' s role in the attenuation of infection risks among transfusion recipients显示文摘 | Cervia J S Wenz B Ortolano G A | 2007 | Clin Infect Dis2007,45,8: | 1 |
| 14 | Mapping of the full length and the truncated interleukin-18 receptor alpha in the mouse brain显示文摘 | Alboni S Cervia D Ross B | 2009 | J Neuroimmunol2009,214,12: | 1 |
| 15 | Inhibitory control of growth hormone secretion by somatostatin in rat pituitary GC cells: sst(2) but not sst(1) receptors are coupled to inhibition of single-cell intracellular free calcium concentrations显示文摘 | Cervia D Petrucci C Bluet-Pajot MT | 2002 | Neuroendocrinology2002,76,2: | 1 |
| 16 | Pharmacological characterisation of native somatostatin receptors in AtT- 20 mouse tumour corticotrophs显示文摘 | Cervia D Nunn C Fehlmann D | 2003 | Br J Pharmacol2003,139,1: | 1 |
| 17 | Comparison of functional profiles at human recombinant somatostatin sst2 receptor: simultaneous determination of intraeellular Ca2+ and luciferase expression in CHO-K1 cells 显示文摘 | Nunn C Cervia D Langenegger D | 2004 | Br J Pharmacol2004,142,1: | 1 |
| 18 | Successful combination Voncomycin and Rifapin therapy in a newborn with community-acquired Flavobacterium meningoseptium neonatal meningitis显示文摘 | TIZER K B CERVIA J S DUMN A M | 1995 | Pediatr Dis1995,14,: | 1 |
| 19 | Interleukin 18 in the CNS显示文摘 | ALBONI S CERVIA D SUGAMA S | 2010 | J Neurolnflammatlon2010,7,: | 1 |
| 20 | Recent advances in cellular and molecular aspects of mammalian retinal ischemia显示文摘Retinal ischemia is a common clinical entity and, due to relatively ineffective treatment, remains a common cause of visual impairment and blindness. Generally, ischemic syndromes are initially characterized by low homeostatic responses which, with time, induce injury to the tissue due to cell loss by apoptosis. In this respect, retinal ischemia is a primary cause of neuronal death. It can be considered as a sort of final common pathway in retinal diseases and results in irreversible morphological and functional changes. This review summarizes the recent knowledge on the effects of ischemia in retinal tissue and points out experimental strategies/models performed to gain better comprehension of retinal ischemia diseases. In particular, the nature of the mechanisms leading to neuronal damage(i.e., excess of glutamate release, oxidative stress and inflammation) will be outlined as well as the potential and most intriguing retinoprotective approaches and the possible therapeutic use of naturally occurring molecules such as neuropeptides. There is a general agreement that a better understanding of the fundamental pathophysiology of retinal ischemia will lead to better management and improved clinical outcome. In this respect, to contrast this pathological state, specific pharmacological strategies need to be developed aimed at the many putative cascades generated during ischemia. | Davide Cervia Giovanni Casini | 2012 | World Journal of Pharmacology2012,1,2: | 1 |