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| 1 | Laparo-endoscopic “Rendezvous” to treat cholecysto-choledocolithiasis: Effective,safe and simplifies the endoscopist’s work显示文摘AIM: To investigate our clinical experience with combined laparo-endoscopic Rendezvous (RV) for the treatment of patients affected by gallstones and common bile duct (CBD) stones and especially to study the never evaluated opinion of the endoscopist concerning the difficulty of the intraoperative endoscopic procedure during the RV in comparison with standard endoscopic retrograde cholangio-pancreatography (ERCP). METHODS: Eighty consecutive patients affected by cholecystolithiasis and diagnosed or suspected CBD stones were treated with a standardized 'tailored' RV. The relevant technical features, the feasibility, the effectiveness in stone clearance, the safety but also the simple evaluation of difficulty and agreement of the endoscopist were analyzed with a questionnaire. RESULTS: The feasibility was 97.5% and the effectiveness 100% concerning CBD clearance and solution of coexisting problems at the papilla. Minor morbidity was 3.3%, the operating time was prolonged by a mean of 14 min, the mean hospital stay was 3.8 d and only one stone’s recurrence occurred. The endoscopist evaluated the procedure to be simpler than standard ERCP-ES in 81.2% of the cases.CONCLUSION: Simultaneous RV carries higheffectiveness and safety at least comparable to those reported for other options. The endoscopist is very often satisfied with this approach because of the minimization of some steps of the endoscopic procedure and avoidance of relevant iatrogenic risk factors. If the mandatory collaboration between surgeons and endoscopists is guaranteed, this approach can often be preferable for the patient, the surgeon, the endoscopist and the hospital. | Gaetano La Greca Francesco Barbagallo Michele Di Blasi Andrea Chisari Rosario Lombardo Rosario Bonaccorso Saverio Latteri Andrea Di Stefano Domenico Russello | 2008 | World Journal of Gastroenterology2008，14，18： | 15 |
| 2 | Aetiology of Legg-Calvé-Perthes disease:A systematic review显示文摘BACKGROUND Legg-Calvé-Perthes disease(LCPD) is a clinical condition affecting the femoral head of children during their growth. Its prevalence is set to be between 0.4/100000 to 29.0/100000 children less than 15 years of age with a peak of incidence in children aged from 4 years to 8 years. LCPD aetiology has been widely studied, but it is still poorly understood.AIM To analyse the available literature to document the up-to-date evidence on LCPD aetiology.METHODS A systematic review of the literature was performed regarding LCPD aetiology,using the following inclusion criteria: studies of any level of evidence, reporting clinical or preclinical results and dealing with the aetiology or pathogenesis of LCPD. Two reviewers searched the PubMed and Science Direct databases from their date of inception to the 20th of May 2018 in accordance with the Preferred Reporting Items for Systemic Reviews and Meta-Analyses guidelines. To achieve the maximum sensitivity of the search strategy, we combined the terms: ‘‘Perthes disease OR LCPD OR children avascular femoral head necrosis' with 'pathology OR aetiology OR biomechanics OR genetics' as either key words or MeSH terms.RESULTS We include 64 articles in this review. The available evidence on LCPD aetiology is still debated. Several hypotheses have been researched, but none of them was found decisive. While emerging evidence showed the role of environmental risk factors and evidence from twin studies did not support a major role for genetic factors, a congenital or acquired predisposition cannot be excluded in disease pathogenesis. One of the most supported theories involved mechanical induced ischemia that evolved into avascular necrosis of the femoral head in sensible patients.CONCLUSION The literature available on the aetiology of LCPD presents major limitations in terms of great heterogeneity and a lack of high-profile studies. Although a lot of studies focused on the genetic, biomechanical and radiological background of the disease, there is a lack of consensus on one or multiple major actors of the etiopathogenesis. More studies are needed to understand the complex and multifactorial genesis of the avascular necrosis characterizing the disease. | Vito Pavone Emanuele Chisari Andrea Vescio Claudio Lizzio Giuseppe Sessa Gianluca Testa | 2019 | World Journal of Orthopedics2019，10，3： | 7 |
