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| 1 | Does Helicobacter pylori infection eradication modify peptic ulcer prevalence? A 10 years' endoscopical survey显示文摘瞄准:为了在病人比较消化性溃疡流行,在根除治疗的进步散开以后在 pre-Helicobacter 时代和十年在二所意大利的医院里为上面的胃肠的内视镜检查法参考了。方法:我们检查了连续地在 1992 和 2002 期间在 1986-1987 和 1995-1996 期间在 Padova 的肠胃病学单位,并且在 Parma 的肠胃病学单位执行的所有内视镜的考试。迟平方测试被用于统计分析。结果:从两个的数据内视镜的中心在溃疡的流行显示出统计上重要的减少:从 12.7% ~ 6.3%(P<0.001 ) 在 Padova 并且从 15.6% ~ 12%(P<0.001 ) 在 Parma。减少两个都是重要的为十二指肠(从 8.8% ~ 4.8% , P<0.001 ) 并且胃溃疡(3.9% ~ 1.5% , P<0.001 ) 在 Padova,并且仅仅为在 Parma 的十二指肠溃疡(9.2% ~ 6.1% , P<0.001;胃溃疡:6.3% ~ 5.8% , NS ) 。结论:在征兆的病人的根除在消化性溃疡流行导致了重要减小的广泛的 Helicobacter pylori (H pylori ) 的十年。这减小在 Padova 是特别地明显的,在为在一般 practioners 之中的 H pylori 根除的致敏的一个工程在 1990 和 1992 之间被执行的地方。假如我们的假设应该是真的, H pylori 根除可能在未来导致是的消化性溃疡一稀罕内视镜的发现。 | Giorgio Nervi Stefania Liatopoulou Lucas Giovanni Cavallaro Alessandro Gnocchi Nadia Dal Bò Massimo Rugge Veronica Iori Giulia Martina Cavestro Marta Maino Giancarlo Colla Angelo Franzè Francesco Di Mario | 2006 | World Journal of Gastroenterology2006，12，15： | 5 |
| 2 | Dynamic and reversibility of heterochromatic gene silencing in human dis-ease显示文摘In eukaryotic organisms cellular fate and tissue specific gene expression are regulated by the activity of proteins known as transcription factors that by interacting with specific DNA sequences direct the activation or repression of target genes. The post genomic era has shown that transcription factors are not the unique key regulators of gene expression. Epigenetic mechanisms such as DNA methylation, post-translational modifications of histone proteins, remodeling of nucleosomes and expression of small regulatory RNAs also contribute to regulation of gene expression, determination of cell and tissue specificity and assurance of inheritance of gene expression levels. The relevant contri- bution of epigenetic mechanisms to a proper cellular function is highlighted by the effects of their deregulation that cooperate with genetic alterations to the development of various diseases and to the establishment and progression of tumors. | Giuseppe ZARDO Francesco FAZI Lorena TRAVAGLINI Clara NERVI | 2005 | Cell Research2005，15，9： | 5 |
| 3 | Retinoic acid receptors and cancer: from molecular mechanisms to therapy显示文摘 | Alessandra di Masi Loris Leboffe Elisabetta De Marinis Francesca Pagano Laura Cicconi Cécile Rochette-Egly Francesco Lo Coco Paolo Ascenzi Clara Nervi | 2014 | Molecular Aspects of Medicine2014，，： | 2 |
| 4 | Chemosensitization of acute myeloid leukemia (AML) following mobilization by the CXCR4 an- tagonist AMD3100显示文摘 | Nervi B Ramirez P Rettig MP | 2009 | Blood2009，113，24： | 1 |
| 5 | Overall prognosis in younger patients with eolorectal carcinoma 显示文摘 | Nervi M Gambaccini D Ambrosini CE | 2006 | Minerva Chir2006，61，5： | 1 |
| 6 | Chemosensitization of acute myeloid leukemia (AML) following mobilizationby the CXCR4 antagonist AMD3100 显示文摘 | Nervi B Ramirez P Rettig MP | 2009 | Blood2009，113，24： | 1 |
| 7 | Chemosensitization of acute myeloid leukemia (AML) following mobilization by the CXCR4 antagonist AMD3100显示文摘 | Nervi B Ramirez P Rettig M P | 2009 | Blood2009，113，24： | 1 |
| 8 | Hepatic cholesterol transport from plasma into bile:implications for gallstone disease显示文摘 | Zanlungo S Rigotti A Nervi F | 2004 | Curr Opin Lipidol2004，15，3： | 1 |
| 9 | Histone deacetylases: a common molecular target for differentiation treatment of acute myeloid leukemias? 显示文摘 | Nervi C Lo CF | 2001 | Oncogene2001，20，24： | 1 |
| 10 | Fusion proteins of the retinoic acid receptor - alpha recruit histone deacetylase in promyelocytic leukemia 显示文摘 | Grignani F De Matteis S Nervi C | 1998 | Nature1998，391，6659： | 1 |
| 11 | Acute promylocytic leukemia: a curable disease显示文摘 | LOCOCO F NERVI C AVVISATI G | 2003 | Leukemia2003，12，5： | 1 |
| 12 | Constitutive degradation of the PML-RARαprotein is present in retinoic acid (RA)2resistant acute promyelocytic leukemia cells and involves the proteasome proteolytic pathway显示文摘 | Fanelli M Nervi C Pelicci P G | 1997 | Blood1997，190，： | 1 |
| 13 | Fusion proteins of the retinoic acid receptor-alpha recruit histone deacetylase in promyelocytic leukaemia显示文摘 | Grignani F De MS Nervi C | 1998 | Nature1998，391，： | 1 |
| 14 | Thyroid carcinoma in intrathoracic goiter显示文摘 | Nervi M Iacconi P Spinelli C | 1998 | Langenbecks Arch Surg1998，383，5： | 1 |
| 15 | Fusion proteins of the retinoic acid receptor-alpha recruit histone deacetylase in promyelocytic leukaemia显示文摘 | Grignani F Dematteis S Nervi C | 1998 | Nature1998，391，： | 1 |
| 16 | The cationic cell-penetrating peptide CPP (TAT) derived from the HIV-1 protein TAT is rapidly transported into living fibroblasts: optical, biophysical, and metabolic evidence显示文摘 | Ziegler A Nervi E Durrenberger M | 2005 | Biochemistry2005，44，： | 1 |
| 17 | Specific deficiency of p561ck expression in T lymphocytes from type 1 diabetic patients 显示文摘 | NERVI S ATLAN G C KAHN P B | 2000 | J Immunol2000，165，10： | 1 |
| 18 | Histone deacetylases:a common molecular target for differentiation treatment of acute myeloid leukemias?显示文摘 | Minucci S Nervi C Lo Coco F | 2001 | Oncogene2001，20，24： | 1 |
| 19 | Thyroid carcinoma in intrathoracic goiter显示文摘 | Nervi M Iacconi P Spinelli C | 1998 | Langebecks Arch Surg1998，383，5： | 1 |
| 20 | Eosinophilic pustular folliculitis :a 40 year retrospect显示文摘 | Nervi SJ Schwartz RA Dmochowski M | 2006 | J Am Acad Dermatol2006，55，2： | 1 |
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