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17篇 您的检索式:作者名="Sykes DB"
    题名 作者 年代 出处 被引量
1Rapid,revers-ible modulation of blood-brain barrier P-glycoproteintransport activity by vascular endothelial growthfactor显示文摘Hawkins BT Sykes DB Miller DS 2010J Neurosci2010,30,4:1
2Hoxa9 immortalizes a granulocyte-macrophage colony-stimulating factor-dependent promyelocyte capable of biphenotypic differentiation to neutrophils or macrophages,independent of enforced meis expression显示文摘Calvo KR Sykes DB Pasillas M 2000Mol Cell Biol2000,20,9:1
3Meis 1a suppresses differentiation by G-CSF and promotes proliferation by SCF:Potential mechanisms of cooperativity with Hoxa9 in myeloid leukemia显示文摘Calvo KR Knoepfler PS Sykes DB 2001PNAS2001,98,13:1
4Rapid, reversible modulation of blood - brain barrier P - plycoprotein transport activity by vascular endothelial growth gactor显示文摘Hawkins BT Sykes DB Miller DS 2010J Neurosci2010,30,:1
5Rapid, reversible modula- tion of blood- brain barrier P-glyeoprotein transport activity by vas- cular endothelial growth factor显示文摘Hawkins BT Sykes DB Miller DS 2010J Neurosci2010,30,4:1
6HoxB8 requires its Pbxinteraction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation显示文摘Knoepfler PS Sykes DB Pasillas M 2001Oncogene2001,20,39:1
7Nup98-HoxA9 immortalizes myeloid progenitors,enforces expression of Hoxa9,Hoxa7 and Meis 1,and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1显示文摘 Sykes DB Pasillas M P 2002Oncogene2002,21,27:1
8An induced Ets re- pressor complex regulates growth arrest during terminal macrophage differentiation 显示文摘Klappacher GW Lunyak VV Sykes DB 2002Cell2002,109,2:1
9An induced Ets repressor complex regulates growth arrest during terminal macrophage differentiation显示文摘Klappacher GW Lunyak VV Sykes DB 2002Cell2002,109,2:1
10The TEMPI syndrome--a novel multisystem disease 显示文摘Sykes DB Schroyens W O'Connell C 2011N Engl J Med2011,365,5:1
11Meis1a suppresses differentiation by G-CSF and promotes proliferation by SCF:potential mechanisms of cooperativity with Hoxa9 in myeloid leukaemia显示文摘Calvo KR Knoepfler PS Sykes DB 0,,:1
12Complete and partial responses of the TEMPI syndrome to bortezomib 显示文摘Schroyens W O' Connell C Sykes DB 2012N Engl J Med2012,367,8:1
13Rapid,reversible modulation of blood-brain barrier P-glycoprotein transport activity by vascular endothelial growth factor显示文摘Hawkins BT Sykes DB Miller DS 2010J Neurosci2010,30,4:1
14HoxB8 requires its Pbx-interaction motif block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation 显示文摘Knoepfler PS Sykes DB Pasillas M 2001Oncogene2001,20,:1
15Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins 显示文摘Sykes DB Kamps MP 2001Blood2001,98,8:1
16NUP98-HoxA9 immortalizes myeloid progenitors,enforces expression of Hoxa9,Hoxa7 and Meisl,and alters cytokinespecific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meisl显示文摘Calvo KR Sykes DB Pasillas MP 0,,:1
17An induced Ets re- pressor complex regulates growth arrest during terminal macrophage differentiation显示文摘KLAPPACHER GW VV LUNYAK DB SYKES 2002Cell2002,109,:1
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