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19篇 您的检索式:作者名="Roberds SL"
    题名 作者 年代 出处 被引量
1BACE knockdown mice are healthy despite lacking the primary beta-secretase activity in brain:implications for Alzheimer' s disease therapeutics 显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
2BACE knockout mice are healthy despite lacking the primary beta-secrerase activity in brain: implication for Alzheimer′s disease therapeutics 显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
3Circulating succinate is elevated in rodent models of hypertension and metabolic disease显示文摘Sadagopan N Li W Roberds SL 0,,11:1
4BACE knockout mice are healthy dispite lacking the primary beta-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
5BACE knockout mice are healthy despite lacking the primary β-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 0,,:1
6BACE knockout mice are healthy despite lacking the pri mary beta-secretase activity in brain: implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
7BACE knockout mice are healthy despite lacking the primary β-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
8Disease mechanisms revealed by transcription profiling in SOD1-G93A transgenic mouse spinal cord显示文摘Olsen MK Roberds SL Ellerbrock BR 0,,:1
9Time-,voltage-,and state-dependent block by quinidine of a cloned human cardiac potassium channel显示文摘Snyders J Knoth KM Roberds SL 1992Mol Pharmacol1992,41,2:1
10A role for dystrophin- associated glycoproteins and utrophin in agrin-induced AChR clustering显示文摘Campanelli JT Roberds SL Campbell KP 1994Cell1994,77,:1
11BACE knockout mice are healthy despite lacking the primary β-secretase activity in brain : implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,:1
12BACE knockout mice are healthy despite lacking the primary betasecretase activity in brain: implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
13BACE knockout mice are healthy despite lacking the primary beta-secretase activity in brain: implications for Alzheimer's disease therapeutics 显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
14BACE knockout mice are healthy despite lacking the primary β-secretase activity in brain:implications for Alzheimers disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,:1
15BACE knockdown mice are healthy despite lacking the primary beta-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,:1
16BACE knockdown mice are healthy despite lacking the primary beta-secretase activity in brain : implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,:1
17BACE knockdown mice are healthy despite lacking the primary beta-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson L Basi G 2001Hum Mol Genet2001,10,12:1
18BACE knockout mice are healthy despite lacking the primary β-secretase activity in brain:implications for Alzheimer's disease therapeutics显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,12:1
19BALE knockout mice arehealthy despite lacking the primary beta-secretase activity in brain:implications for Alzheimer's disease therapeutics 显示文摘Roberds SL Anderson J Basi G 2001Hum Mol Genet2001,10,:1
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