| 3 | Pathogenesis of hepatitis B virus infection显示文摘 | F.V. Chisari M. Isogawa S.F. Wieland | 2009 | Pathologie Biologie2009，，4： | 4 |
| 4 | Hepatitis B virus immunopathogenesis显示文摘 | Chisari FV Ferrari C | | 0，，： | 3 |
| 5 | IMMUNOBIOLOGY AND PATHOGENESIS OF VIRAL HEPATITIS显示文摘 | Luca G. Guidotti Francis V. Chisari | 2006 | Annual Review of Pathology: Mechanisms of Disease2006，，： | 3 |
| 6 | Bifidobacterium longum with Fructo-Oligosaccharides in Patients with Non Alcoholic Steatohepatitis显示文摘 | Michele Malaguarnera Marco Vacante Tijana Antic Maria Giordano Giuseppe Chisari Rosaria Acquaviva Silvana Mastrojeni Giulia Malaguarnera Antonio Mistretta Giovanni Volti Fabio Galvano | 2012 | Digestive Diseases and Sciences2012，，2： | 2 |
| 7 | Hepatitis B virus immunopathogenesis显示文摘 | Chisari F V Ferrari C | 1995 | Ann Rev Immuol1995，13，： | 2 |
| 8 | Toxic hepatitis in occupational exposure to solvents显示文摘The liver is the main organ responsible for the metabolism of drugs and toxic chemicals, and so is the primary target organ for many organic solvents. Work activities with hepatotoxins exposures are numerous and, moreover, organic solvents are used in various industrial processes. Organic solvents used in different industrial processes may be associated with hepatotoxicity. Several factors contribute to liver toxicity; among these are: species differences, nutritional condition, genetic factors, interaction with medications in use, alcohol abuse and interaction, and age. This review addresses the mechanisms of hepatotoxicity. The main pathogenic mechanisms responsible for functional and organic damage caused by solvents are: inflammation, dysfunction of cytochrome P450, mitochondrial dysfunction and oxidative stress. The health impact of exposure to solvents in the workplace remains an interesting and worrying question for professional health work. | Giulia Malaguarnera Emanuela Cataudella Maria Giordano Giuseppe Nunnari Giuseppe Chisari Mariano Malaguarnera | 2012 | World Journal of Gastroenterology2012，18，22： | 2 |
| 9 | Hepatitis B Virus immunopathogenesis 显示文摘 | Chisari F Ferrari C | 1995 | Annu Rev Immuol1995，13，3： | 1 |
| 10 | Hepatocarcinogenesis due to chronic liver cell injury in hepatitis B virus transgenic mice显示文摘 | Dunsford HA Sell S Chisari FV | 1990 | Cancer Res1990，50，11： | 1 |
| 11 | Protein transfer of preformed MHC -peptide complexes sensitizes target cells to T cell eytolysis显示文摘 | Huang JH Getty RR Chisari FV | 1994 | Immunity1994，1，7： | 1 |
| 12 | Pathogenesis of hapatitis Bvirus infection显示文摘 | Chisari FV Isogawa M Wieland SF | 2010 | pathol Biol (Paris)2010，58，4： | 1 |
| 13 | Inhibition of hepa- titis B virus repIication during Schistosoma Mansoni in fection in transgenic mice显示文摘 | Mcclary H Koch R Chisari FV | 2000 | Exp Meal2000，192，2： | 1 |
| 14 | Interleukin-12 inhibits hepatitis B virus replication in transgenic mice显示文摘 | Cavanangh VJ Guidotti LG Chisari FV | 1997 | J Virol1997，71，： | 1 |
| 15 | Hepatitis B virus transgenic mice: insights into the virus and the disease显示文摘 | Chisari FV | 1995 | Hepatology1995，22，： | 1 |
| 16 | Interence of hepatitis C Virus RNA replication by shore interfering RNAs显示文摘 | Kapadia SB Chisari FV | 2003 | Proc Natl Acad Sci USA2003，100，4： | 1 |
| 17 | Hepatitis B virus transgenic mice:insights into the virus and the disease显示文摘 | Chisari FV | 1995 | Hepatology1995，22，： | 1 |
| 18 | Recombinant duck interferon Gamma inhibits duck hepat itis B virus replication in primary hepato-cytes显示文摘 | Schultz U Chisari F V | 1999 | Virol1999，73，4： | 1 |
| 19 | Relative sensitivity of hepatitis B virus and other hepatotropic viruses to the antiviral effects of cytokines显示文摘 | McClary H Koch Chisari F V | 2000 | J viral2000，74，5： | 1 |
| 20 | Inhibition of dsRNA-induced signaling in hepatitis C virus-infected cells by NS3 protease-dependent and independent mechanisms显示文摘 | Cheng G Zhong J Chisari FV | | 0，，： | 1 |
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