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6篇 您的检索式:作者名="FRIEDEMANN WEBER"
    题名 作者 年代 出处 被引量
1Interaction of hepatitis C virus with the type I interferon system显示文摘Hepatitis C virus (HCV) needs to tightly manipulate host defences in order to establish infection. The innate immune response slows down viral replication by activating cytokines such as the type I interferons (IFN-α/ β), which trigger the synthesis of antiviral proteins and modulate the adaptive immune system. HCV has therefore developed a number of countermeasures to stay ahead of the IFN system. Here, I will attempt to summarize the current state of research regarding IFN responses against HCV and the viral escape strategies. Particular emphasis will be put on the newly discovered mechanisms HCV employs to avoid the induction of IFN in infected cells.Friedemann Weber 2007World Journal of Gastroenterology2007,13,36:3
2A target on the move: Innate and adaptive immune escape strategies of hepatitis C virus显示文摘Robert Thimme Volker Lohmann Friedemann Weber 2005Antiviral Research2005,,3:1
3Regulatory,biosafety and safety challenges for novel cells as substrates for human vaccines显示文摘Ralf D Hess Friedemann Weber Keith Watsond 2012Vaccine2012,30,:1
4Fazakerley,Bunyamwera bunyavirus nonstructural product that contributes to viral pathogenesis显示文摘 FRIEDEMANN WEBER JOHN K 2001PNSA2001,98,:1
5Antigenic drift, antigenic shift and interferon antagonists: how bunyaviruses counteract the immune system显示文摘Friedemann Weber Richard M Elliott 2002Virus Research2002,,1:1
6Viperin is an iron-sulfur protein that inhibits genome synthesis of tick-borne encephalitis virus via radical SAM domain activity显示文摘Arunkumar S Upadhyay Kirstin Vonderstein Andreas Pichlmair Oliver Stehling Keiryn L Bennett Gerhard Dobler Ju Tao Guo Giulio Superti Furga Roland Lill Anna K Overby Friedemann Weber 2014Cell Microbiol2014,16,6:1
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