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62篇 您的检索式:作者名="SERVIDDIO G"
    题名 作者 年代 出处 被引量
1Glutamatergic alterations and mitochondrial impairment in a murine model of Alzheimer disease显示文摘Cassano T Serviddio G Gaetani S 2012Neurobiol Aging2012,33,6:1
2Glutamatergic alterations and mitochondrial impairment in a murine model of Alzheimer disease 显示文摘Cassano T Serviddio G Gaetani S 2012Neurobiol Aging2012,33,:1
3Postconditioning is an effective strategy to reduce renal ischemia /reperfusion injury 显示文摘Serviddio G Romano AD Gesualdo L 2008Nephrol Dial Transplant2008,23,5:1
4Brief hypoxia before normoxic reperfusion (postconditioning) protects the heart against ischemia-reperfusion injury by preventing mitochondria peroxyde production and glutathione depletion 显示文摘SERVIDDIO G DI VENOSA N FEDERICI A 2005Faseb J2005,19,3:1
5Targeting mitochondria: a new promising approach for the treatment of liver diseases 显示文摘Serviddio G Bellanti F Sastre J 2010Current Medicinal Chemistry2010,17,22:1
6Mitochondrial involvement in non-alcoholic steatohepatitis 显示文摘SERVIDDIO G SASTRE J BELLANTI F 2008Mol Aspects Med2008,29,12:1
7Brief hypoxia before normoxic reperfusion(postconditioning) protects the heart against ischemia reperfusion injury by preventing mitochondria peroxyde production and glutathione depletion 显示文摘SERVIDDIO G VENOSA D N FEDERICI A 2005FASEB J2005,19,:1
8Mitochondrial involvement in non-alcoholic steatohepatitis 显示文摘Serviddio G Sastre J Bellanti F 2008Mol Aspects Med2008,29,12:1
9Brief hypoxia before normoxic reperfusion (posteonditioning) protects the heart against isehemia-reperfusion injury by preventing mitoehondria peroxyde pro- duction and glutathione depletion 显示文摘Serviddio G Di Venosa N Federici A 2005FASEB J2005,19,3:1
10Postcondi- tioning is an effective strategy to reduce renal ischaemia/ reperfusion injury 显示文摘Serviddio G Romano A D Gesualdo L 2008Nephrol Dial Transplant2008,23,5:1
11Mitochondrialinvolvement in no- alcoholic steatohepatitis 显示文摘SERVIDDIO G SASTRE J BELLANTI F etal 2008Mol As-pects Med2008,29,3:1
12Mitochondrial function in liver diseaes显示文摘Sastre J Serviddio G Pereda J 2007Front Biosci2007,12,:1
13Brief hypoxia before normoxic reperfusion (postconditioning) protects the heart against ischemia-reperfusion injury by preventing mitochondria peroxyde production and glutathione depletion 显示文摘Serviddio G Di Venosa N Federici A 2005Faseb J2005,19,3:1
14Mitochondrial involvement in non-alcoholic steatohepatitis显示文摘SERVIDDIO G SASTRE J BELLANTI F 0,,1:1
15Uncoupling protein-2 (UCP2) induces mitochondrial proton leak and increases susceptibility of non-alcoholic steatohepatitis (NASH) liver to ischaemia-reperfusion injury显示文摘Serviddio G Bellanti F Tamborra R 2008Gut2008,57,7:1
16Brief hypoxia before normoxic reperfusion (postconditioning)protects the heart against ischemia-reperfusion injury by preventing mitochondria peroxide production and glutathione depletion显示文摘Serviddio G Di Venosa N Federici A 2005FASEB J2005,19,3:1
17Brief hypoxia before normoxic reperfusion (postconditioning) protects the heart against ischemia - reperfusion injury by preven- ting mitochondria peroxyde production and glutathione depletion 显示文摘Serviddio G Di Venosa N Federici A 2005Faseb J2005,19,3:1
18Posteonditioning is an effective strategy to reduce renal isehaemia reperfusion injury显示文摘Serviddio G Romano AD Gesualdo L 2008Nephrol Dial Transplant2008,23,5:1
19Brief hypoxia be- fore normoxlc reperfuslon (postconditloning) protects the heart against ischemia - reperfusion injury by preventing mi- toehondria peroxide production and glutathione depletion显示文摘Serviddio G Venosa ND Federiei A 2005Faseb J2005,19,3:1
20Mitochondrial function in liver disease 显示文摘SASTER J SERVIDDIO G PEREDA J 2007Front Biosci2007,12,12:1
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