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26篇 您的检索式:作者名="Pravdic D"
    题名 作者 年代 出处 被引量
1Differences in production of reactive oxygen species and mitochondrial uncoupling as events in the preconditioning signaling cascade between desflurane and sevoflurane 显示文摘Sedlie F Pravdic D Ljubkovic M 2009Anesth Analg2009,109,2:1
2Biphasic effect of nitric oxide on thecardiac voltage-depmdengt anion channel显示文摘Cheng Q Sedlic F Pravdic D y 2011Febs Lett2011,585,:1
3Aerobic interval training attenuates remodelling and mitochondrial dysfunction in the post-infarction failing rat heart 显示文摘Kraljevic J Marinovic J Pravdic D 2013Cardiovasc Res2013,99,:1
4Aerobic interval training attenuates remodeling and mitochondrial dysfunction in post-infarction failing rat heart显示文摘KRALJEVIC J MARINOVIC J PRAVDIC D 2013Cardiovasc Res2013,99,1:1
5Aerobic interval training attenuates remodellingand mitochondrial dysfunction in the post-infarctionfailing rat heart显示文摘KRALJEVIC J MARINOVIC J PRAVDIC D 2013Cardiovasc Res2013,99,1:1
6Differences in production of reactive oxygen species and mitoehondrial uncoupling asevents in the preconditioning signaling cascade between desflurmle and sevoflurane 显示文摘Sedlie F Pravdic D Ljubkovic M 2009Anesth Analg2009,109,40:1
7Differences in production ofreactive oxygen species and mitochondrial uncoupling as events inthe preconditioning signaling cascade between desflurane and sevoflu-rane 显示文摘Sedlic F Pravdic D Ljubkovic M 2009Anesth Analg2009,109,2:1
8Mitochondrial depolarization underlies delay in permeability transition by preconditioning with isoflurane:roles of ROS and Ca2 +显示文摘Sedlic F Sepac A Pravdic D 0,,02:1
9Isoflurane protects cardiomyocytes and mitochondria by immediate and cytosol-independent action at reperfusion显示文摘Pravdic D Mio Y Sedlic F 0,,02:1
10Isoflurane postconditioning protects against reperfusion injury by preventing mitochondrial permeability transition by an endothelial nitric oxide synthase -dependent meehanism显示文摘Ge Z D Pravdic D Bienengraeber M 2010Anesthesilogy2010,112,1:1
11Isoflurane postconditioning protects against reperfusion injury by preventing mitochondrial permeability transition by an endothelial nitric oxide synthase-dependent mechanism 显示文摘Ge ZD Pravdic D Bienengraeber M et ol 2010Anesthesiology2010,112,1:1
12Anesthetic-induced preconditioning delays opening of mitochondrial permeability transition pore via protein kinase C-epsilon-mediated pathway显示文摘PRAVDIC D SEDLIC F MIO Y 2009Anesthesiology2009,111,2:1
13Aerobic interval training attenuates remodelling and mitochondrial dysfunction in the post- infarction failing rat heart 显示文摘Kraljevic J Marinovic J Pravdic D 2013Cardiovascular research2013,99,1:1
14Aerobic interval training attenuates remodelling and mitochondrial dysfunction in the post-infarction failing rat heart显示文摘Kraljevic J Marinovic J Pravdic D 2013Cardiovasc Res2013,99,1:1
15Anesthetic-induced pre- conditioning delays opening of mitochondrial permeability transi- tion pore via protein Kinase C-epsilon-mediated pathway 显示文摘PRAVDIC D SEDLIC F MIO Y 2009An- esthesiology2009,111,2:1
16Differences in production of re- active oxygen species and mitochondrial uncoupling as events in the pre- conditioning signaling cascade between desflurane and sevoflurane 显示文摘Sedlic F Pravdic D Ljubkovic M 2009Anesth Analg2009,109,2:1
17Isoflurane postcondi- tioning protects against reperfusion injury by preventing mitochondri-al permeabihty transition by an endothelial nitric oxide synthase-de- pendent mechanism显示文摘Ge ZD Pravdic D Bienengraeber M 2010Anesthesiology2010,112,:1
18Anesthetic - induced preconditioning delays opening of mitochondrial permeability transition pore via protein kinase C - ε - mediated pathway 显示文摘Pravdic D Sedlic F Mio Y 2009Anesthesiology2009,111,2:1
19Aerobic interval training attenuates remodelling and mitochondrial dysfunction in the post-infarction failing rat heart显示文摘Kraljevic J Marinovic J Pravdic D 2013Cardiovasc Res2013,99,1:1
20Isoflurane protects cardiomyocytes and mitochondria by immediate and cytosol-independent action at reperfusion显示文摘Pravdic D Mio Y Sedlic F 2010Br J Pharmaco12010,160,2:1
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