维普期刊资源整合服务平台（V6.5）

40篇 您的检索式：作者名="Neviani P"

选中清除导出查看参考文献查看引证文献引用追踪共2页首页上一页第1页下一页末页 /2 跳转

	题名	作者	年代	出处	被引量
1	AMAPK/HNRPK pathway controls BCR/ABL oncogenic potential by regulating MYC mRNA translatlon显示文摘	Notarl M Neviani P Santhanam R	2006	Blood2006，107，6：	1
2	The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein 显示文摘	Neviani P Santhanam R Trotta R	2005	Cancer Cell2005，8，5：	1
3	BCR/ABL, mRNA translation and apoptosis 显示文摘	Perrotti D Turturro F Neviani P	2005	Cell Death Differ2005，12，6：	1
4	Wnt/Ca^2+/NFAT signa- ling maintains survival of Ph + leukemia ceils upon inhibition of Ber - Abl 显示文摘	Gregory MA Phang TL Neviani P	2010	Cancer Cell2010，18，1：	1
5	Protein phosphatase 2A: a target for anticancer therapy显示文摘	Perrotti D Neviani P	2013	Lancet Oncol2013，14，6：	1
6	Bcl-xL anti-apoptot- ic network is dispensable for development and mainte-nance of CML but is required for disease progression where it represents a new therapeutic target 显示文摘	Harb J G Neviani P Chyla B J	2013	Leuke- mia2013，27，10：	1
7	The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein显示文摘	Neviani P Santhanam R Trotta R	2005	Cancer Cell2005，8，5：	1
8	miR-328 functions as an RNA decoy to modulate hnRNP E2 regulation of mRNA translation in leukemic blasts显示文摘	Eiring AM Harb JG Neviani P	2010	Cell2010，140，：	1
9	Wnt/Ca2+/NFAT signaling maintains survival of Ph+leukemia cells upon inhibition of Bcr-Abl显示文摘	Gregory MA Phang TL Neviani P		Cancer Cell0，18，1：	1
10	The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein显示文摘	Neviani P Santhanam R Trotta R		Cancer Cell0，，：	1
11	A MAPK/ HNRPK pathway controls BCR/ABL oncogenie potential by regulating MYC mRNA translation显示文摘	Notari M Neviani P Santhanam R	2006	Blood2006，107，6：	1
12	Wnt/Ca2 +/NFAT signa- ling maintains survival of Ph + leukemia cells upon inhibition of Bcr-Abl显示文摘	Gregory MA Phang TL Neviani P	2010	Cancer Cell2010，18，1：	1
13	miR-328 functions as an RNA decoy to modulate hnRNP E2 regulation of mRNA translation in leukemic blasts显示文摘	Eiring AM Harb JG Neviani P	2010	Cell2010，140，5：	1
14	miR-328 function as an RNA decoy to modulate hnRNP E2 regulation of mRNA translation in leukemic blasts显示文摘	EIRING A M HARB J G NEVIANI P	2010	Cell2010，140，5：	1
15	Identification of novel posttranscriptional targets of the BCR/ABL oncoprotein by ribonomics: requirement of E2F3 for BCR/ABL leukemogenesis 显示文摘	Eiring AM Neviani P Santhanam R	2008	Blood2008，111，2：	1
16	miR-328 functions as an RNA decoy to modulate hnRNP E2 regulation of mRNA transla-tion in leukemic blasts显示文摘	EIRING A M HARB G NEVIANI P		0，，05：	1
17	Resetting PP2A tumour suppressor activity in blast crisis and imatinib-resistant chronic myelogenous leukemia显示文摘	Perrotti D Neviani P	2006	Br J Cancer2006，95，7：	1
18	A MAPK/HNRPK pathway controls BCR/ABL oncogenic potential by regulating MYC mRNA translation 显示文摘	Notari M Neviani P Santhanam R	2006	Blood2006，107，6：	1
19	The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein显示文摘	Neviani P Santhanam R Trotta R	2005	Cancer cell2005，8，5：	1
20	miRs-328function as an RNA decoy to modulate hnRNP E2Regulation of mRNA translation in leukemic blasts显示文摘	EIRING A M HARB J G NEVIANI P	2010	Cell2010，140，：	1

返回顶部每页显示：

共2页首页上一页第1页下一页末页 /2 跳转

互联网出版许可证新出网证(渝)字10号全国400电话 - 免长途话费

用户提示