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| 1 | Neutrophil depletion-but not prevention of Kupffer cell activation-decreases the severity of cerulein-induced acute pancreatitis显示文摘瞄准:为了决定嗜中性的弄空和 Kupffer 房间抑制是否可能联合,他们的 protective 完成尖锐胰腺炎的严厉到减少。方法:老鼠有 cerulein 管理导致尖锐胰腺炎并且是有任何一个反老鼠 neutrophil 浆液或金轧氯化物(GdCl3 ) 的 pretreated 阻止 Kupffer 房间激活,或两个处理。损害在胰和肺被估计。Myeloperoxidases (军邮局) 估计了嗜中性的渗入。Interleukin-6 (IL-6 ) 和 IL-10 在浆液,胰,肺和肝被测量。结果:在有尖锐胰腺炎的老鼠,嗜中性的弄空减少了胰腺炎和联系胰腺炎的肺损害的严厉。在由 GdCl3 的激活的 Kupffer 房间有更少的保护的效果,尽管 IL-6 和 IL-10 集中显著地被减少。嗜中性的弄空带的普雷茨氏疗法没被 Kupffer 房间在激活提高,两个处理没联合他们的保护的效果。结论:当 Kupffer 房间激活的角色是不太明显的时,我们的结果在在尖锐胰腺炎加重机关损害证实激活的 neutrophils 的角色。 | Catherine M Pastor Alain Vonlaufen Fabianna Georgi Antoine Hadengue Philippe Morel Jean-Louis Frossard | 2006 | World Journal of Gastroenterology2006，12，8： | 22 |
| 2 | Age,smoking and overweight contribute to the development of intestinal metaplasia of the cardia显示文摘AIM:To assess the role of Helicobacter pylori(H.pylori),gastroesophageal reflux disease(GERD),age,smoking and body weight on the development of intestinal metaplasia of the gastric cardia(IMC).METHODS:Two hundred and seventeen patients scheduled for esophagogastroduodenoscopy were enrolled in this study.Endoscopic biopsies from the esophagus,gastroesophageal junction and stomach were evaluated for inflammation,the presence of H.pylori and intestinal metaplasia.The correlation of these factors with the presence of IMC was assessed using logistic regression.RESULTS:IMC was observed in 42% of the patients.Patient age,smoking habit and body mass index(BMI) were found as potential contributors to IMC.The risk of developing IMC can be predicted in theory by combining these factors according to the following formula:Risk of IMC = a + s-2B where a = 2,…6 decade of age,s = 0 for non-smokers or ex-smokers,1 for < 10 cigarettes/d,2 for > 10 cigarettes/d and B = 0 for BMI < 25 kg/m2(BMI < 27 kg/m2 in females),1 for BMI > 25 kg/m2(BMI > 27 kg/m2 in females).Among potential factors associated with IMC,H.pylori had borderline signif icance(P = 0.07),while GERD showed no signif icance.CONCLUSION:Age,smoking and BMI are potential factors associated with IMC,while H.pylori and GERD show no significant association.IMC can be predicted in theory by logistic regression analysis. | Christian Felley Hanifa Bouzourene Marianne Bründler G VanMelle Antoine Hadengue Pierre Michetti Gian Dorta Laurent Spahr Emiliano Giostra Jean Louis Frossard | 2012 | World Journal of Gastroenterology2012，18，17： | 5 |
| 3 | Budd–Chiari syndrome: a review by an expert panel显示文摘 | Harry L.A Janssen Juan-Carlos Garcia-Pagan Elwyn Elias Gilles Mentha Antoine Hadengue Dominique-Charles Valla | 2003 | Journal of Hepatology2003，，3： | 3 |
| 4 | High prevalence of cholestasis, with increased conjugated bile acids in inflammatory bowel diseases patients显示文摘AIM To investigate the prevalence and causes of cholestasis in patients with inflammatory bowel diseases in the Swiss Inflammatory Bowel Diseases Cohort. METHODS A retrospective cohort study was performed of all the patients in the Swiss Inflammatory bowel disease Cohort. Total bile acid was measured for all patients and cholestasis was defined as a concentration > 8 μmol/L. The characteristics of patients with or without cholestasis were compared. Bile acid profiles were then determined for 80 patients with high total bile acid and 80 matched patients with low total bile acid. Bile acid profiles were compared for smokers vs nonsmokers, ileal vs colonic disease, and inflammatory vs non inflammatory diseases.RESULTS Ninety-six patients had more than 8 μmol/L total bile acid, giving a prevalence of 7.15%. Patients with an obvious cause of cholestasis, such as primary sclerosing cholangitis, were then excluded, leaving 1190 participants with total bile acid < 8 μmol/L and 80 with total bile acid > 8 μmol/L. In multivariate analysis, calcium