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| 1 | Low-gain integral control of continuous-time linear systems subject to input and output nonlinearities显示文摘 | FLIEGNER T LOGEMANN H RYAN E P | 2003 | Automatica2003，39，： | 1 |
| 2 | Up-regulation of PPAR gamma in myocardial infarction 显示文摘 | FLIEGNER D WESTERMANN D RIAD A | 2008 | Eur J Heart Fail2008，10，1： | 1 |
| 3 | Up-regulation of PPARγ in myocardial infarction显示文摘 | Fliegner D Westermann D Riad A | 2008 | Eur J Heart Fail2008，10，1： | 1 |
| 4 | Up-regulation of PPARγ in myocardial infarction显示文摘 | Fliegner D Westermann D Riad A | 2008 | Eur J Heart Fail2008，10，1： | 1 |
| 5 | Management of vaginal agenesis with a functioning uterus Is hysterectomy advisable显示文摘 | Fliegner JR Pepperell RJ | | 0，，04： | 1 |
| 6 | Low-gain integral control of continuous-time linear systems subject to input and output nonlinearities显示文摘 | Fliegner T Logemann H Ryan E P | 2003 | Automatica2003，39，3： | 1 |
| 7 | Low-gain integral control of continuous-time linear systems subjected to input and output nonlinearities显示文摘 | Fliegner T Logemarm H Ryan E P | 2003 | Automatic2003，39，： | 1 |
| 8 | Exemplary for Drivage of major Cross Sections in soft Soil? 显示文摘 | Eberhard Fliegner The Mount Baker Tunnel/USA | 1990 | Tunnel1990，，4： | 1 |
| 9 | Up-regulation of PPAR gamma in myocardial infarction显示文摘 | Fliegner D Westermann D Riad A | 2008 | Eur J Heart Fail2008，10，： | 1 |
| 10 | Low-gain integral control of continuous-time linear systems subject to input and output nonlinearities显示文摘 | Fliegner T Logemann H Ryan E P | 2003 | Automatica2003，39，3： | 1 |
| 11 | Evaluation of the class Ⅱ region of the major histocompatibility complex of the greyhound with the genomic matching technique and sequence-based typing显示文摘 | Fliegner RA Holloway SA Lester S | 2008 | Tissue Antigens2008，72，2： | 1 |
| 12 | Cellular and molecular biology of neuronal intermediate filaments显示文摘 | Fliegner KH Liem RKH | 1991 | Int Res Cytol1991，131，： | 1 |
| 13 | Sex-specific pathways in early cardiac response to pressure overload in mice显示文摘Pressure overload(PO) first causes cardiac hypertrophy and then heart failure(HF), which are associated with sex differences in cardiac morphology and function. We aimed to identify genes that may cause HF-related sex differences. We used a transverse aortic constriction(TAC) mouse model leading to hypertrophy without sex differences in cardiac function after 2 weeks, but with sex differences in hypertrophy 6 and 9 weeks after TAC. Cardiac gene expression was analyzed 2 weeks after surgery. Deregulated genes were classified into functional gene ontology(GO) categories and used for pathway analysis. Classical marker genes of hypertrophy were similarly upregulated in both sexes(α-actin, ANP, BNP, CTGF). Thirty-five genes controlling mitochondrial function(PGC-1, cytochrome oxidase, carnitine palmitoyl transferase, acyl-Co A dehydrogenase, pyruvate dehydrogenase kinase) had lower expression in males compared to females after TAC. Genes encoding ribosomal proteins and genes associated with extracellular matrix remodeling exhibited relative higher expression in males(collagen 3, matrix metalloproteinase 2, TIMP2, and TGFβ2, all about twofold) after TAC. We confirmed 87% of the gene expression by real-time polymerase chain reaction. By GO classification, female-specific genes were related to mitochondria and metabolism and males to matrix and biosynthesis. Promoter studies confirmed the upregulation of PGC-1 by E2. Less downregulation of metabolic genes in female hearts and increased protein synthesis capacity and deregulation of matrix remodeling in male hearts characterize the sex-specific early response to PO. These differences could contribute to subsequent sex differences in cardiac function and Hf. | Henning Witt Carola Schubert Juliane Jaekel Daniela Fliegner Adam Penkalla Klaus Tiemann Joerg Stypmann Stefan Roepcke Sebastian Brokat Shokoufeh Mahmoodzadeh Eva Brozova Mercy M.Davidson Patricia Ruiz Noppinger Christian Grohé Vera Regitz-Zagrosek | 2015 | 世界最新医学信息文摘2015，15，A0： | 1 |
| 14 | Hydroxylmethyl-phytochelatinsare metal induced peptides of the Peacea显示文摘 | Klapheck S Fliegner W Zimmer I | 1994 | Plant Physiology1994，104，： | 1 |
| 15 | Behaviour of helical pitch in cholesteric and chiral smectic C' phases 显示文摘 | Ch Bahr C Escher D Fliegner | 2010 | Gen- eral & Introductory Chemistry2010，95，12： | 1 |
| 16 | Sex differences in animal models for cardiovascular diseases and the role of estrogen显示文摘 | Mabmoodzadeh S Fliegner D Dworatzek E | | 0，，214： | 1 |
| 17 | Sex differences in animal models for cardiovascular diseases and the role of estro- gen显示文摘 | Mahmoodzadeh S Fliegner D Dworatzek E | 2012 | Handb Exp Pharmacol2012，214，9： | 1 |
| 18 | Female sex and estrogen receptor-beta attenuate cardiac remodeling and apoptosis in pressure overload 显示文摘 | Fliegner D Schubert C Penkalla A | 2010 | Am J Physiol Regul In- tegr Comp Physiol2010，298，6： | 1 |
| 19 | Female sex and estrogen receptor-β attenuate cardiac remodeling and apoptosis in pressure overload显示文摘 | Fliegner D Schubert C Penkalla A | 2010 | Am J Physiol Regul Integr Comp Physiol2010，298，6： | 1 |
| 20 | Female sex and estrogen receptor-beta attenuate cardiac remodeling and apoptosis in pressure overload显示文摘 | Fliegner D Schuvert C Penkalla A | 2010 | Am J Physiol Regul Integr Comp Physiol2010，298，6： | 1 |
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