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28篇 您的检索式：作者名="Kompa AR"

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	题名	作者	年代	出处	被引量
1	Angiotensin receptor nepri- ysin inhibitor LCZ696 attenuates cardiac remodeling and dysfunction tfter myocardial infarction by reducing cardiac fibrosis and hypertrophy显示文摘	van Lueder TG Wang BH Kompa AR	2015	3irc Heart Fail2015，8，1：	1
2	The angiotensin-receptor nepril- ysin inhibitor LCZ696 attenuates cardiac remodeling and dysfunction after myo- cardial infarction by reducing cardiac fibrosis and hypertrophy显示文摘	von Lueder TG Wang BH Kompa AR	2015	Circ Heart Fail2015，8，1：	1
3	Myocardial in- farctionimpairs renal function, induces renal interstitial fibro- sis, and increases renal KIM-1 expression: implications for cardiorenalsyndrome显示文摘	Lekawanvijit S Kompa AR Zhang Y	2012	Am J Physiol Heart Circ Physiol2012，302，9：	1
4	Chronic urotensin-Ⅱ infusion induces diastolic dysfunction and enhances collagen production in rats显示文摘	Tran L Kompa AR Kemp W Phrommintikul A Wang BH Krum H	2010	Am J Physiol Heart Circ Physiol2010，298，2：	1
5	Long-term but not short-termp38 mitogen-activated protein kinase inhibition improves cardiacfunction and reduces cardiac remodeling post-myocardial infarction显示文摘	Kompa AR See F Lewis DA	2008	J Pharmacol Exp Ther2008，325，3：	1
6	Inotropie responses to hmnan gene 2 (B29) relaxin in a rat model of myocardial infarction: effect of pertussis toxin 显示文摘	Kompa AR Samuel CS Summers RJ	2002	Br J Pharmaeol2002，137，5：	1
7	Cardiorenal syndrome: the emerging role of protein-bound uremic toxins 显示文摘	Lekawanvijit S Kompa AR Wang BH eL al	2012	Circ Res2012，111，11：	1
8	Long-term but not short-term p38 mitogen-activated protein kinase inhibition improves cardiac function and reduces cardiac remodeling post-myocardial infarction 显示文摘	Kompa AR See F Lewis DA	2008	The Journal of Pharmacology and Experimental Therapeutics2008，325，3：	1
9	Angiotensin Receptor Neprilysin Inhibitor LCZ696 Attenuates Cardiac Remodeling and Dysfunction After Myoeardial Infarction by Reducing Cardiac Fibrosis and Hypertrophy显示文摘	yon LTG Wang BH Kompa AR	2015	Cite Heart Fail2015，8，1：	1
10	Cardiorenal syn- drome: the emerging role of protein-bound uremic toxins显示文摘	Lekawanvijit S Kompa AR Wang BH	2012	Circ Res2012，111，11：	1
11	Antagonists of organic anion trans- porters 1 and 3 ameliorate adverse cardiac remodeling induced by uremic toxin indoxyl sulfate 显示文摘	Liu S Wang BH Kompa AR	2012	Int J Cardiol2012，158，3：	1
12	Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate 显示文摘	Lekawanvijit S Kompa AR Manabe M	2012	PLoS One2012，7，41：	1
13	Myocardial infarction impairs renal function, induces renal interstitial fibrosis, and increases renal KIM-I expression: implications for cardiorenal syndrome 显示文摘	Lekawanvijit S Kompa AR Zhang Y	2012	Am J Physiol Heart Cire Physiol2012，302，9：	1
14	Antagonists of organic anion transporters 1 and 3 ameliorate adverse cardiac remodelling induced by uremic toxin indoxyl sulfate显示文摘	Liu S Wang BH Kompa AR	2012	Int J Cardiol2012，158，3：	1
15	Subtotal nephrectomy accelerates pathological cardiac remodeling post-myocardial infarction: implications for cardiorenal syndrome 显示文摘	Liu S Kompa AR Kumfu S	2013	Int J Cardiol2013，168，3：	1
16	Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate 显示文摘	Lekawanvijit S Kompa AR Manabe M	2012	PLoS One2012，7，41：	1
17	Antagonists of organic anion transporters 1 and 3 ameliorate adverse cardiac remodelling induced by uremic toxin indoxyl sulfate 显示文摘	Liu S Wang BH Kompa AR	2012	Int J Cardiol2012，158，3：	1
18	Cardiorenal syndrome: the emerging role of protein-bound uremic toxins 显示文摘	Lekawanvijit S Kompa AR Wang BH	2012	Circ Res2012，111，1：	1
19	Early and delayed tranilast :reatment reduces pathological fibrosis following myocardial infarction 显示文摘	See F Watanabe M Kompa AR	2013	Heart Lung Circ2013，22，2：	1
20	Cardiovascular role of urotensin Ⅱ:effect of chronic infusion in the rat显示文摘	Kompa AR Thomas WG See F	2004	Peptides2004，25，10：	1

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