supplementation was significantly associated with cholestasis(odds ratio, 2.36, 95%CI: 1.00-5.21, P = 0.040) whereas current smoking significantly reduced the risk of cholestasis(odds ratio, 0.42, 95%CI: 0.17-0.91, P = 0.041). Levels of all conjugated bile acids were higher in the cholestasis group than in the control group. When we compared patients with ileal vs colonic disease, the former had higher levels of primary, secondary, and tertiary bile acids whereas patients with colonic disease had higher levels of conjugated bile acids.CONCLUSION Prevalence of cholestasis is high. Smoking appears to reduce cholestasis. Conjugated bile acids are higher in cholestasis and in colonic disease whereas unconjugated in ileal disease. | Marc Girardin Antoine Hadengue Jean-Louis Frossard | 2018 | World Journal of Clinical Cases2018，6，4： | 2 |
| 5 | EASL International Consensus Conference on Hepatitis B 显示文摘 | De Franehis R Hadengue A Lau G | 2003 | J Hepatol2003，39，： | 1 |
| 6 | Beneficial effects of the 2-days administration of terlipressin in patients with cirrhosis and hepatorenal syndrome显示文摘 | Hadengue A Gadano A Moreau R | 1998 | J Hepatol1998，29，： | 1 |
| 7 | Current outcome of portal vein thrombosis in adults: Risk and benefit of anticoagulant therapy显示文摘 | Bertrand Condat Fabienne Pessione Sophie Hillaire Marie-Helene Denninger Marie-Claude Guillin Marc Poliquin Antoine Hadengue Serge Erlinger Dominique Valla | 2001 | Gastroenterology2001，，2： | 1 |
| 8 | Pulmonary hypertension complicating portal hypertension:prevalence and relation to splanchnic hemodynamics 显示文摘 | Hadengue A Benhayoun MK Lebree D | 1991 | Gastroenterology1991，100，2： | 1 |
| 9 | Hepatic venous outflow block caused by short-length hepatic vein stenoses显示文摘 | Hadengue A Boudet MJ | 1997 | Hepatology1997，25，4： | 1 |
| 10 | Effect of hyperthermia on NF-kappa B binding activity in cerulein-induced acute pancreatitis显示文摘 | Frossard JL Pastor CM Hadengue A | 2001 | Am J Physiol Gastrointest Liver Physiol2001，280，6： | 1 |
| 11 | Transjugular intrahepatic portosystemic shunts:comparison with paracentesis in patients with cirrhosis and refractory ascites:randomized trial显示文摘 | Lebrec D Giuily N Hadengue A | 1996 | J Hepatol1996，25，： | 1 |
| 12 | The changingscene of hepatic vein thrombosis: recognition of a symptomatic cases显示文摘 | Hadengue A Poliquin M Vilgrain V | 1994 | Gastroenterology1994，106，4： | 1 |
| 13 | EASL international consensus conference on hepatitis B显示文摘 | DE FRANCHIS R HADENGUE A LAU G | 2003 | J Hepatol2003，，： | 1 |
| 14 | Pulmonary hypertension complicating portal hypertension: prevalence and relation to splanchnic hemodynamics 显示文摘 | Hadengue A Benhayoun MK Lebree D | 1991 | Gastroenterology1991，100，2： | 1 |
| 15 | Outcome of Budd-Chiari syndrome:a multivariate analysis of factors related to survival including surgical portosystemic shunting显示文摘 | Escolano S Hadengue A | 1999 | Hepatology1999，30，1： | 1 |
| 16 | What Can E-Learning Do for University Libraries? 显示文摘 | Ve'ronique Hadengue | 2004 | Library Review2004，53，8： | 1 |
| 17 | EASL interna- tional consensus conference on hepatitis B显示文摘 | de Franchis R Hadengue A Lau G | 2003 | J Hepatol2003，39，1： | 1 |
| 18 | Choledocholithiasis:a prospective study of spontaneous common bile duct stone migration显示文摘 | FROSSARD JL HADENGUE A AMOUYAL G | 2000 | Gastroiutest Endosc2000，51，： | 1 |
| 19 | EASL International Consensus Conference on Hepatitis B 显示文摘 | DE Franchis R Hadengue A Lau G | 2003 | J Hepatol2003，39，11： | 1 |
| 20 | Hemodynamic and metabolic effects of terlipressin in patients with cirrhosis receiving a nonselectiveβ-blocker显示文摘 | Florence Vachiery MD Dr. Richard Moreau MD Dr. Adrian Gadano MD Song Yang MD Phillippe Sogni MD Antoine Hadengue MD Stephane Cailmail Thierry Soupison MD Didier Lebrec MD | 1996 | Digestive Diseases and Sciences1996，，9： | 1 |
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