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| 1 | Fatigue of self-piercing riveted joints in aluminum alloy 6111显示文摘 | Fu Maofeng Mallick P K | 2003 | International Journal of Fatigue2003，25，3： | 1 |
| 2 | Effect of process variables on the static and fatigue properties of self-piercing riveted joints in aluminum alloy 5754显示文摘 | Fu Maofeng Mallick P K | 2001 | SAE Paper2001，，： | 1 |
| 3 | Fatigue of self-piercing riveted joints in aluminum alloy 6111显示文摘 | Fu Maofeng Mallick P K | | 0，，03： | 1 |
| 4 | Fatigue of self-piercing riveted joints in aluminum alloy 6111 显示文摘 | Maofeng Fu Mallick P K | 2003 | International Journal of Fatigue2003，25，： | 1 |
| 5 | Fatigue of self-piercing riveted joints in aluminum alloy 6111显示文摘 | Maofeng Fu P.K Mallick | 2002 | International Journal of Fatigue2002，，3： | 1 |
| 6 | Effect of process variables on the static and fatigue properties of self-piercing riveted joints in aluminun alloy 5754 显示文摘 | Maofeng Fu Mallick P K | 2001 | Society of Automotive Engineers2001，2，： | 1 |
| 7 | Fatigue of Self-Piercing Riveted Joints in an Aluminum Alloy6111显示文摘 | PK Mallick | 2003 | International Journal of Fatigue2003，3，2： | 1 |
| 8 | Fatigue of self-piercing riveted joints in aluminum alloy 6111 显示文摘 | Maofeng Fu Mallick P K | 2003 | International Journal of Fatigue2003，25，3： | 1 |
| 9 | Effect of Process Variables on the Static and Fatigue Properties of Self-Piercing Riveted Joints in Aluminum Alloy 5754显示文摘 | PK Mallick | 2001 | Sae Paper2001，，1： | 1 |
| 10 | Fatigue of self-piercing riveted joints in aluminum alloy 6111 显示文摘 | Fu Maofeng Mallick P K | 2003 | Iutemational Journal of Fatigue2003，25，： | 1 |
| 11 | Fatigue of self-piercing riveted joints in aluminum alloy 6111显示文摘 | FU Maofeng MALLICK P K | 2003 | International Journal of Fatigue2003，25，3： | 1 |
| 12 | Fatigue of self-piercing riveted joints in aiuminum alloy 6111 显示文摘 | Fu Maofeng Mallick P K | 2003 | International Journal of Fatigue2003，25，： | 1 |
| 13 | Making Use of Plant uORFs to Control Transgene Translation in Response to Pathogen Attack显示文摘Reducing crop loss to diseases is urgently needed to meet increasing food production challenges caused by the expanding world population and the negative impact of climate change on crop productivity.Disease-resistant crops can be created by expressing endogenous or exogenous genes of interest through transgenic technology.Nevertheless,enhanced resistance by overexpressing resistance-produced genes often results in adverse developmental affects.Upstream open reading frames(uORFs)are translational control elements located in the 5′untranslated region(UTR)of eukaryotic mRNAs and may repress the translation of downstream genes.To investigate the function of three uORFs from the 5′-UTR of ACCELERATED CELL 11(uORFsACD11),we develop a fluorescent reporter system and find uORFsACD11 function in repressing downstream gene translation.Individual or simultaneous mutations of the three uORFsACD11 lead to repression of downstream translation efficiency at different levels.Importantly,uORFsACD11-mediated translational inhibition is impaired upon recognition of pathogen attack of plant leaves.When coupled with the PATHOGENESIS-RELATED GENE 1(PR1)promoter,the uORFsACD11 cassettes can upregulate accumulation of Arabidopsis thaliana LECTIN RECEPTOR KINASE-VI.2(AtLecRKVI.2)during pathogen attack and enhance plant resistance to Phytophthora capsici.These findings indicate that the uORFsACD11 cassettes can be a useful toolkit that enables a high level of protein expression during pathogen attack,while for ensuring lower levels of protein expression at normal conditions. | Gan Ai Jin Liu Xiaowei Fu Tianli Li Hai Zhu Ying Zhai Chuyan Xia Weiye Pan Jialu Li Maofeng Jing Danyu Shen Ai Xia Daolong Dou | 2022 | BioDesign Research2022，，1： | 0 |
| 14 | The role of Class Ⅱ KNOX family in controlling compound leaf patterning in Medicago truncatula显示文摘Compound leaf development requires the coordination of genetic factors, hormones, and other signals. In this study, we explored the functions of Class Ⅱ KNOTTED-like homeobox(KNOXII)genes in the model leguminous plant Medicago truncatula. Phenotypic and genetic analyses suggest that Mt KNOX4, 5 are able to repress leafet formation, while Mt KNOX3, 9, 10 are not involved in this developmental process. Further investigations have shown that Mt KNOX4 represses the CK signal transduction, which is downstream of Mt KNOXⅠ-mediated CK biosynthesis. Additionally,two boundary genes, FUSED COMPOUND LEAF1(orthologue of Arabidopsis Class M KNOX) and NO APICAL MERISTEM(orthologue of Arabidopsis CUP-SHAPED COTYLEDON), are necessary for Mt KNOX4-mediated compound leaf formation.These findings suggest, that among the members of Mt KNOXⅡ, Mt KNOX4 plays a crucial role in integrating the CK pathway and boundary regulators,providing new insights into the roles of Mt KNOXⅡ in regulating the elaboration of compound leaves in M. truncatula. | Xiao Wang Juanjuan Zhang Maofeng Chai Lu Han Xiaohua Cao Jing Zhang Yiming Kong Chunxiang Fu Zeng‐Yu Wang Kirankumar SMysore Jiangqi Wen Chuanen Zhou | 2023 | Journal of Integrative Plant Biology2023，65，10： | 0 |
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