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199篇 您的检索式:作者名="Hetz"
    题名 作者 年代 出处 被引量
1Plasma Disappearance Rate of Indocyanine Green in Liver Dysfunction显示文摘P. Faybik H. Hetz 2006Transplantation Proceedings2006,,3:3
2Gene therapy in Parkinson's disease: targeting the endplasmic reticulum proteostasis network显示文摘Parkinson’s disease(PD)is the second most common neurodegenerative disease affecting 1%of the population over 60 years of age.The progressive degeneration of dopaminergic neurons at the substantia nigra pars compacta(SNpc)results in a severe and gradual depletion of dopamine content in the striatum,a phenomena that is responsible for the characteristic motor symptoms of this disease.There is no cure for PD and available treatments only aim to restore dopamine deficits.Administration of the dopamine precursor Levodopa(L-DOPA)is the main temporal palliative treatment that increases overall dopamine levels.However,its chronic use limits its effectiveness and generates a number of adverse effects such as debilitating dyskinesia.Valentina Castillo Gabriela Mercado Claudio Hetz 2015Neural Regeneration Research2015,10,7:2
3Biogradation of aliphaticaromatic copolyesters by Thermomonospora fusca and other thermophilic compost isolates显示文摘Kleeberg I Hetz C Kroppenstedt R M 1998Applied and Environmental Microbiology1998,64,:1
4In vitro generation of infectious scrapie prions 显示文摘Castilla J Saa' P Hetz C 2005Cell2005,121,:1
5The unfolded protein response:controlling cell fate decisions under ER stress and beyond显示文摘Hetz C 2012Nat Rev Mol Cell Biol2012,13,2:1
6Targeting the unfolded protein response in disease 显示文摘Hetz C Chevet E Harding HP 2013Nat Rev Drug Discov2013,12,9:1
7Modulating stress responses by the UPRosome: a matter of life and death 显示文摘Woehlbier U Hetz C 2011Trends Biochem Sci2011,36,6:1
8Targeting the unfolded protein response in disease 显示文摘Hetz C Chevet E Harding HP 2013Nat Rev Drug Discov2013,,:1
9Turning off the unfolded protein response An interplay betw een the apoptosis machi nery and ER stress signaling显示文摘Lisbona F Hetz C 2009Cell Cycle2009,8,11:1
10Is MELD score sufficient to predict not only death on waiting list,but also posttransplant survival?显示文摘Silberhumer G R Hetz H Rasoul-Rockenschaub S 2006Transpl Irt2006,19,:1
11Caspase-cleaved cytokeratin 18 and 20S proteasome in liver degeneration显示文摘Hetz H Hoetzenecker K Hacker S 0,,02:1
12Bilateral lung transplantation with intra- and postoperatively prolonged ECMO support in patients with pulmonary hypertension显示文摘Arpad Pereszlenyi Georg Lang Heinz Steltzer Hubert Hetz Alfred Kocher Petra Neuhauser Wilfried Wisser Walter Klepetko 2002European Journal of Cardio-Thoracic Surgery2002,,5:1
13Short‐term versus long‐term induction therapy with antithymocyte globulin in orthotopic liver transplantation显示文摘Thomas Soliman Hubert Hetz Christoph Burghuber Georg Gy?ri Gerd Silberhumer Rudolf Steininger Ferdinand Mühlbacher Gabriela A. Berlakovich 2007Transplant International2007,,5:1
14Microcin E492,a channel-forming hacteriocin from Klebsiella pneumoniae,induces apoptosis in some human cell lines显示文摘HETZ C BONO M R BARROS L F 2002Proc Natl Acad Sci USA2002,99,5:1
15Crosstalk between the UPR and autophagy pathway contributes to handling cellular stress in neurodegenerative disease显示文摘Vidal RL Hetz C 2012Autophagy2012,8,6:1
16The unfolded protein response: controlling cell fate decisions under ER stress and beyond 显示文摘Hetz C 2012Nat Rev Mol Cell Biol2012,13,2:1
17Unfolded protein response transcription factor XBP-1 does not influence prion replication or pathogenesis显示文摘Hetz C Lee AH Gonzalez-Romero D 2008Proc Natl Acad Sci USA2008,105,2:1
18Proapoptotic BAX and BAK modulate the unfolded protein response by a direct interaction with IRE1 - alpha 显示文摘HETZ C BERNASCONI P FISHER J 2006Science2006,312,:1
19The unfolded protein response : controlling cell fate de- cisions under ER stress and beyond 显示文摘HETZ C 2012Nature Reviews:Molec- ular Cell Biology2012,13,2:1
20XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy显示文摘Hetz C Thielen P Matus S 2009Genes Dev2009,23,:1
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