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17篇 您的检索式:作者名="Adnot P"
    题名 作者 年代 出处 被引量
1内脏脂肪组织通过产生骨桥蛋白调控成纤维细胞衰老促进心脏衰老显示文摘老化通过增加包括骨桥蛋白(osteopontin,OPN)在内的细胞外基质(extracellular matrix,ECM)蛋白的沉积引起心脏结构和功能改变进而导致进行性间质纤维化。虽然已知OPN参与各种病理状况,但其在心肌衰老中的作用仍不清楚。Sawaki D Czibik G Pini M Ternacle J Suffee N Mercedes R Marcelin G Surenaud M Marcos E Gual P Clément K Hue S Adnot S Hatem SN Tsuchimochi I Yoshimitsu T Hénégar C Derumeaux G 刘青 叶鹏 2018中华高血压杂志2018,26,3:14
2Structurally stable composite Pd-Ag membranes : introduction of a diffusion barrier 显示文摘Shu J Adnot A Grandjean B P A 1996Thin Solid Films1996,286,:1
3ESCA spectroscopy of poly ( methyl methacrylate) grafted onto wood fibers 显示文摘KAMDEM D P RIEDL B ADNOT A 1991Journal of Applied Polymer Science1991,43,10:1
4Effects of HIV protease inhibitors on progression of monocrotaline- and hypoxiainduced pulmonary hypertension in rats 显示文摘Gary-Bobo G Houssaini A Amsellem V Rideau D Pacaud P Perrin A Bregeon J Marcos E Dubois-Rande JL Sitbon 0 Savale L Adnot S 2010Circulation2010,122,19:1
55-Hydroxytryptamine and the pulmonary circulation:receptors,transporters and relevance to pulmonary arterial hypertension显示文摘MacLean MR Herve P Eddahibi S Adnot S 2000Br J Pharmacol2000,131,2:1
6Serotonin-induced smooth muscle hyperplasia in various forms of human pulmonary hypertension显示文摘Marcos E Fadel E Sanchez O Humbert M Dartevelle P Simooneau G Hamon M Adnot S Eddahibi S 0,,:1
7RhoA and Rho kinase activation in human pulmonary hypertension:role of 5-HT signaling显示文摘Guilluy C Eddahibi S Agard C Guignabert C Izikki M Tu L Savale L Humbert M Fadel E Adnot S Loirand G Pacaud P 0,,12:1
8Serotonin transporter inhibition prevents and reverses monocrotaline-induced pulmonary hypertension in rats显示文摘Guignabert C Raffestin B Benferhat R Raoul W Zadigue P Rideau D Hamon M Adnot S Eddahibi S 0,,21:1
9Transgenic mice overexpressing the 5-hydroxytryptamine transporter gene in smooth muscle develop pulmonary hypertension显示文摘Guignabert C Izikki M Tu L I Li Z Zadigue P Barlier-Mur A M Hanoun N Rodman D Hamon M Adnot S Eddahibi S 0,,10:1
10X-ray photoelectron spectroscopy study of sodium reactions incarbon cathode blocks of aluminum oxide raduction cells显示文摘Brisson P Y Darmstad H Fafard M Adnot A Servant G Soucy G 0,,08:1
11Structurally stablecomposite Pd-Ag alloy membranes: Introduction of a diffusionbarrier 显示文摘SHU J ADNOT A GRAND JEAN B P A 1996Thin Solid Films1996,286,12:1
12Structurally stable composite Pd-Ag alloy membranes: introduction of a diffusion barrier 显示文摘Shu J Adnot A Grandjean B P A Kaliaguine S 1996Thin Solid Films1996,286,12:1
135-hydroxytryptamine and the pulmonary circulation: receptors, transporters and relevance to pulmonary arterial hypertension显示文摘MacLean MR Herve P Eddahibi S Adnot S 2000Br J Pharmacol2000,131,2:1
14Resistance to the lipolytic action of epinephrine:a new feature of protein GS deficiency显示文摘Carel JC Le Stunff C Condamine L Mallet E Chaussain JL Adnot P 1999J Clin Endocrinol Metab1999,84,:1
15Atrial natriuretic factor attenuates the pulmonary pressor response to hypoxia显示文摘 Chabrier P E Bran C 1988J Appl Physiol1988,65,5:1
16镰状细胞病患者肺高压的血流动力学研究显示文摘肺动脉高压以存在毛细血管前肺高压,无左侧心脏疾病、肺部疾病、慢性血栓栓塞疾病为特征。在组织病理学方面,肺动脉高压则以肺小动脉内层平滑肌细胞和内皮细胞增殖为特征。肺动脉高压可以是原发性、遗传性或与其他疾病相关,如结缔组织疾病和先天性心脏病等。以往的研究发现,肺高压特别是肺动脉高压是镰状细胞病较常见的并发症[1]。Parent F Bachir D Inamo J Lionnet F Driss F Loko G Habibi A Bennani S Savale L Adnot S Yaci A Hajji L O'Callaghan DS Clerson P Girot R Galacteros F Maitre B Simonneau G 叶鹏 2011中华高血压杂志2011,19,8:0
17肺动脉高压中骨桥蛋白是衰老肺血管细胞表达的重要介质显示文摘衰老的肺动脉平滑肌细胞(pulmonaryarterysmoothmusclecells,PA-SMC)可通过产生分泌因子促进肺高血压的发病。该研究的目的是探讨由衰老的PA-SMC释放的细胞外基质蛋白在肺高血压中的作用。方法:对正进行复制的衰老的人类PA-SMC进行聚合酶链反应阵列分析,发现骨桥蛋白上调,其介导了衰老PA-SMC介质和基质的刺激作用,以及对PA-SMC生长和迁移的作用。刘莉 叶鹏 Saker M Lipskaia L Marcos E Abid S Parpaleix A Houssaini A Validire P Girard P Noureddine H Boyer L Vienney N Amsellem V Marguerit L Maitre B Derumeaux G Dubois-Rande JL Jourdan-Lesaux C Delcroix M Quarck R Adnot S 2016中华高血压杂志2016,24,9:0
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