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| 1 | Mechanisms involved in the inhibition of myoblast proliferation and differentiation by myostatin显示文摘 | Dominique Joulia Henri Bernardi Véronique Garandel Fanjaniriana Rabenoelina Barbara Vernus Gérard Cabello | 2003 | Experimental Cell Research2003，，2： | 3 |
| 2 | Myostatin and the skeletal muscle atrophy and hypertrophy signaling pathways显示文摘 | Rodriguez J Vernus B Chelh I | 2014 | Cell Mol Life Sci2014，71，22： | 1 |
| 3 | Myostatin up-regulation isassociated with the skeletal muscle response to hypoxic stimuli显示文摘 | Hayot M Rodriguez J Vernus B | 2011 | Mol Cell Endocrinol2011，332，12： | 1 |
| 4 | Myostatin up - regulation is associated with the skeletal muscle response to hypoxic stimuli 显示文摘 | Hayot M Rodriguez J Vernus B | 2011 | Mol Cell Endocrinol2011，332，12： | 1 |
| 5 | Myostatin up-regulation is associated with the skeletal muscle response to hypoxic stimuli显示文摘 | Maurice Hayot Julie Rodriguez Barbara Vernus Gilles Carnac Elise Jean David Allen Lucie Goret Philippe Obert Robin Candau Anne Bonnieu | 2010 | Molecular and Cellular Endocrinology2010，，1： | 1 |
| 6 | Myostatin up-regulation isassociated with the skeletal muscle response to hypoxic stimuli显示文摘 | Hayot M Rodriguez J Vernus B | 2011 | Mol Cell Endocrinol2011，332，12： | 1 |
| 7 | Myostatin up- regulation is associated with the skeletal muscle response to hypoxic stimuli显示文摘 | Hayot M Rodriguez J Vernus B | 2011 | Mol Cell Endocrinol2011，332，12： | 1 |
| 8 | Long term omeprazole therapy for reflux esophagitis:follow-up in serum gastrin levels,EC cell hyperplasia and neoplasia显示文摘AIM To evaluate the long-term safety ofomeprazole in patients of gastroesophagealreflux disease resistant to treatment with H2receptor antagonist.METHODS We prospectively followed 33patients on omeprazole therapy for severeerosive esophagitis for 5-8 years,with periodicgastrin levels,H.pylori infection,gastricbiopsies for incidence of ECL cell hyperplasia,carcinoids,gastric atrophy and neoplasia.Atotal 185 patient follow-up years and 137 gastricbiopsies were done.RESULTS Among the 33 patients,36% reachedtheir peak gastrin levels in an average of 8months to one year,then drifted Down slowlyover 1-2 year period to just above their baselinelevel,24% of the patients had a peak gastrinlevel above 400ng·L^1 and one patient had apeak level above 1000 ng·L^1.One patient had amild ECL cell hyperplasia which was self limitingand did not show any dysplastic changes.Eighteen percent of patients were positive forH.pylori infection.The gastric biopsies did notshow gastric atrophy,intestinal metaplasia orneoplastic changes.CONCLUSION In a series of 33 patients followed for 5 - 8 years on omeprazole therapy for severe reflux esophagitis, we did not observe any evidence of significant ECL cell hyperplasia, gastric atrophy, intestinal metaplasia, dysplasia or neoplastic changes. | Pankaj Singh Anant Indaram Ronald Greenberg Vernu Visvalingam Simmy Bank | 2000 | World Journal of Gastroenterology2000，6，6： | 1 |
| 9 | Myostatin and the skeletal muscle atrophy and hypertrophy signaling pathways显示文摘 | Rodriguez J Vernus B Chelh I | 2014 | Cell Mol Life Sci2014，71，： | 1 |
| 10 | Myostatin and the skeletal muscle atrophy and hypertrophy, signaling pathways 显示文摘 | Rodriguez J Vernus B Chelh I | 2014 | Cell Mol Life Sci2014，71，： | 1 |
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